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Interaction analysis demonstrated that the following genotypes: MS4A14 DI+II, SLC2A DI+II and ABCB 5 CG+GG, were associated with a prothrombin time >/=12 sec and with Recurrent pregnancy loss (RPL) risk
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No significant influence of the three newly identified ABCB5 polymorphisms.
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The ABCB5 activates the NF-kappaB pathway by inhibiting p65 ubiquitination to enhance p65 protein stability.
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Data indicate that the energy metabolism was virtually unaffected by ATP-binding cassette member B5 (ABCB5) expression.
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The expression of ZEB1 in tissues is positively relevant to ABCB5 in breast cancer.
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Findings demonstrated that the expression of ABCB5 was upregulated in ocular surface squamous neoplasia (OSSN) and that elevated expression of ABCB5 may be involved in the pathogenesis of OSSN.
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Human limbal mesenchymal stem cells express ABCB5 and can grow on amniotic membrane.
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ABCB5 has a role in chemoresistance in Merkel Cell Carcinoma
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Results suggest that ATP binding cassette subfamily B member 5 ABCB5/Abcb5 upregulates cellular glutathione levels to protect cells from various poisons.
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ATP-binding cassette member B5 (ABCB5) identifies dermal immunoregulatory cells capable of exerting therapeutic immunoregulatory functions through engagement of programmed cell death 1.
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This is the first study showing the relationship between miR-340-5p and expression of ABCB5 in melanoma cells
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c-MYC confers resistance to 5-fluorouracil through regulating ABCB5 expression in human colon cancer cells.
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ABCB5 alleles alter susceptibility to haloperidol-induced toxicity (HIT) in mouse and humans.
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The resistance of CD133(+) melanoma subpopulation is attributed to the enhanced drug efflux mediated by ATP-binding cassette sub-family B member 5 (ABCB5), since the knockdown of ABCB5 was found to sensitize CD133(+) cells to CAPE.
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In melanoma-initiating cells, ABCB5 controls IL1beta secretion, which serves to maintain slow cycling, chemoresistant cells through an IL1beta/IL8/CXCR1 cytokine signaling circuit.
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Suggest that the helicase HAGE has a key role in the resistance of ABCB5+ malignant melanoma stem cells to IFNalpha treatment by promoting SOCS1 expression.
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ABCB5 and MDR1 gene expression correlate with drug resistance in acute leukemia patients.
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ATP-binding cassette, sub-family B, member 5 (ABCB5) marks limbal stem cells (LSCs) and is required for LSC maintenance, corneal development and repair
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Our results show that ABCB5 is preferentially expressed in the cytotrophoblast layer of placental villi.
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Novel associations of the ABCB5 K115E polymorphism with human pigmentation phenotype and melanoma risk are observed.
