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rs1497577 not significantly associated with adaptation to subjective responses to alcohol
Switching the intracellular domains of the gamma2 and gamma1 subunits substantiated this inference. Because this region determines post-synaptic localization, we hypothesize that GABAARs containing gamma1 and gamma2 use different mechanisms for synaptic clustering.
A significant GABRG1 genotype influences self-administration of alcohol and GABAergic manipulation.
we genotyped 13 single nucleotide polymorphisms spanning GABRG1 and GABRA2 in African Americans with alcohol dependence
An intronic single nucleotide polymorphism (SNP) (rs17536211) in GABRG1 shows the strongest statistical evidence of association of recovery from eating disorders.
A single nucleotide polymorphism of the GABRG1 gene (rs1497571) is associated with level of response to alcohol and drinking patterns.
Logistic regression analysis indicated that genetic elements in the GABRG1 haplotype block likely contribute to alcohol dependence risk in an additive manner.
study of patterns of htSNPs & linkage disequilibrium (LD)in 6 populations; LD extended from most of the GABRA2 gene through the GABRG1 locus in the same GABAA cluster region suggesting possible association of these genes with alcohol dependence
results of the present study suggest that hippocampal neurons expressing GABAAgamma receptor subunits resist the progression of neurofibrillary degeneration in Alzheimer disease hippocampus
it is concluded that the anxiolytic-like activities of rubimetide and typical agonist peptides of the FPR2 were mediated successively by the PGD2-DP1 receptor, adenosine-A2A receptor, and GABA-GABAA receptor systems downstream of the FPR2.
A single nucleotide polymorphism (SNP) at the nucleotide position 462 (C/G) in the b1 sequence of the GABAA reseptor was found. This SNP was observed in 5 mice from the sensitive strain and in 36 mice from the resistant strain. The Fischer exact test was used to compare two strains of mice for SNP in the cDNA sequence of the b1 subunit
This study demonstrated that chronic GABAAalpha1 deficiency remodels cortical synapses and suggest that neuroplasticity within the healthy oPFC gates the influence of reward-related contextual stimuli.
Hypnotic activity of melatonin may be mediated through picrotoxin site on GABAA receptor.
deficits in GABAergic inhibition cause behavioral and endocrine abnormalities and selective antidepressant drug responsiveness indicative of anxious-depressive disorders such as melancholic depression.
Prostaglandin E2 attenuates preoptic expression of GABAA receptors via EP3 receptors
A mutated axJ gene locus, encoding the ubiquitin-specific protease 14 (Usp14), negatively influences GABAA receptor turnover.
The protein encoded by this gene belongs to the ligand-gated ionic channel family. It is an integral membrane protein and plays an important role in inhibiting neurotransmission by binding to the benzodiazepine receptor and opening an integral chloride channel. This gene is clustered with three other family members on chromosome 4.
gamma-aminobutyric acid A receptor, gamma 1
, GABA(A) receptor subunit gamma-1
, gamma-aminobutyric acid receptor subunit gamma-1
, GABA(A) receptor, gamma
, gamma-1 polypeptide
, gamma-aminobutyric acid (GABA-A) receptor, subunit gamma 1