Influenza A Virus H1N1 antibody

Catalog No. ABIN643615

This Goat not_set antibody specifically detects Influenza A Virus H1N1 in IF, IHC (p) and H. It exhibits reactivity toward Influenza A Virus H1N1.

Quick Overview for Influenza A Virus H1N1 antibody (ABIN643615)

Target: Influenza A Virus H1N1

Reactivity: Influenza A Virus H1N1

Host: Goat

Conjugate: Un-conjugated

Application: Immunofluorescence (IF), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Haemagglutination (H)

Product Details

Specificity: DS-PB-01152 is specific for Influenza A virus H1N1 glycoprotein and does not react with Influenza B, RSV, Para 1-3 or Adenovirus. It does not react with HEp-2 cells but may react with some chicken cellular proteins.

Characteristics: Goat Anti-Influenza A H1N1

Purification: Purified

Immunogen: Influenza A, strain USSR (H1N1)

Isotype: IgG

Application Details

Restrictions: For Research Use only

Handling

Format: Liquid

Concentration: IgG concentration 1.0 mg/ml

Buffer: Phosphate buffered saline pH 7.2

Preservative: Sodium azide

Precaution of Use: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Storage: 4 °C

Target Details

Target: Influenza A Virus H1N1

Alternative Name: Influenza A H1N1

Target Type: Influenza Virus

Background: Influenza type A viruses are divided into subtypes based on the antigenic differences of two viral surface proteins, hemagglutinin (H) and neuraminidase (N). On infection of the respiratory tract, the hemagglutinin molecule binds to sialic acid-containing receptors on the epithelial cells resulting in endocytosis. Once the virus has been engulfed, the hemagglutinin allows the viral membrane to fuse with the endosomal membrane. Neuraminidase functions to aid viral release from host cells by cleaving terminal sialic acid residues from carbohydrate moieties on the cell surface. Subtype antigenic variations result from a process known as antigenic drift whereby these surface proteins constantly mutate in order to evade the host immune response. Subtype A (H1N1) was the cause of Spanish flu that killed approximately 500,000 people between 1918-1919.