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Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. Additionally we are shipping APOH Kits (80) and APOH Proteins (35) and many more products for this protein.
Showing 10 out of 148 products:
Human Polyclonal APOH Primary Antibody for IHC, IHC (p) - ABIN4281228
Häggmark, Neiman, Drobin, Zwahlen, Uhlén, Nilsson, Schwenk: Classification of protein profiles from antibody microarrays using heat and detergent treatment. in New biotechnology 2012
Show all 2 Pubmed References
Human Polyclonal APOH Primary Antibody for WB - ABIN513421
Mulla, Brosens, Chamley, Giles, Pericleous, Rahman, Joyce, Panda, Paidas, Abrahams: Antiphospholipid antibodies induce a pro-inflammatory response in first trimester trophoblast via the TLR4/MyD88 pathway. in American journal of reproductive immunology (New York, N.Y. : 1989) 2009
Human Polyclonal APOH Primary Antibody for IHC, IHC (p) - ABIN4281227
Kato, Nicholson, Neiman, Rantalainen, Holmes, Barrett, Uhlén, Nilsson, Spector, Schwenk: Variance decomposition of protein profiles from antibody arrays using a longitudinal twin model. in Proteome science 2011
Human Polyclonal APOH Primary Antibody for ELISA, WB - ABIN4281229
Hirose, Williams, Alberts, Furie, Chartash, Jain, Sison, Lahita, Merrill, Cucurull, Gharavi, Sammaritano, Salmon, Hashimoto, Sawada, Chu, Gregersen, Chiorazzi: A role for the polymorphism at position 247 of the beta2-glycoprotein I gene in the generation of anti-beta2-glycoprotein I antibodies in the antiphospholipid syndrome. in Arthritis and rheumatism 1999
A1-A1, which is a soluble analog of ApoER2 (show LRP8 Antibodies) that binds pathological beta2GPI/anti-beta2GPI complexes, has a positive impact on hemodynamics in lupus-prone mice with spontaneous anti-beta2GPI antibodies and hypertension
These results suggested that CRP (show CRP Antibodies)/oxidized low density lipoprotein/beta 2-Glycoprotein I complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (show MAPK14 Antibodies) and NFkappaB signaling pathways.
TLR2 increase the stimulating effect of beta2GP1/anti-beta2GP1 complex on the expression of TNF-alpha (show TNF Antibodies) in mouse peritoneal macrophages
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury.
These results indicate that both NF-kappaB (show NFKB1 Antibodies) and c-Jun/AP-1 (show JUN Antibodies) are involved in regulating anti-beta2GPI-induced expression of prothrombotic and proinflammatory molecules in vivo.
Phospholipid scramblase 1 regulates phosphatidylserine exposure in response to oxygen stress, leading to beta2-glycoprotein I and IgM binding and lipid-mediated, inflammatory responses.
beta2-GPI-D-I-tDCs may offer a novel approach for developing therapy for APS (show SH2B2 Antibodies) patients
Deletion of beta2 GPI accelerates and potentiates the autoimmune phenotype in male BXSB.Yaa mice
Interaction of beta2-glycoprotein I with lipopolysaccharide leads to Toll-like receptor 4 (TLR4 (show TLR4 Antibodies))-dependent activation of macrophages
Data are consistent with pathogenic mechanisms in antiphospholipid syndrome involving both neutralization of beta(2)-glycoprotein I (beta(2)GPI) function and beta(2)GPI-immunoglobulin complex formation.
Data suggest that, in patients with anti-GPIIb/IIIa-mediated immune thrombocytopenia, diminished plasma levels of beta2-GPI are linked to enhanced complement activation.
The aim of this study was to assess clinical utility of the anti-Domain 1 B2GP1 antibodies in the diagnosis and risk stratification of antiphospholipid syndrome. Anti-Domain 1 test does not add accuracy in predicting Antiphospholipid syndrome thrombotic complications on the top of accuracy offered by classic tests.
we have demonstrated a pathogenic role for aPL (show FASL Antibodies) containing samples, mediated via aPL (show FASL Antibodies)-2bGPI interactions, resulting in activation of the pro-apoptotic p38 MAPK (show MAPK14 Antibodies) pathway.
Pretransplant IgA (show IgA Antibodies)-aB2GP1 was the main risk factor for graft thrombosis and early graft loss after renal transplantation.
These results suggest that antibodies directed against domain 1 of beta-2-glycoprotein 1 may contribute to antiphospholipid syndrome risk assessment, due to their association with the diagnosis of systemic autoimmunity and with more aggressive triple antiphospholipid antibody profiles, such as triple positivity and lupus anticoagulant activity.
Circulating Immune Complexes of IgA (show IgA Antibodies) Bound to Beta 2 Glycoprotein are Strongly Associated with the Occurrence of Acute Thrombotic Events.
Genome-wide significant results in or near the APOH gene on chromosome 17 have been found for plasma apolipoprotein H levels in middle-aged and older adults.
Prevalence and pathogenicity of IgA (show IgA Antibodies) aB2GPI antibodies may vary from a population of ESRD patients to another.
The association of IL28B (show IL28B Antibodies) and APOH single nucleotide polymorphisms (SNPs) with sustained virological response and of ITPA (show ITPA Antibodies) SNPs with anemia related phenotypes.
We identified APOH as an obesity-resistance gene that interacts with FTO (show FTO Antibodies) rs9939609 to more than double the occurrence of thinness.
Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.
, apolipoprotein H (beta-2-glycoprotein I)
, APC inhibitor
, activated protein C-binding protein
, beta-2-glycoprotein 1
, beta-2-glycoprotein I
, anticardiolipin cofactor