Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Mouse (Murine) Ephrin B3 Antibodies:
anti-Rat (Rattus) Ephrin B3 Antibodies:
anti-Human Ephrin B3 Antibodies:
Go to our pre-filtered search.
EphA2 (show EPHA2 Antibodies) mAb treatment could partially inhibit LPSinduced inactivation of EphBEphrin B3 signalling, while Ephrin B3 overexpression could abrogate LPSinduced activation of EphA2Ephrin A1 signalling. EphB1 (show EPHB1 Antibodies)/Ephrin B3 signalling may antagonise the EphA2 (show EPHA2 Antibodies)/Ephrin A1dependent pathway following LPS (show TLR4 Antibodies) treatment
Data suggest that molecules in the erythropoietin (show EPO Antibodies)-producing hepatocellular receptor B family (EPHB) / ephrinB (EFNB) signalling pathways, specifically ephrin B3 and GRIP1 (show NCOA2 Antibodies), are involved blood pressure regulation.
Data reveal that ephrinB2 (show EFNB2 Antibodies) and ephrinB3 signaling is required to control progenitor identities in the ventral spinal cord.
an autonomous receptor-like role for ephrin-B reverse signaling in the tangential migration of interneurons into the neocortex using ephrin-B (EfnB1 (show EFNB1 Antibodies)/B2/B3) conditional triple mutant (TM(lz)) mice, is reported.
ephrin B3/EphB2 (show EPHB2 Antibodies) are obvious candidates for driving the Syk (show SYK Antibodies)-dependent repulsive response.
The Eph (show EPHA1 Antibodies)/ephrin-B3-mediated heterotypic or homotypic cell interactions between thymocytes and thymic epithelial cells (TECs), or between TECs and themselves, contribute to the early maturation of MTS20(+) TECs.
The morphological and behavioural abnormalities in ephrin-B3 mutant mice are rescued by conditional knock-in of wild-type ephrin-B3 during the critical period when axon targeting and fear responses are initiated.
ephrin-B3 specifies the synaptic localization of PSD-95 (show DLG4 Antibodies) and likely links the synaptic stability of PSD-95 (show DLG4 Antibodies) to changes in neuronal activity
Ephrin-B3 knockout mice had significantly reduced pre-pulse inhibition compared with controls.
Conclude that EphB3 (show EPHB3 Antibodies) mediates cell death in the adult cortex through a novel dependence receptor-mediated cell death mechanism in the injured adult cortex and is attenuated following ephrinB3 stimulation.
Data show that Ephrin B3 was concomitantly expressed with EphA2 (show EPHA2 Antibodies) and Ephrin A1 (show EFNA1 Antibodies) with higher Ephrin B3 levels found in non-squamous than in squamous tumors.
Study found up-regulated expression of ephrinB3/EphB3 (show EPHB3 Antibodies) in intractable temporal lobe epilepsy patients and experimental temporal lobe epilepsy rats, which suggested that ephrinB3/EphB3 (show EPHB3 Antibodies) might be involved in the pathogenesis of temporal lobe epilepsy
Data suggest that fusion of Nipah viruses with host cells is facilitated by two of viral membrane proteins, the G protein and the F protein (show HPD Antibodies); G head domain binds to human ephrins B2 and B3 altering conformational density of entire G head domain.
Ephrin-B3 binds to B (show TDO2 Antibodies) lymphocytes, most likely via a non-classical receptor, and induces migration of the memory B cell subpopulation.
Phosphoproteomic profiling of nonsmall cell lung cancer cells reveals that ephrin B3 regulates pro-survival signaling through Akt1 (show AKT1 Antibodies)-mediated phosphorylation of the EphA2 (show EPHA2 Antibodies) receptor.
evidence for an unknown ephrin-B3-binding cell-surface proteoglycan (show Vcan Antibodies) involved in cellular signalling
EphrinB3 is a bona fide alternate receptor for NiV entry, and two residues in the G-H loop of the ephrin B-class ligands are critical determinants of NiV receptor activity.
Immunohistochemistry shows robust staining for phosphorylated ephrin-B and ephrin-B3 in invading glioblastoma cells.
Transgenic EphB1 (show EPHB1 Antibodies) and ephrin-B3 cooperatively regulate the proliferation and migration of neural progenitors in the hippocampus
EFNB3, a member of the ephrin gene family, is important in brain development as well as in its maintenance. Moreover, since levels of EFNB3 expression were particularly high in several forebrain subregions compared to other brain subregions, it may play a pivotal role in forebrain function. The EPH and EPH-related receptors comprise the largest subfamily of receptor protein-tyrosine kinases and have been implicated in mediating developmental events, particularly in the nervous system. EPH Receptors typically have a single kinase domain and an extracellular region containing a Cys-rich domain and 2 fibronectin type III repeats. The ephrin ligands and receptors have been named by the Eph Nomenclature Committee (1997). Based on their structures and sequence relationships, ephrins are divided into the ephrin-A (EFNA) class, which are anchored to the membrane by a glycosylphosphatidylinositol linkage, and the ephrin-B (EFNB) class, which are transmembrane proteins. The Eph family of receptors are similarly divided into 2 groups based on the similarity of their extracellular domain sequences and their affinities for binding ephrin-A and ephrin-B ligands.
, ephrin B3
, ephrin ligand B3
, EPH-related receptor transmembrane ligand ELK-L3
, Ephrin B3
, eph-related receptor tyrosine kinase ligand 8