Browse our anti-PIGV (PIGV) Antibodies

Full name:
anti-Phosphatidylinositol Glycan Anchor Biosynthesis, Class V Antibodies (PIGV)
On are 25 Phosphatidylinositol Glycan Anchor Biosynthesis, Class V (PIGV) Antibodies from 8 different suppliers available. Additionally we are shipping PIGV Proteins (3) and many more products for this protein. A total of 31 PIGV products are currently listed.
B330013B03, D430024F16Rik, GPI-MT-II, HPMRS1, PIG-V, RGD1309526

Most Popular Reactivities for anti-PIGV (PIGV) Antibodies

Select your species and application

anti-Human PIGV Antibodies:

anti-Mouse (Murine) PIGV Antibodies:

anti-Rat (Rattus) PIGV Antibodies:

All available anti-PIGV Antibodies

Go to our pre-filtered search.

Top referenced anti-PIGV Antibodies

  1. Human Polyclonal PIGV Primary Antibody for EIA, WB - ABIN954140 : Krawitz, Schweiger, Rödelsperger, Marcelis, Kölsch, Meisel, Stephani, Kinoshita, Murakami, Bauer, Isau, Fischer, Dahl, Kerick, Hecht, Köhler, Jäger, Grünhagen, de Condor, Doelken, Brunner, Meinecke, Passarge, Thompson, Cole, Horn, Roscioli, Mundlos, Robin: Identity-by-descent filtering of exome sequence data identifies PIGV mutations in hyperphosphatasia mental retardation syndrome. in Nature genetics 2010 (PubMed)
    Show all 4 references for 954140

  2. Cow (Bovine) Polyclonal PIGV Primary Antibody for IHC, WB - ABIN2782920 : Kang, Hong, Ashida, Shishioh, Murakami, Morita, Maeda, Kinoshita: PIG-V involved in transferring the second mannose in glycosylphosphatidylinositol. in The Journal of biological chemistry 2005 (PubMed)

More Antibodies against PIGV Interaction Partners

Human Phosphatidylinositol Glycan Anchor Biosynthesis, Class V (PIGV) interaction partners

  1. Data indicate that mannosyltransferases PIGV mutations are the major cause of hyperphosphatasia-mental retardation syndrome (HPMRS) which displays a broad clinical variability regarding associated malformations and growth patterns.

  2. PIGV is the rate-limiting enzyme in GPI (show GNPDA1 Antibodies) biosynthesis under limited dolicholphosphate mannose availability.

  3. Hyperphosphatasia resulted from secretion of ALP (show ALP Antibodies), a GPI (show GNPDA1 Antibodies)-anchored protein normally expressed on the cell surface, into serum due to PIGV deficiency.

  4. novel compound heterozygous mutations in the PIGV gene c.467G>A and c.1022C>A and a homozygous mutation c.1022C>A in hyperphosphatasia-mental retardation syndrome

  5. PIGV mutations are associated with hyperphosphatasia mental retardation syndrome.

  6. PIG-V is the second mannosyltransferase in GPI anchor biosynthesis (show PIGA Antibodies).

PIGV Antigen Profile

Antigen Summary

This gene encodes a mannosyltransferase enzyme involved in the biosynthesis of glycosylphosphatidylinositol (GPI). GPI is a complex glycolipid that functions as a membrane anchor for many proteins and plays a role in multiple cellular processes including protein sorting and signal transduction. The encoded protein is localized to the endoplasmic reticulum and transfers the second mannose to the GPI backbone. Mutations in this gene are associated with hyperphosphatasia mental retardation syndrome. Alternatively spliced transcript variants have been observed for this gene.

Alternative names and synonyms associated with PIGV

  • phosphatidylinositol glycan anchor biosynthesis, class V (PIGV) antibody
  • phosphatidylinositol glycan anchor biosynthesis, class V (Pigv) antibody
  • B330013B03 antibody
  • D430024F16Rik antibody
  • GPI-MT-II antibody
  • HPMRS1 antibody
  • PIG-V antibody
  • RGD1309526 antibody

Protein level used designations for PIGV

GPI mannosyltransferase 2 , GPI mannosyltransferase II , Ybr004c homolog , dol-P-Man dependent GPI mannosyltransferase , GPI-MT-II , PIG-V , phosphatidylinositol glycan class V , phosphatidylinositol glycan, class V , phosphatidylinositol-glycan biosynthesis class V protein

55650 Homo sapiens
478172 Canis lupus familiaris
519213 Bos taurus
230801 Mus musculus
366478 Rattus norvegicus
Selected quality suppliers for anti-PIGV (PIGV) Antibodies
Did you look for something else?