Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
Human KIT ELISA Kit for Sandwich ELISA - ABIN414919
Suh, Chon, Choi: Protective effects of honokiol against methylglyoxal-induced osteoblast damage. in Chemico-biological interactions 2016
activation of KIT by a gain-of-function, somatic mutation is a novel mechanism of resistance to crizotinib in ROS1 rearranged non-small cell lung cancer
Mutational activation of Kit-, Ras/Raf (show RAF1 ELISA Kits)/Erk (show EPHB2 ELISA Kits)- and Akt (show AKT1 ELISA Kits)- pathways indicate the biological importance of these pathways and their components as potential targets for therapy.
KIT exon 11 codons 557-558 deletion enhanced CXCL12 (show CXCL12 ELISA Kits)-mediated GIST cell migration.
Data show the kinetic behavior of a G-rich sequence located within the c-KIT proximal promoter (kit2) in the presence of monovalent cations K+ and Na+.
Long-term follow-up of patients with metastatic GIST treated with regorafenib suggests particular benefit among patients with primary KIT exon 11 mutations and those with SDH (show SARDH ELISA Kits)-deficient GIST. Dose modifications are frequently required to manage treatment-related toxicities
c-kit-positive cells derived from right atrium tissue were associated with serum BNP
For hot spots in KIT and PDGFRA (show PDGFRA ELISA Kits) genes, 23 out of 146 KIT/PDGFRA (show PDGFRA ELISA Kits) wild-type cases carried mutations according to next-generation sequencing (NGS).
KIT and DNMT1 (show DNMT1 ELISA Kits) co-expression promotes, whereas dual inactivation of them suppresses, lung cancer cell proliferation and metastatic growth in vitro and in vivo, in a synergistic manner.
Data indicate that BRAF (show BRAF ELISA Kits), NRAS (show NRAS ELISA Kits) and C-KIT melanomas constitute distinct clinico-pathological entities.
our data established CD117 as a direct target of miR (show MLXIP ELISA Kits)-34-5p and demonstrated that this regulation interferes with several CD117-mediated effects on osteosarcoma cells
the stem cell gene Kit is regulated inversely from Krt5 (show KRT5 ELISA Kits)/Krt14 (show KRT14 ELISA Kits) by RA signaling
TPO (show THPO ELISA Kits) and its receptor Mpl (show MPL ELISA Kits) are dispensable for platelet survival in adult mice
Artificially applied c-kit(+) cells interact with the target organ endothelium following ischemia reperfusion injury. This interaction seems to depend on TLR-MyD88 (show MYD88 ELISA Kits) signaling.
a critical role for PRL2 (show PTP4A2 ELISA Kits) phosphatase in mediating Notch (show NOTCH1 ELISA Kits) and c-Kit signals in early T cell progenitors, is reported.
These results suggest that CD44 (show CD44 ELISA Kits)(+)CD117(+) T cells are stem cells and a specific T-cell phenotype that initially develops in the thymus, but they do not progress through DN3 and DN4 stages, lack a DP stage, and potently suppress T-cell proliferation and modulate the CTLA-4 (show CTLA4 ELISA Kits) pathway.
we studied the efficacy of ACK2, an antibody that blocks KIT, followed by low-dose irradiation as a preconditioning regimen for hematopoietic cell transplantation using a murine model of Mucopolysaccharidosis type II.
C-kit-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 (show NOX1 ELISA Kits) and catalase (show CAT ELISA Kits), but less of lactoperoxidase (show LPO ELISA Kits) than in matured mononuclear cells.
c-KIT signaling regulates self-renewal capacity and prevents neurodifferentiation in culture.
These findings identify functional redundancy among Kit-dependent hematopoietic lineages and establish an unanticipated capacity of megakaryocytes to mediate IL-1 (show IL1A ELISA Kits)-driven systemic inflammatory disease.
WNT (show WNT2 ELISA Kits) signaling at an early stage (E12 (show ELSPBP1 ELISA Kits)-E15) of submandibular salivary gland (SMG (show SNRPG ELISA Kits)) development inhibits end bud morphogenesis and differentiation into proacini by suppressing Kit expression.
FGF7 (show FGF7 ELISA Kits) may be an important regulator for oocyte growth and its action is mediated via the KIT/KITLG (show KITLG ELISA Kits) signaling pathway.
mRNA expression of c-kit and SCF was decreased in gallbladder tissues in the HCD group. Consistent with the findings of RT-PCR, a lower expression level of c-kit and SCF protein was also observed in HCD animals.
Delayed enrichment for c-kit and inducing cardiac differentiation attenuated protective effects of BMSCs' transplantation in pig model of acute myocardial infarction.
Pravastatin improves function in hibernating myocardium by mobilizing CD133+ and cKit+ bone marrow progenitor cells and promoting myocytes to reenter the growth phase of the cardiac cell cycle.
c-kit is primarily expressed in the spermatogonia and spermatocytes of goat testes.
This gene encodes the human homolog of the proto-oncogene c-kit. C-kit was first identified as the cellular homolog of the feline sarcoma viral oncogene v-kit. This protein is a type 3 transmembrane receptor for MGF (mast cell growth factor, also known as stem cell factor). Mutations in this gene are associated with gastrointestinal stromal tumors, mast cell disease, acute myelogenous lukemia, and piebaldism. Multiple transcript variants encoding different isoforms have been found for this gene.
mast/stem cell growth factor receptor Kit
, p145 c-kit
, piebald trait protein
, proto-oncogene c-Kit
, proto-oncogene tyrosine-protein kinase Kit
, soluble KIT variant 1
, tyrosine-protein kinase Kit
, v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene-like protein
, Dominant white spotting
, Steel Factor Receptor
, c-kit proto-oncogene protein
, dominant spotting
, spotted sterile male
, c-kit receptor tyrosine kinase
, mast/stem cell growth factor receptor
, protein kinase
, c-kit receptor
, mast cell growth factor receptor
, c-KIT gene1
, receptor tyrosine kinase c-kit
, Mast/stem cell growth factor receptor
, Proto-oncogene c-Kit
, Tyrosine-protein kinase Kit
, caprine c-kit protein