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High levels of GLUT1 are associated with Lung Adenocarcinoma.
Glucose transporter type 1 deficiency syndrome is the result of impaired glucose transport into the brain. Patients with glucose transporter type 1 syndrome may present with infantile seizures, developmental delay, acquired microcephaly, spasticity and ataxia.
The results demonstrated the high frequency of C allele of SLC2A1 HaeIII in Kurdish patients with diabetic nephropathy. It was also found that this polymorphism is a significant risk factor for diabetic nephropathy. The effect of this polymorphism on clinical and laboratory characteristics of diabetic nephropathy patients was significant.
Expression of GLUT1 is stimulated by hyperglycemia and low oxygen supply, and this overexpression was associated with increased activity of GLUT1 in the cell membrane that contributes to the impairment of the RPE (show RPE Proteins) secretory function of PEDF (show SERPINF1 Proteins).
A heterozygous SLC2A1 mutation in the severely affected child was inherited from his less severely affected mother who was mosaic for the mutation
UCP2 stimulates hnRNPA2/B1, GLUT1 and PKM2 expression and sensitizes pancreatic cancer cells to glycolysis inhibition.
ablation of Glut1 attenuated apoptosis and increased drug resistance via upregulation of p-Akt (show AKT1 Proteins)/p-GSK-3beta (Ser9)/beta-catenin (show CTNNB1 Proteins)/survivin (show BIRC5 Proteins).
Data show that SALL4 (show SALL4 Proteins) promotes the expression of Glut1 and open chromatin through a HP1alpha (show CBX5 Proteins)-dependent mechanism.
Results show that PPARalpha (show PPARA Proteins) directly targeted the consensus PPRE motif of Glut1 promoter region resulting in Glut1 transcription repression leding to decreased influx of glucose in cancer cells.
Strong GLUT1 staining was inversely associated with circulating levels of fasting glucose in high grade serous ovarian cancer.
Immunoreactivity of vGluT1 in continuous theta-burst stimulation (iTBS; cTBS (show CTBS Proteins)) repeated session (RS) decreased, while GLT-1 (show SLC1A2 Proteins) increased in cTBS (show CTBS Proteins) SS and cTBS (show CTBS Proteins) RS, compared to control
GLUT1 may play an important role in Prostate Cancer progression via mediating glycolysis and proliferation. There is potential crosstalk between GLUT1-mediated glycolysis and androgen sensitivity in Prostate Cancer.
ARAP2 knockdown did not affect fatty acid uptake but reduced basal glucose uptake, total levels of the glucose transporter GLUT1, and GLUT1 levels in the plasma membrane and the lipid micro-domain fraction.
TBC1D5 (show TBC1D5 Proteins) shuttling to autophagosomes during metabolic stress facilitates retromer-dependent GLUT1 trafficking.
inhibition of GLUT1 activity and/or expression is shown to impair TGF-beta (show TGFB1 Proteins)-driven fibrogenic processes, including cell proliferation and production of profibrotic mediators
B cell leukemia-induced inhibition of T cell Akt (show AKT1 Proteins)/mTORC1 signaling and glucose metabolism drives T cell dysfunction; metabolic defects included reduced Akt (show AKT1 Proteins)/mammalian target of rapamycin (show FRAP1 Proteins) complex 1 (mTORC1) signaling, decreased expression of the glucose transporter Glut1 and hexokinase 2 (show HK2 Proteins), and reduced glucose uptake
This study demonstrates a strict requirement for GLUT1 in the early stages of mammary tumorigenesis in vitro and in vivo.
GLUT1-dependent glycolysis regulates fibrogenesis in aged lung.
Data (including data from studies using transgenic mice) suggest that Glut1 (glucose transporter type 1) is a critical downstream target of Hif1a (hypoxia-inducible factor 1 (show HIF1A Proteins), alpha subunit (show POLG Proteins)) mediating hyperglycemia-induced extracellular matrix accumulation in kidney via regulation of Nox4 (show NOX4 Proteins) (NADPH oxidase (show NOX1 Proteins) type 4) expression in nephropathy due to diabetes type 1.
Glut-1 expression globally depended on histological subtypes and the staining patterns (diffuse or zonal) were different between thymic carcinomas and type B3 thymomas
Glut-1 glucose transporter expression in esophageal squamous cell carcinoma is associated with tumor aggressiveness.
Glucose transporter 1 transcript levels were higher in the right ventricle than the left ventricle.
pGlcT, together with MEX1, contributes significantly to the export of starch degradation products from chloroplasts in A. thaliana leaves and and that this starch-mediated pathway for photoassimilate export via pGlcT and MEX1 is essential for the growth and development of A. thaliana. [pGlcT]
Low GLUT1 and GLUT3 (show SLC2A3 Proteins) expression in nonclassical monocytes was unaltered during differentiation into macrophages. GLUT4 (show SLC2A4 Proteins) mRNA was only detectable in unstimulated macrophages. Neither monocytes nor macrophages were insulin (show INS Proteins) responsive.
the different conformations of the GLUT-1 transporter in luminal (blood facing) and abluminal (brain facing) membranes of bovine cerebral endothelial cells arise from differential phosphorylation of GLUT-1
Significant increases in GLUT1 gene expression were observed during early lactation.
Hyperthermia-induced Hsp90 (show HSP90 Proteins).eNOS (show NOS3 Proteins) preserves mitochondrial respiration in hyperglycemic endothelial cells by down-regulating Glut-1 and up-regulating G6PD (show G6PD Proteins) activity.
distinct domains of the glucose transporter GLUT1 mediate HTLV envelope binding and virus entry
Expression of GLUT1 was evaluated in LLC-PK1 cells grown on porous membranes for the development of an artificial kidney.
results suggest that glucose is transported to the axonal cleft intracytoplasmically and delivered to the cleft by GLUT1 transporters
This gene encodes a major glucose transporter in the mammalian blood-brain barrier. The encoded protein is found primarily in the cell membrane and on the cell surface, where it can also function as a receptor for human T-cell leukemia virus (HTLV) I and II. Mutations in this gene have been found in a family with paroxysmal exertion-induced dyskinesia.
, glucose transporter type 1, erythrocyte/brain
, hepG2 glucose transporter
, human T-cell leukemia virus (I and II) receptor
, solute carrier family 2, facilitated glucose transporter member 1
, solute carrier family 2, member 1
, Solute carrier family 2 a 1 (facilitated glucose transporter) brain
, Solute carrier family 2, facilitated glucose transporter member 1
, glucose transporter
, glucose transporter 1
, lethal (3) S007412
, solute carrier family 2 (facilitated glucose transporter), member 1
, solute carrier family 2 member 1
, excitatory amino acid transporter 1
, glial glutamate transporter
, glutamate transporter
, glutamate/aspartate transporter
, sodium-dependent glutamate/aspartate transporter 1
, solute carrier family 1, member 3
, glucose transporter protein
, glucose transporter type 1
, solute carrier family 2 (facilitated glucose transporter), member 1 L homeolog
, glucose transport protein