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The protein encoded by AGT3, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. Additionally we are shipping Angiotensin III Kits (14) and and many more products for this protein.
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Urinary angiotensinogen (show AGT Antibodies) and renin (show REN Antibodies) excretion are elevated in CKD patients. Both parameters are negatively associated with eGFR (show EGFR Antibodies) and these associations are independent of urinary albumin (show ALB Antibodies) excretion
Reduced IL-18 (show IL18 Antibodies) serum concentration in children after HUS (show CFH Antibodies) with no difference in its urine concentration may indicate a loss of the protective effects of this cytokine on renal function due to previously occurred HUS (show CFH Antibodies).
Angiotensin II initiates hepatocyte epithelial-mesenchymal transition by activating the NOX-derived H2O2-mediated NLRP3 (show NLRP3 Antibodies) inflammasome/IL-1ss/Smad (show SMAD1 Antibodies) circuit.
present study has demonstrated, for the first time, that high glucose augments AGT (show AGXT Antibodies) in human RPTCs through HNF-5, which provides a potential therapeutic target for diabetic nephropathy
AngII-dependent phosphorylation of LCP1 (show LCP1 Antibodies) in cultured podocytes was mediated by the kinases ERK (show EPHB2 Antibodies), p90 (show CANX Antibodies) ribosomal S6 kinase (show RPS6KB1 Antibodies), PKA, or PKC (show PRRT2 Antibodies). LCP1 (show LCP1 Antibodies) phosphorylation increased filopodia formation.
Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (show AGT Antibodies) and renin (show REN Antibodies) excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Quaternary interactions and supercoiling modulate the cooperative DNA binding of AGT (show AGXT Antibodies).
results show that SNPs in the Hap (show SAFB Antibodies)-I of the hAGT gene promote high-fat diet-induced binding of transcription factors GR, CEBP-beta (show CEBPB Antibodies) and STAT3 (show STAT3 Antibodies), which lead to elevated expression of the hAGT gene in hepatic and adipose tissues
Angiotensinogen (show AGT Antibodies) import and subsequent trafficking to the mitochondria occurs in proximal kidney tubules.
Transgenic mice expressing human AGT (show AGXT Antibodies) in the subfornical organ AGT (show AGXT Antibodies) and possibly ANG I/ANG II (show AGT Antibodies) into the cerebral ventricles.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)