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anti-Human MSX1 Antibodies:
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Human Monoclonal MSX1 Primary Antibody for ELISA, WB - ABIN969296
Li, Cui, Fang, Mei: [A novel mutation of MSX1 gene in a Chinese pedigree with oligodontia]. in Zhonghua kou qiang yi xue za zhi = Zhonghua kouqiang yixue zazhi = Chinese journal of stomatology 2008
Show all 2 Pubmed References
Human Monoclonal MSX1 Primary Antibody for ELISA, WB - ABIN966599
Zhonghua, Chunpin, Yong, Kezhi, Yaou: Cloning and bioactivity analysis of a CXC ligand in black seabream Acanthopagrus schlegeli: the evolutionary clues of ELR+CXC chemokines. in BMC immunology 2008
Show all 3 Pubmed References
Human Polyclonal MSX1 Primary Antibody for IF (cc), IF (p) - ABIN1385926
Mu, Wu, Zhu, Li, Tang, Yao, Yang, Peng, Li, Hua: The function of Msx1 gene in promoting meiosis of dairy goat male germline stem cells (mGSCs). in Cell biochemistry and function 2013
Human Polyclonal MSX1 Primary Antibody for IHC (p), IHC - ABIN268531
Blin-Wakkach, Lezot, Ghoul-Mazgar, Hotton, Monteiro, Teillaud, Pibouin, Orestes-Cardoso, Papagerakis, Macdougall, Robert, Berdal: Endogenous Msx1 antisense transcript: in vivo and in vitro evidences, structure, and potential involvement in skeleton development in mammals. in Proceedings of the National Academy of Sciences of the United States of America 2001
Cow (Bovine) Polyclonal MSX1 Primary Antibody for IHC, WB - ABIN2777275
Djoussé, Knowlton, Hayden, Almqvist, Brinkman, Ross, Margolis, Rosenblatt, Durr, Dode, Morrison, Novelletto, Frontali, Trent, McCusker, Gómez-Tortosa, Mayo Cabrero, Jones, Zanko, Nance, Abramson, Suchowersky, Paulsen, Harrison, Yang, Cupples, Mysore, Guse: Evidence for a modifier of onset age in Huntington disease linked to the HD gene in 4p16. in Neurogenetics 2004
Show all 2 Pubmed References
Study results suggest that the neural crest-like reprogramming process conferred by MSX1 contributes to the metastatic spread of melanoma.
A novel mutation of MSX1 in oligodontia found in a Chinese pedigree inhibits odontogenesis of dental pulp stem cells via the ERK pathway.
germline variant in MSX1 was identified in a Dutch family with clustering of Barrett's esophagus and esophageal adenocarcinoma
60 child patients (age 13 to 17) with various types of tooth agenesis were examined. Whole gene sequences of PAX9, MSX1, AXIN2, EDA, EDAR and WNT10a genes were sequenced by next generation sequencing on the Illumina MiSeq platform. We found previously undescribed heterozygous nonsense mutation g.8177G>T (c.610G>T) in MSX1 gene in one child.
Gene-gene interaction between MSX1 and TP63 may influence the risk of nonsyndromic cleft lip with or without cleft palate in Asian populations.
MSX1 exerts tumor-suppressive functions by inducing G1/S cell cycle arrest and apoptosis in breast tumorigenesis.
In male infants, there was no association between the mother's homozygous MSX1 p(CA) *4/*4 genotype and nsCL+/-P (RR, 0.98; 95% CI, 0.63-1.54), but this maternal genotype resulted in a doubling of risk for female infants (RR, 2.21; 95% CI, 1.13-4.34).
p19(INK4d) plays an active role during human tooth development along with MSX1 and MSX2
Novel human mutation and CRISPR/Cas genome-edited mice reveal the importance of C-terminal domain of MSX1 in tooth and palate development.
Hypomethylation of CpG sites within the MSX1 gene is associated with resistant high-grade serous ovarian cancer at presentation and identifies expression of MSX1 as conferring platinum drug sensitivity.
MSX1 homeobox protein accumulation is associated with the secretory phase in endometrium of fertile couples, and is widely disrupted in infertile patients. It is the first study to examine MSX1 protein localization in the human endometrium, and supported by genetic findings in mice, suggests that genes regulated by MSX1 are linked to the loss of epithelial cell polarity required for uterine receptivity during implantation
a strong correlation between observed phenotypes and the location in the MSX1 protein structure of the disease causing mutations (review)
this study shows that MSX1 is an important component of RLR-mediated signaling and reveal mechanisms on innate immune responses against RNA viruses
MSX1 and DCLK1 might be used in colorectal cancer detection or as target of cancer therapies.
We found the c.*6C > T variation in the MSX1 gene. This variant in the 3' untranslated region is located 6 bp downstream of the stop codon (TAG) in exon 2. We revealed a statistically significant association between the MSX1 c.*6C > T variant and Nonsyndromic cleft lip/palate in Turkey ( p = 0.01).
The results of this study suggest an association between CL/P susceptibility and SNP1, located near the MSX1 gene, in the Mexican population.
Our study showed that TGFA/TGFB3/MSX1 gene polymorphisms were associated with congenital NSHI. The distribution of genotype frequencies and allele frequencies of TGFA rs3771494, TGFB3 rs3917201 and rs2268626, and MSX1 rs3821949 and rs62636562 were significantly different between the case and the control groups
MSX1 was able to inhibit the Wnt/beta-catenin signaling pathway, and that the ability to regulate the Wnt/beta-catenin signaling pathway is critical for MSX1 to suppress glioblastoma cell migration and invasion.
mutations in MSX1 gene might play an important role in hypodontia cases involving pre-molars and is a risk factor for this ethnic population mainly of Arabs and is first report linking these mutations with tooth agenesis.
Statistically significant relations were found between detected 7 variations in MSX1gene and tooth size and were detected in which 5 of them are novel and one of them lead to amino acid change.
Msx1 may play a role in repressing gene expression in the fetal and postnatal periods.
Findings indicate that nuclear translocation of MSX-1, alone or in combination with HES-1, produce chondrocyte-like cells, and simultaneous activation of HES-1 and MSX-1 increases the generation of smooth muscle and neuronal cells.
We further determined that Prmt1 depletion resulted in significant downregulation of Msx1 in calvaria-derived preosteoblast and primordium of frontal bone and mandible. Our study reveals critical roles of PRMT1 in the formation of CNC-derived craniofacial bones and suggests that Prmt1 is an upstream regulator of Msx1 in craniofacial bone development
Full digit tip was regenerated only in the MSX1/2 group and was related to boosted Bmp4, Fgf8, and K14 gene expression and to limb-patterning properties resulting from Msx1 and Msx2 overexpression.
endocytic adaptor protein Numb regulates the balance between neural and non-neural fates in Msx1 progenitors.
these data reveal a novel mechanism that the Bmp4-Msx1 pathway and Osr2 control tooth organogenesis through antagonistic regulation of expression of secreted Wnt antagonists.
Msx1 and Msx2 are expressed both in Primordial germ cell precursors and in neighboring somatic cells. Msx1(-/-); Msx2(-/-) mutant embryos have defects in Primordial germ cell migration as well as a reduced number of Primordial germ cells.
Functional analysis of each TE revealed step-by-step retroposition/transposition and co-option together with acquisition of a binding site for Msx1 for its full enhancer function during mammalian evolution.
Data show that the spatiotemporal expression of claudin-1 is dysregulated in homeobox (Msx) genes Msx1d/d/Msx2d/d uteri.
MSX1 linked the arterial shear stimulus to arteriogenic remodeling by activating the endothelial but not medial layer to a proinflammatory state because EC but not smooth muscle cell
findings highlight an unexpected role of uterine Msx in limiting aberrant inflammatory responses to maintain embryonic diapause
Msx1 and Msx2 are direct transcriptional targets of Lhx2.
Msx1 appears to act as a modeling factor for membranous bone.
analysis of a BMP signaling pathway that regulates the expression of the odontogenic gene Msx1 and determines the fate of dental mesenchyme during early tooth development
Msx1 supports vascular mineralization by directing the osteogenic programming of aortic progenitors in diabetic arteriosclerosis.
Msx1 and Msx2 proteins activate Atoh1 transcription by specifically interacting with several homeodomain binding sites in the Atoh1 3' enhancer.
Study concludes that MSX1 may promote proliferation and prevent the differentiation of dental mesenchymal cells by the inhibition of Bmp2 and Bmp4 expression.
Msx1 interacts with Lhx6 and Sna in vivo. A network of transcription factors operates during early tooth morphogenesis.
Msx1 functions as a negative regulator early in pituitary development by repressing the gonadotrope-specific alphaGSU and GnRHR genes.
Msx1 and Tbx2 antagonistically regulate Bmp4 expression during the bud-to-cap stage transition in tooth development.
Suppression of MSX1 affects oocyte maturation, embryo cleavage rate, and expression of numerous genes suggesting important role for MSX1 in embryo development.
This gene encodes a member of the muscle segment homeobox gene family. The encoded protein functions as a transcriptional repressor during embryogenesis through interactions with components of the core transcription complex and other homeoproteins. It may also have roles in limb-pattern formation, craniofacial development, particularly odontogenesis, and tumor growth inhibition. Mutations in this gene, which was once known as homeobox 7, have been associated with nonsyndromic cleft lip with or without cleft palate 5, Witkop syndrome, Wolf-Hirschom syndrome, and autosomoal dominant hypodontia.
, homeobox protein Hox-7
, homeobox protein MSX-1
, msh homeo box 1
, msh homeobox 1-like protein
, msh homeobox homolog 1
, homeo box, msh-like 1
, homeobox, msh-like 1
, muscle-segment homeobox
, msh homeo box homolog 1
, Hbox 7
, homeobox protein GHOX-7