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Three candidate tumor-suppressor genes, SEMA3B (show SEMA3B Proteins), AXUD1 and GNAT1 (show GNAT1 Proteins) may be involved in oral squamous cell carcinoma.
Axud1 mediated stress-induced cardiomyocytes apoptosis through activating Wnt (show WNT2 Proteins)/beta-catenin (show CTNNB1 Proteins) signaling pathway.
significant increases in Col1a1 (show COL1A1 Proteins), serine/threonine-protein kinase 1, Ctnnb1 (show CTNNB1 Proteins), CSRNP1, Ddit4 (show DDIT4 Proteins), Cyp2e1 (show CYP2E1 Proteins), and Krit1 (show KRIT1 Proteins) expressions and great decreases inreceptor D2, Neu1 (show NEURL Proteins), and Dhcr7 (show DHCR7 Proteins) expressions following long-term exposure to TiO2 NPs (show NPS Proteins)
This gene encodes a protein that localizes to the nucleus and expression of this gene is induced in response to elevated levels of axin. The Wnt signalling pathway, which is negatively regulated by axin, is important in axis formation in early development and impaired regulation of this signalling pathway is often involved in tumors. A decreased level of expression of this gene in tumors compared to the level of expression in their corresponding normal tissues suggests that this gene product has a tumor suppressor function.
AXIN1 up-regulated 1
, TGF-beta induced apoptosis protein 3
, TGF-beta-induced apoptosis protein 3
, axin-1 up-regulated gene 1 protein
, cysteine/serine-rich nuclear protein 1
, cysteine-serine-rich nuclear protein 1
, Axin-1 up-regulated gene 1 protein