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TAZ encodes a protein that is expressed at high levels in cardiac and skeletal muscle. Additionally we are shipping TAZ Proteins (13) and TAZ Kits (3) and many more products for this protein.
Showing 10 out of 134 products:
Human Monoclonal TAZ Primary Antibody for ELISA - ABIN395886
Zimmerman, Cox, Lakdawala, Cirino, Mancini-DiNardo, Clark, Leon, Duffy, White, Baxter, Alaamery, Farwell, Weiss, Seidman, Seidman, Ho, Rehm, Funke: A novel custom resequencing array for dilated cardiomyopathy. in Genetics in medicine : official journal of the American College of Medical Genetics 2010
Show all 5 references for ABIN395886
Human Monoclonal TAZ Primary Antibody for ELISA - ABIN394550
Strakova, Reed, Ihnatovych: Human transcriptional coactivator with PDZ-binding motif (TAZ) is downregulated during decidualization. in Biology of reproduction 2010
Show all 5 references for ABIN394550
Human Polyclonal TAZ Primary Antibody for WB - ABIN871734
Kanai, Marignani, Sarbassova, Yagi, Hall, Donowitz, Hisaminato, Fujiwara, Ito, Cantley, Yaffe: TAZ: a novel transcriptional co-activator regulated by interactions with 14-3-3 and PDZ domain proteins. in The EMBO journal 2001
Show all 4 references for ABIN871734
Human Monoclonal TAZ Primary Antibody for BI, WB - ABIN967665
Hong, Hwang, McManus, Amsterdam, Tian, Kalmukova, Mueller, Benjamin, Spiegelman, Sharp, Hopkins, Yaffe: TAZ, a transcriptional modulator of mesenchymal stem cell differentiation. in Science (New York, N.Y.) 2005
Show all 3 references for ABIN967665
Human Polyclonal TAZ Primary Antibody for EIA, WB - ABIN453847
Gedeon, Wilson, Colley, Sillence, Mulley: X linked fatal infantile cardiomyopathy maps to Xq28 and is possibly allelic to Barth syndrome. in Journal of medical genetics 1995
Show all 2 references for ABIN453847
Dog (Canine) Polyclonal TAZ Primary Antibody for WB - ABIN374615
Brandner, Mick, Frazier, Taylor, Meisinger, Rehling: Taz1, an outer mitochondrial membrane protein, affects stability and assembly of inner membrane protein complexes: implications for Barth Syndrome. in Molecular biology of the cell 2005
Human Polyclonal TAZ Primary Antibody for FACS, IHC (p) - ABIN652751
Bione, DAdamo, Maestrini, Gedeon, Bolhuis, Toniolo: A novel X-linked gene, G4.5. is responsible for Barth syndrome. in Nature genetics 1996
Taz is required in the anteroposterior patterning of the pronephric progenitor domain in the intermediate mesoderm, acting in part by regulating retinoic acid signaling in the pronephric progenitor field in the intermediate mesoderm.
Taz-depleted larvae displayed patterning defects in ventral cranial vessels that correlate with lateral displacement of thyroid follicles.
Yap (show YAP1 Antibodies)/Taz-Tead activity is necessary and sufficient for optic vesicle progenitors to adopt retinal pigment epithelium identity in zebrafish.
When transcriptional coactivators Yap (show YAP1 Antibodies) and Taz were restricted from interacting with Tead transcription factors through expression of a dominant negative transgene, cardiac precursors failed to migrate completely to the midline.
knockdown phenotype demonstrates that abnormal cardiac development, with a linear, nonlooped heart, and hypomorphic tail and eye development proves that tafazzin is essential for overall zebrafish development, especially of the heart.
these data reveal a previously unknown role for TAZ and the Hippo pathway in the progression of aggressive subtypes of endometrial cancer.
two novel and non-identical TAZ gene rearrangements were found in the offspring of a single female carrier of Barth syndrome.
The TAZ play an essential role in amino acid-induced mTORC1 activation, particularly under nutrient-limiting conditions.
factor:TAZ transcriptional co-activator and activation of sE-Cadherin/proto-oncogene (show RAB1A Antibodies) protein HER-2 (show ERBB2 Antibodies) signaling could be potential oncogenic pathways for breast cancer (BC) metastasis.
preS2 upregulates TAZ expression by repressing miRNA-338-3p. TAZ is necessary for preS2-promoted HCC (show FAM126A Antibodies) proliferation and migration.
Actin remodeling confers BRAF (show BRAF Antibodies) inhibitor resistance to melanoma cells through YAP (show YAP1 Antibodies)/TAZ activation
Data suggest that activation of TAZ (tafazzin) inhibits adipogenesis in mesenchymal stem cells; interaction of TAZ and protein phosphatases (PP1A (show PPP1CA Antibodies), PP2A (show PPP2R4 Antibodies)) up-regulates dephosphorylation and transport of TAZ to cell nucleus.
Our findings elucidate the mechanistic basis of the oncogenic properties of TAZ-CAMTA1 (show CAMTA1 Antibodies) fusion
Results revealed that TAZ is a differentially expressed molecule in human hepatocellular carcinoma (HCC (show FAM126A Antibodies)) suggesting it as an important regulator, TAZ might be as a new diagnostic biomarker in HCC (show FAM126A Antibodies).
Suggest that high expression of TAZ plays a significant role in retinoblastoma's aggressiveness, and predicts poor prognosis for patients with retinoblastoma.
These findings uncover important roles for Yap (show YAP1 Antibodies) and Taz in cranial neural crest diversification and development.
The impact of endurance training on the cardiac and skeletal muscle phenotype in young TAZ knock-down mice.
impaired Taz-function with onset at adult age does not enhance susceptibility to ischemia-reperfusion injury.
Study discovered Ythat AP/TAZ are bona fide downstream effectors of the alternative Wnt (show WNT2 Antibodies) signaling pathway.
A novel role for Taz in helping to maintain genome integrity in spermatocyte meiosis and facilitating germ cell differentiation.
Hippo signalling effectors, YAP (show YAP1 Antibodies) and TAZ, promote both the proliferation of intestinal stem/progenitor cells and their differentiation into goblet cells.
Tafazzin deficiency in mouse embryonic fibroblasts also led to impaired oxidative phosphorylation and severe oxidative stress
This study identifies a novel mechanism of TAZ regulation by YAP (show YAP1 Antibodies), which has significant implications for our understanding of Hippo pathway regulation, YAP (show YAP1 Antibodies)-isoform specific signaling, and the role of these proteins in cell proliferation, apoptosis, and tumorigenesis.
YAP (show YAP1 Antibodies)/TAZ regulate transcriptional elongation from the enhancer elements by recruiting the Mediator complex and promoting phenotypes of overgrowth and tumorigenesis.
Expression of TAZ promotes embryonic neural stem cell maintenance by enhancing stemness of neural stem cells during mammalian brain development.
This gene encodes a protein that is expressed at high levels in cardiac and skeletal muscle. Mutations in this gene have been associated with a number of clinical disorders including Barth syndrome, dilated cardiomyopathy (DCM), hypertrophic DCM, endocardial fibroelastosis, and left ventricular noncompaction (LVNC). Multiple transcript variants encoding different isoforms have been described. A long form and a short form of each of these isoforms is produced\; the short form lacks a hydrophobic leader sequence and may exist as a cytoplasmic protein rather than being membrane-bound. Other alternatively spliced transcripts have been described but the full-length nature of all these transcripts is not known.
, protein G4.5
, Barth syndrome)
, endocardial fibroelastosis 2
, tafazzin (cardiomyopathy, dilated 3A (X-linked)
, tafazzin (cardiomyopathy, dilated 3A (X-linked); endocardial fibroelastosis 2; Barth syndrome)