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Glut-1 expression globally depended on histological subtypes and the staining patterns (diffuse or zonal) were different between thymic carcinomas and type B3 thymomas
Glut-1 glucose transporter expression in esophageal squamous cell carcinoma is associated with tumor aggressiveness.
Glucose transporter 1 transcript levels were higher in the right ventricle than the left ventricle.
pGlcT, together with MEX1, contributes significantly to the export of starch degradation products from chloroplasts in A. thaliana leaves and and that this starch-mediated pathway for photoassimilate export via pGlcT and MEX1 is essential for the growth and development of A. thaliana. [pGlcT]
Results show that resistin (show RETN Proteins) down-regulates the transcription of GLUT1 by suppressing the expression of PPARG (show PPARG Proteins), thus causing impaired glucose transportation in endothelial cell layers.
Metabolically active CD4 (show CD4 Proteins)+ T cells expressing Glut1 and OX40 (show TNFRSF4 Proteins) preferentially harbor HIV during in vitro infection.
we found that PPARdelta (show PPARD Proteins) directly regulated neutral amino acid transporter (show SLC6A19 Proteins) SLC1 (show MCHR1 Proteins)-A5 (solute carrier family 1 member 5 (show SLC1A5 Proteins)) and glucose transporter-1 (Glut1) gene transcription, leading to uptake of glucose and amino acid, activation of mTOR (show FRAP1 Proteins) signaling, and tumor progression. In contrast, silence of PPARdelta (show PPARD Proteins) or its antagonist inhibited this event.
SLC2A1/GLUT1 is expressed late in the adenoma-carcinoma sequence during carcinogenesis in intraductal papillary mucinous neoplasms of the pancreas.
Paraoxonase 2 (show PON2 Proteins) facilitates pancreatic ductal cancer growth and metastasis by stimulating GLUT1-mediated glucose transport.
Data show that Prima-1 (show PRIMA1 Proteins) kills hypoxic wt p53 (show TP53 Proteins) KRAS-mutant cells resistant to 3-bromopyruvate (3-BrPA), partly by decreasing GLUT-1 expression.
A de novo 5'-UTR variant in SLC2A1, generating a novel translation initiation codon, severely compromising SLC2A1 function was identified in a GLUT1 deficiency syndrome patient.
High GLUT1 expression is associated with metastasis and epithelial-mesenchymal transition in hepatocellular carcinoma.
High GLUT-1 expression predicted shorter overall survival (OS) in patients with pancreatic cancer, and was was associated with a tumor size of >2 cm and presence of lymph node metastasis.
detected significantly reduced GLUT1 expression only on red blood cells from patients with GLUT1-Deficiency Syndrome.
ARAP2 knockdown did not affect fatty acid uptake but reduced basal glucose uptake, total levels of the glucose transporter GLUT1, and GLUT1 levels in the plasma membrane and the lipid micro-domain fraction.
TBC1D5 (show TBC1D5 Proteins) shuttling to autophagosomes during metabolic stress facilitates retromer-dependent GLUT1 trafficking.
inhibition of GLUT1 activity and/or expression is shown to impair TGF-beta (show TGFB1 Proteins)-driven fibrogenic processes, including cell proliferation and production of profibrotic mediators
B cell leukemia-induced inhibition of T cell Akt (show AKT1 Proteins)/mTORC1 signaling and glucose metabolism drives T cell dysfunction; metabolic defects included reduced Akt (show AKT1 Proteins)/mammalian target of rapamycin (show FRAP1 Proteins) complex 1 (mTORC1) signaling, decreased expression of the glucose transporter Glut1 and hexokinase 2 (show HK2 Proteins), and reduced glucose uptake
This study demonstrates a strict requirement for GLUT1 in the early stages of mammary tumorigenesis in vitro and in vivo.
GLUT1-dependent glycolysis regulates fibrogenesis in aged lung.
Data (including data from studies using transgenic mice) suggest that Glut1 (glucose transporter type 1) is a critical downstream target of Hif1a (hypoxia-inducible factor 1 (show HIF1A Proteins), alpha subunit (show POLG Proteins)) mediating hyperglycemia-induced extracellular matrix accumulation in kidney via regulation of Nox4 (show NOX4 Proteins) (NADPH oxidase (show NOX1 Proteins) type 4) expression in nephropathy due to diabetes type 1.
CRISPR/Cas9-mediated disruption of the Hdac2 gene increased Slc2a1 expression, suggesting that it is one of the responsible histone deacetylases (HDACs). These results confirm that b-OHB is a HDAC inhibitor and show that b-OHB plays an important role in fasting-induced epigenetic activation of a glucose transporter gene in the brain.
Taken together, the data suggest that curcumin binds directly to GLUT1 at a site that overlaps with the cytochalasin B binding site and thereby inhibits glucose transport.
overnutrition during early life induces short-term metabolic disturbances, impairment in heart insulin (show INS Proteins) signaling, up-regulates GLUT-1 (show SLC1A3 Proteins) and switch cardiac fuel preference in juvenile mice
Low GLUT1 and GLUT3 (show SLC2A3 Proteins) expression in nonclassical monocytes was unaltered during differentiation into macrophages. GLUT4 (show SLC2A4 Proteins) mRNA was only detectable in unstimulated macrophages. Neither monocytes nor macrophages were insulin (show INS Proteins) responsive.
the different conformations of the GLUT-1 transporter in luminal (blood facing) and abluminal (brain facing) membranes of bovine cerebral endothelial cells arise from differential phosphorylation of GLUT-1
Significant increases in GLUT1 gene expression were observed during early lactation.
Hyperthermia-induced Hsp90 (show HSP90 Proteins).eNOS (show NOS3 Proteins) preserves mitochondrial respiration in hyperglycemic endothelial cells by down-regulating Glut-1 and up-regulating G6PD (show G6PD Proteins) activity.
distinct domains of the glucose transporter GLUT1 mediate HTLV envelope binding and virus entry
This gene encodes a major glucose transporter in the mammalian blood-brain barrier. The encoded protein is found primarily in the cell membrane and on the cell surface, where it can also function as a receptor for human T-cell leukemia virus (HTLV) I and II. Mutations in this gene have been found in a family with paroxysmal exertion-induced dyskinesia.
, glucose transporter type 1, erythrocyte/brain
, hepG2 glucose transporter
, human T-cell leukemia virus (I and II) receptor
, solute carrier family 2, facilitated glucose transporter member 1
, solute carrier family 2, member 1
, Solute carrier family 2 a 1 (facilitated glucose transporter) brain
, Solute carrier family 2, facilitated glucose transporter member 1
, glucose transporter
, glucose transporter 1
, lethal (3) S007412
, solute carrier family 2 (facilitated glucose transporter), member 1
, solute carrier family 2 member 1
, excitatory amino acid transporter 1
, glial glutamate transporter
, glutamate transporter
, glutamate/aspartate transporter
, sodium-dependent glutamate/aspartate transporter 1
, solute carrier family 1, member 3
, glucose transporter protein
, glucose transporter type 1
, solute carrier family 2 (facilitated glucose transporter), member 1 L homeolog
, glucose transport protein