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Human CTNNB1 ELISA Kit for Sandwich ELISA - ABIN832441
Gaudio, Privitera, Battaglia, Torrisi, Sidoti, Pulvirenti, Canzonieri, Tringali, Fiore: Sclerostin levels associated with inhibition of the Wnt/?-catenin signaling and reduced bone turnover in type 2 diabetes mellitus. in The Journal of clinical endocrinology and metabolism 2012
Rat (Rattus) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN431880
Shao, Cai, Sheng, Yin: Role of SDF-1 and Wnt signaling pathway in the myocardial fibrosis of hypertensive rats. in American journal of translational research 2015
Human CTNNB1 ELISA Kit for Sandwich ELISA - ABIN416829
Gupta, Khan, Kumar, Kumar, Sharma: Versican and its associated molecules: potential diagnostic markers for multiple myeloma. in Clinica chimica acta; international journal of clinical chemistry 2015
Mouse (Murine) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN424163
Chen, Feng, Bao, Li, Zhang, Shen, Zhao, Guo, Jing, Lin, Zong: Adverse Effects of Osteocytic Constitutive Activation of ß-Catenin on Bone Strength and Bone Growth. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2015
temporal expression of CTNNB1 mRNA during dermal wound repair in the horse
Cardiomyocyte hypertrophy is blunted with cardiac fibroblast-specific loss of beta-catenin after trans-aortic constriction in vivo.
These results collectively suggest that sustained activation of Wnt (show WNT2 ELISA Kits)/beta-catenin signaling in endothelial cells might be a cause of heart failure through suppressing neuregulin-ErbB (show EGFR ELISA Kits) signaling.
Our findings indicate that H2O2 inhibits NaV1.5 (show SCN5A ELISA Kits) expression by activating the Wnt/b-catenin signaling and beta-catenin interacts with TCF4 (show TCF4 ELISA Kits) to transcriptionally suppress cardiac NaV1.5 (show SCN5A ELISA Kits) expression.
TGF-beta (show TGFB1 ELISA Kits) and beta-catenin crosstalk in proximal tubules may have a role in tubular injury in two models of chronic kidney disease
Results indicate that ablation of epithelial Wnt (show WNT2 ELISA Kits)/beta-catenin signaling affected epididymal epithelial cell proliferation but did not inhibit cell differentiation.
Interactions between the Wnt (show WNT2 ELISA Kits)/beta-catenin and the Kras/ERK (show EPHB2 ELISA Kits)/Foxm1 (show FOXM1 ELISA Kits) pathways are essential to restrict SOX9 (show SOX9 ELISA Kits) expression in basal cells during pulmonary branching morphogenesis
The collective results indicate that the osteoanabolic response to loading can occur on the periosteal surface when beta-cat levels are significantly reduced in Dmp1 (show DMP1 ELISA Kits)-expressing cells.
L. donovani triggered AKT (show AKT1 ELISA Kits) activation to regulate GSK-3beta (show GSK3b ELISA Kits)/beta-catenin/FOXO-1 (show FOXO1 ELISA Kits) axis.
Barx2 (show BARX2 ELISA Kits) and Pax7 (show PAX7 ELISA Kits) regulate the canonical Wnt (show WNT2 ELISA Kits) target gene Axin2 (show AXIN2 ELISA Kits), which mediates critical feedback to terminate the transcriptional response to Wnt (show WNT2 ELISA Kits) signals.
NFkB/beta-catenin signaling pathway plays a key role in the regulation of breast cancer-induced bone cell activity and osteolysis.
Net1-regulated beta-catenin activation plays a crucial role in the dorsal axis formation during zebrafish development.
Wnt (show WNT2 ELISA Kits)/beta-catenin signaling induces expression of col12a1a/b and deposition of Collagen XII, which is necessary for axons to actively navigate the non-neural lesion site environment.
Mechanistically, Hif-3alpha2 binds to beta-catenin and destabilizes the nuclear beta-catenin complex.
Nuclear beta-catenin is an indication of an activated Wnt (show WNT2 ELISA Kits) pathway, therefore suggesting a possible role for Wnt (show WNT2 ELISA Kits) signalling during zebrafish tooth development and replacement.
Findings suggest that microRNA 19b (miR (show MYLIP ELISA Kits)-19b) regulates laterality development and heart looping in embryos by targeting beta-catenin ctnnb1.
Custos binds to beta-catenin in a Wnt (show WNT2 ELISA Kits) responsive manner without affecting its stability, but rather modulates the cytoplasmic to nuclear translocation of beta-catenin.
The role of the beta-catenin mechanosensitive pathway in mesoderm identity has been conserved over the large evolutionary distance separating zebrafish and Drosophila.
The Wnt (show WNT2 ELISA Kits)/beta-catenin signaling pathway establishes neuroanatomical asymmetries and their laterality. (Review)
This study demonstrated that beta-catenin/Wnt (show WNT2 ELISA Kits) signaling is initially required to activate cell-cycle re-entry in Muller glia following injury and that Wnt (show WNT2 ELISA Kits) signaling subsequently controls the fate of the progeny of those cell divisions.
A novel role for Eaf1 and Eaf2 (show EAF2 ELISA Kits) in inhibiting canonical Wnt (show WNT2 ELISA Kits)/beta-catenin signaling, which might form the mechanistic basis for Eaf1 and Eaf2 (show EAF2 ELISA Kits) tumor suppressor activity.
We identified the amphibian leap2 (show LEAP2 ELISA Kits) gene which is highly related to its mammalian orthologues at both structural and sequence levels. The gene is expressed in the embryo mostly in the endoderm-derived tissues. Accordingly it is induced in pluripotent animal cap cells by FGF, activin (show Actbeta ELISA Kits) or a combination of vegT/beta-catenin.
Together, these results indicate that capsaicin inhibits the patterning of the dorso-ventral and anterior-posterior body axes of embryo by repressing PP2A and thereby down-regulating the Wnt (show WNT2 ELISA Kits)/beta-catenin signaling.
maternal Wnt (show WNT2 ELISA Kits)/STOP signaling, but not beta-catenin signaling, has a role in cleavage after fertilization and cell cycle progression
Wnt (show WNT2 ELISA Kits) signalling can benefit from nucleo-cytoplasmic shuttling of APC (show APC ELISA Kits), Axin (show AXIN1 ELISA Kits) and GSK3 (show GSK3b ELISA Kits), although they are in general beta-catenin antagonising proteins.
Phosphoinositide 3-kinase and Rac (show AKT1 ELISA Kits) polarization depend specifically on the N-cadherin (show CDH2 ELISA Kits)-p120 catenin (show CTNND1 ELISA Kits) complex, whereas myosin II light chain and actin filament polarization depend on the N-cadherin (show CDH2 ELISA Kits)-beta-catenin complex.
HERG (show KCNH2 ELISA Kits) channel activity is stimulated by beta-catenin
Zic3 (show ZIC3 ELISA Kits) can suppress Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and control development of the notochord and Spemann's organizer.
Notch (show NOTCH1 ELISA Kits) initially destabilises beta-catenin in a process that does not depend on its phosphorylation by GSK3 (show GSK3b ELISA Kits)
The tyrosine kinase receptor (show KDR ELISA Kits), PTK7 (show PTK7 ELISA Kits), is implicated in beta-catenin-dependent developmental processes.
Data indicate cross-talks between MYCN (show MYCN ELISA Kits) and beta-catenin signalling, which repress normal beta-catenin mediated transcriptional regulation.
mutations in TERT (show TERT ELISA Kits) promoter and in CTNNB1 gene represent specific cancer signatures in the pathogenesis of viral related HCC (show FAM126A ELISA Kits).
Multiple myeloma that is driven by deregulated iron homeostasis and/or Pyk2 (show PTK2B ELISA Kits)/beta-cateninn signaling is susceptible to deferasirox-induced apoptosis.
Results indicate that protein disulfide isomerase family 6 (PDIA6) overexpression promoted the proliferation of HeLa cells by suppressing the phosphorylation of beta-catenin, thereby inhibiting the degradation of beta-catenin through the ubiquitin-proteasome pathway.
Results indicate that SFRP1 (show SFRP1 ELISA Kits) rs7832767 C > T, CTNNB1 rs2293303 C > T, and WISP1 (show WISP1 ELISA Kits) rs16893344 C > T were all strongly correlated with myocardial infarction (MI) susceptibility.
Data suggest that casein kinase 2 (CK2 (show CSNK2A1 ELISA Kits)) inhibition is a promising approach to blocking beta-catenin in MPNST cells, although combinatorial therapies may be required for maximal efficacy.
Beta-catenin knockdown inhibited cell proliferation, promoted apoptosis and suppressed cell migration in A549 and H460 cells accompanied by the decreased expression of Myc (show MYC ELISA Kits), PCNA (show PCNA ELISA Kits), VEGF (show VEGFA ELISA Kits), CD44 (show CD44 ELISA Kits), MMP-9 (show MMP9 ELISA Kits), MMP-13 (show MMP13 ELISA Kits) and activated bax (show BAX ELISA Kits)/caspase-3 (show CASP3 ELISA Kits) pathway.
Integrin alphavbeta3 (show ITGAV ELISA Kits) has a role in enhancing beta-catenin signaling in acute myeloid leukemia (show BCL11A ELISA Kits) harboring Fms-like tyrosine kinase-3 (show FLT3 ELISA Kits) internal tandem duplication mutations
lncRNA-BCAT1 (show BCAT1 ELISA Kits) is a tumor suppressor and that lncRNA-BCAT1 (show BCAT1 ELISA Kits) may be an effective prognostic biomarker in Colorectal cancer.
Enhancer of zeste homolog 2 (EZH2 (show EZH2 ELISA Kits)) expression is positively correlated with the expression of Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and negatively correlated with the expression of GSK-3beta (show GSK3b ELISA Kits) and TP53 (show TP53 ELISA Kits) in cervical cancer tissues.
Results highlight a potential role for the beta-catenin/Wnt (show WNT2 ELISA Kits) and Notch (show NOTCH1 ELISA Kits) signalling pathways during the infection of crypt cells by L. intracellularis.
Flutamide exposure led to decreased beta-catenin in corpus luteum of mid/late pregnancy.
beta-catenin signaling is involved in formation of contractile membranes by dedifferentiated retinal pigment epithelium cells.
Wnt3a (show WNT3A ELISA Kits) produced a significant increase in heart valve interstitial cell number at day 4 and in the percentage of BrdU-positive nuclei at 24 h. The increase in proliferation was abolished by beta-catenin siRNA.
beta-catenin controls myocardin (show MYOCD ELISA Kits)-related transcription factor-dependent transcription and emerges as a critical regulator of an array of cytoskeletal genes
beta-Catenin plays a critical role in mediating TGF-beta1 (show TGFB1 ELISA Kits)-induced myofibroblast differentiation in aortic valve interstitial cells.
scratching-induced injury and repair of bronchial epithelial cells may involve inhibition of Glycogen synthase kinase 3beta activity which can lead to activation of the downstream signaling through beta-catenin
Data show that Wnt3a (show WNT3A ELISA Kits) can inhibit the adipogenic differentiation of porcine AMSCs, and suggest that Wnt (show WNT2 ELISA Kits)/beta-catenin signaling inhibits adipogenic differentiation potential and alters the cell fate from adipocytes to osteoblasts.
TNF-alpha (show TNF ELISA Kits) inhibits adipogenesis through stabilization of beta-catenin protein in porcine preadipocytes.
Bovine herpesvirus 1 transiently increased beta-catenin protein levels in bovine kidney cells, but not in rabbit skin cells.
These results demonstrate that activation of AKT (show AKT1 ELISA Kits) is required for gonadotropin regulation of CTNNB1 accumulation and subsequent ovarian E2 production.
beta-catenin, a transcription factor activated by the canonical Wnt signaling pathway, was frequently detected in bovine herpesvirus 1 ORF2-positive trigeminal ganglionic neurons of latently infected, but not mock-infected, calves.
These data demonstrate for the first time that FSH (show BRD2 ELISA Kits) regulates CTNNB1 protein and WNT2 (show WNT2 ELISA Kits) mRNA expressions in bovine granulosa cells, suggesting a potential role of canonical WNT (show WNT2 ELISA Kits) signaling in ovarian steroidogenesis and follicular growth of cattle.
These data provide evidence that testosterone increases cellular beta-catenin content which promotes the expression of beta-catenin-targeted genes and myogenesis in the muscle-derived stem cells of cattle.
that beta-catenin is a plasma membrane-associated protein (show PDZK1IP1 ELISA Kits) in airway smooth muscle that regulates active tension development, presumably by stabilizing cell-cell contacts and thereby supporting force transmission between neighboring cells.
This study demonstrated a relationship between the timing of the development of in vitro-produced bovine embryos and the distribution and localization of the junction protein beta-catenin.
TGF-beta3 (show TGFB3 ELISA Kits) induces the chondrogenic differentiation of pericytes by inducing Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and T-cell factor-induced gene transcription.
cAMP/PKA regulation of GSK3beta (show GSK3b ELISA Kits)/beta-catenin signaling contributes to the increase in progesterone production in corpus luteum.
Palmatin effect on osteoarthritis is likely mediated via the Wnt (show WNT2 ELISA Kits)/beta-catenin and Hedgehog (show SHH ELISA Kits) signaling
These results collectively showed that 2-Deoxy-D-glucose regulates dedifferentiation via beta-catenin pathway in rabbit articular chondrocytes.
The protein encoded by this gene is part of a complex of proteins that constitute adherens junctions (AJs). AJs are necessary for the creation and maintenance of epithelial cell layers by regulating cell growth and adhesion between cells. The encoded protein also anchors the actin cytoskeleton and may be responsible for transmitting the contact inhibition signal that causes cells to stop dividing once the epithelial sheet is complete. Finally, this protein binds to the product of the APC gene, which is mutated in adenomatous polyposis of the colon. Mutations in this gene are a cause of colorectal cancer (CRC), pilomatrixoma (PTR), medulloblastoma (MDB), and ovarian cancer. Three transcript variants encoding the same protein have been found for this gene.
catenin beta 1 subunit
, catenin beta-1
, beta catenin 1
, catenin (cadherin-associated protein), beta 1, 88kDa
, catenin beta-1-like
, catenin (cadherin associated protein), beta 1, 88kDa
, catenin beta
, beta catenin