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anti-Human s100b Antibodies:
anti-Rat (Rattus) s100b Antibodies:
anti-Mouse (Murine) s100b Antibodies:
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Human Monoclonal s100b Primary Antibody for IHC (p) - ABIN3043667
Huang, Zhu, Zhang, Zhu, Liu, Zhu, Wang, Li, Yang, Dong, Liu, Chen, Zhang, Yang, Deng, Fan, Wang, Liu, Ma, Fu, Wu: S100+ cells: a new neuro-immune cross-talkers in lymph organs. in Scientific reports 2013
Show all 26 Pubmed References
Human Polyclonal s100b Primary Antibody for ICC, IF - ABIN4351813
Chaichana, Guerrero-Cazares, Capilla-Gonzalez, Zamora-Berridi, Achanta, Gonzalez-Perez, Jallo, Garcia-Verdugo, Quiñones-Hinojosa: Intra-operatively obtained human tissue: protocols and techniques for the study of neural stem cells. in Journal of neuroscience methods 2009
Show all 6 Pubmed References
Human Monoclonal s100b Primary Antibody for IHC, ELISA - ABIN969391
Sorci, Riuzzi, Arcuri, Giambanco, Donato: Amphoterin stimulates myogenesis and counteracts the antimyogenic factors basic fibroblast growth factor and S100B via RAGE binding. in Molecular and cellular biology 2004
Show all 3 Pubmed References
Human Polyclonal s100b Primary Antibody for WB - ABIN3044359
Liu, Zhang, Zhao, Cui, Cao, Guo: Effects of hypothermia on S100B and glial fibrillary acidic protein in asphyxia rats after cardiopulmonary resuscitation. in Cell biochemistry and biophysics 2015
Show all 3 Pubmed References
Human Monoclonal s100b Primary Antibody for ICC, IF - ABIN448390
Zhang, Freitas, Qian, Lux, Acab, Trujillo, Herai, Nguyen Huu, Wen, Joshi-Barr, Karpiak, Engler, Fu, Muotri, Almutairi: Layered hydrogels accelerate iPSC-derived neuronal maturation and reveal migration defects caused by MeCP2 dysfunction. in Proceedings of the National Academy of Sciences of the United States of America 2016
Show all 2 Pubmed References
Human Monoclonal s100b Primary Antibody for FACS, ICC - ABIN4351815
Liao, Tang, Lin, Liang Hsieh: Long-term electrical stimulation at ear and electro-acupuncture at ST36-ST37 attenuated COX-2 in the CA1 of hippocampus in kainic acid-induced epileptic seizure rats. in Scientific reports 2017
Human Monoclonal s100b Primary Antibody for IHC, ELISA - ABIN966987
Shapiro, Marks, Whitaker-Azmitia: Increased clusterin expression in old but not young adult S100B transgenic mice: evidence of neuropathological aging in a model of Down Syndrome. in Brain research 2004
Show all 3 Pubmed References
Human Polyclonal s100b Primary Antibody for IF (p), IHC (p) - ABIN676703
Zhang, Li, Lu, Liu et al.: Cerebral potential biomarkers discovery and metabolic pathways analysis of ?-synucleinopathies and the dual effects of Acanthopanax senticosus Harms on central nervous system through metabolomics ... in Journal of ethnopharmacology 2015
Human Polyclonal s100b Primary Antibody for CM, ICC - ABIN2747403
Ito, Minamiya, Kawai, Saito, Saito, Nakagawa, Imai, Hirokawa, Ogawa: Tumor-derived TGFbeta-1 induces dendritic cell apoptosis in the sentinel lymph node. in Journal of immunology (Baltimore, Md. : 1950) 2006
In this study demonstrated that S100b is elevated in Alzheimer disease cases. and the increased levels in African Americans here may be indicative of increased severity in specific populations.
In neonates, NSE (show ENO2 Antibodies) and s100B levels increase after bypass surgery and return below preoperative baseline levels by postoperative day seven. The levels of s100B were positively correlated with circulatory arrest time and negatively correlated with age at time of surgery.
found that S100B plays a crucial role in blocking the interaction site between RAGE (show AGER Antibodies) V domain and S100A1 (show S100A1 Antibodies). A cell proliferation assay WST (show EEF1A2 Antibodies)-1 also supported our results. This report could potentially be useful for new protein development for cancer treatment
the serum levels of S100B protein mediated the association between S100B gene polymorphism and scene selectivity in the retrosplenial cortex
There is a significant correlation between mortality in the critically ill patients in the intensive care unit and increased serum concentration of S100B and NSE (show ENO2 Antibodies).
Our findings showed that both S100beta and NSE (show ENO2 Antibodies) levels similarly increased during CPB (show CPB1 Antibodies) and immediately after CPB (show CPB1 Antibodies) during sevoflurane and propofol based anesthesia.
This study demonstrated that S100A12 (show S100A12 Antibodies) mRNA levels were significantly decreased in the new cases of untreated MS patients in comparison to healthy controls.
Increased serum levels of S100B protein (and NSE (show ENO2 Antibodies)) were observed postoperatively in patients with postoperative cognitive dysfunction.
high S100B expression in Multiple Sclerosis (MS) patient samples suggests its usefulness as a diagnostic biomarker for MS.
the underlying mechanism of S100B-mediated effects on cancer stem-like cell stemness was not dependent on its binding with a receptor for advanced glycation end products (RAGE (show AGER Antibodies)), but might be through intracellular regulation, through the inhibition of p53 (show TP53 Antibodies) expression and phosphorylation.
Data (including data from studies in knockout mice) suggest that S100b acting as a humoral factor impairs glycolysis in muscle (myoblasts, myotubes, and skeletal muscles) independent of insulin (show INS Antibodies) action; this effect appears to be due to inhibition of Gapdh (show GAPDH Antibodies) activity from enhanced poly(ADP-ribosyl)ation of Gapdh (show GAPDH Antibodies). (S100B = S100 protein, beta polypeptide (show MS4A2 Antibodies), neural; Gapdh (show GAPDH Antibodies) = glyceraldehyde-3-phosphate dehydrogenase (show GAPDH Antibodies))
S100B inhibits C3H/10T1/2 murine embryonic mesenchyma.l cells into osteoblasts. S100B stimulates C3H/10T1/2 cell differentiation into adipocytes.
The results of this study showed that S100B affects behavioral despair in female mice through functional interaction with the 5-HT7 receptor.
Data show that S100B has direct effects on macrophages, enhancing particularly CCL22 (show CCL22 Antibodies) and IL-1beta (show IL1B Antibodies) expression and modulates the inflammatory response in uveoretinitis and this is likely to be, at least in part, via a direct effect on macrophages.
Data show that high glucose increased protein-protein interaction between Steap4 (show STEAP4 Antibodies) and S100B in mesangial (MES13) cells.
high glucoseinduced profibrotic genes (TGFbeta (show TGFB1 Antibodies), type IV collagen (show COL4 Antibodies) and fibronectin (show FN1 Antibodies)) and cell hypertrophyrelated p21WAF1 are dependent on S100B.
S100A1 (show S100A1 Antibodies) and S100B are dispensable for endochondral ossification during skeletal development.
Data suggest up-regulation of S100b/RAGE (show AGER Antibodies) (advanced glycosylation end-product receptor) signaling plays role in inflammatory interaction between adipocytes/macrophages; adipocyte secretion of S100b is up-regulated by Tnf (tumor necrosis factor-alpha (show TNF Antibodies)).
Gioma production of S100B enhancestumor growth through CCL2 (show CCL2 Antibodies) upregulation and tumor-associated macrophages chemoattraction.
HMGB1 (show HMGB1 Antibodies), S100B, and RAGE (show AGER Antibodies) signaling modulate the hippocampal inflammatory response and might play key roles in surgery-induced cognitive decline.
As CSF (show CSF2 Antibodies)-S100B levels in calves with neurologic diseases widely differed, the utility of CSF (show CSF2 Antibodies)-S100B as a diagnostic marker for neurologic diseases in cattle remains inconclusive.
S100B might participate in the pathophysiology of brain inflammatory disorders via RAGE (show AGER Antibodies)-dependent regulation of several inflammation-related events including activation and migration of microglia
X-ray crystallography was used here to characterize an interaction between Ca(2 (show CA2 Antibodies))(+)-S100B and TRTK-12, a target that binds to the p53 (show TP53 Antibodies)-binding site on S100B.
Intracellular S100B might modulate myoblast differentiation by interfering with MyoD (show MYOD1 Antibodies) expression in an NF-kappaB (show NFKB1 Antibodies)-dependent manner.
S100b activates guanylate cyclase in a calcium-dependent manner [review]
Structural studies in combination with biochemical data are used to develop a model for calcium-induced activation of human nuclear serine/threonine kinase (NDR (show STK38 Antibodies)) kinase by S100B.
S100B shows a sufficient thermostability to resist pasteurization but not spry-drying in milk formulas for preterm and term infants.
Structures of pentamidine (Pnt (show ETS2 Antibodies)) bound to Ca(2 (show CA2 Antibodies)+)-loaded and Zn(2+),Ca(2 (show CA2 Antibodies)+)-loaded S100B were determined by X-ray crystallography at 2.15 A (R(free)=0.266) and 1.85 A (R(free)=0.243) resolution, respectively.
The time course of S100B serum values following spinal cord decompression correlates with outcome; the initial degree of paresis is not a prognostic factor to predict outcome.
This study demonstrated that One singular glomerulus (mdG2) exhibits S100 and parvalbumin (show PVALB Antibodies)-positive fibers, apparently originating from all crypt cells plus some microvillous olfactory sensory neuronss.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21\; however, this gene is located at 21q22.3. This protein may function in Neurite extension, proliferation of melanoma cells, stimulation of Ca2+ fluxes, inhibition of PKC-mediated phosphorylation, astrocytosis and axonal proliferation, and inhibition of microtubule assembly. Chromosomal rearrangements and altered expression of this gene have been implicated in several neurological, neoplastic, and other types of diseases, including Alzheimer's disease, Down's syndrome, epilepsy, amyotrophic lateral sclerosis, melanoma, and type I diabetes.
S-100 calcium-binding protein, beta chain
, S-100 protein subunit beta
, S100 calcium-binding protein, beta (neural)
, protein S100-B
, S-100 protein beta chain
, S100 calcium-binding protein B
, S100 calcium-binding protein beta (neural)
, S100 protein, beta polypeptide, neural
, S100 protein, beta polypeptide
, S100 calcium binding protein, beta (neural)
, S100 calcium-binding protein, beta
, S-100 calcium-binding protein beta subunit