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Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. Additionally we are shipping and APOH Proteins (38) and many more products for this protein.
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Human APOH ELISA Kit for Sandwich ELISA - ABIN414966
Lackner, Beck, Eichmeir, Gemeiner, Hummel, Pullirsch, Razzazi-Fazeli, Seifner, Miller: Contamination of therapeutic human immunoglobulin preparations with apolipoprotein H (?2-glycoprotein I). in Electrophoresis 2014
Human APOH ELISA Kit for Sandwich ELISA - ABIN2683481
Kim, Park, Heo, Moon, Park: Plasma apolipoprotein H levels are different between aspirin induced respiratory diseases and aspirin tolerant asthma. in Pulmonary pharmacology & therapeutics 2014
During the parasite's hepatic replication, the C-terminal region of EXP-1 binds ApoH. This molecular interaction plays a pivotal role for successful Plasmodium liver-stage development. Down-regulation of ApoH expression in either hepatic cells or mouse livers by RNA interference resulted in impaired intrahepatic development.
A1-A1, which is a soluble analog of ApoER2 (show LRP8 ELISA Kits) that binds pathological beta2GPI/anti-beta2GPI complexes, has a positive impact on hemodynamics in lupus-prone mice with spontaneous anti-beta2GPI antibodies and hypertension
These results suggested that CRP (show CRP ELISA Kits)/oxidized low density lipoprotein/beta 2-Glycoprotein I complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (show MAPK14 ELISA Kits) and NFkappaB signaling pathways.
TLR2 increase the stimulating effect of beta2GP1/anti-beta2GP1 complex on the expression of TNF-alpha (show TNF ELISA Kits) in mouse peritoneal macrophages
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury.
These results indicate that both NF-kappaB (show NFKB1 ELISA Kits) and c-Jun/AP-1 (show JUN ELISA Kits) are involved in regulating anti-beta2GPI-induced expression of prothrombotic and proinflammatory molecules in vivo.
Phospholipid scramblase 1 regulates phosphatidylserine exposure in response to oxygen stress, leading to beta2-glycoprotein I and IgM binding and lipid-mediated, inflammatory responses.
beta2-GPI-D-I-tDCs may offer a novel approach for developing therapy for APS patients
Deletion of beta2 GPI accelerates and potentiates the autoimmune phenotype in male BXSB.Yaa mice
Interaction of beta2-glycoprotein I with lipopolysaccharide leads to Toll-like receptor 4 (TLR4 (show TLR4 ELISA Kits))-dependent activation of macrophages
Data suggest alpha-1-antitrypsin (A1AT (show SERPINA1 ELISA Kits)) and APOH (beta-2-glycoprotein-1) could represent promising markers for renal carcinogenesis in Von Hippel-Lindau disease (VHLD) patients.
Suggest that anti-beta2 GPI antibodies bind to beta2 GPI-PS complexes expressed on microparticles. Consequent loss of beta2 GPI-PS expression on MPs may impair scavenging and contribute to the accumulation of circulating PS-negative MPs, a possible source of autoantigens in systemic lupus erythematosus.
Data suggest that, in patients with anti-GPIIb/IIIa-mediated immune thrombocytopenia, diminished plasma levels of beta2-GPI are linked to enhanced complement activation.
The aim of this study was to assess clinical utility of the anti-Domain 1 B2GP1 antibodies in the diagnosis and risk stratification of antiphospholipid syndrome. Anti-Domain 1 test does not add accuracy in predicting Antiphospholipid syndrome thrombotic complications on the top of accuracy offered by classic tests.
we have demonstrated a pathogenic role for aPL (show FASL ELISA Kits) containing samples, mediated via aPL (show FASL ELISA Kits)-2bGPI interactions, resulting in activation of the pro-apoptotic p38 MAPK (show MAPK14 ELISA Kits) pathway.
Pretransplant IgA (show IgA ELISA Kits)-aB2GP1 was the main risk factor for graft thrombosis and early graft loss after renal transplantation.
These results suggest that antibodies directed against domain 1 of beta-2-glycoprotein 1 may contribute to antiphospholipid syndrome risk assessment, due to their association with the diagnosis of systemic autoimmunity and with more aggressive triple antiphospholipid antibody profiles, such as triple positivity and lupus anticoagulant activity.
Circulating Immune Complexes of IgA (show IgA ELISA Kits) Bound to Beta 2 Glycoprotein are Strongly Associated with the Occurrence of Acute Thrombotic Events.
Genome-wide significant results in or near the APOH gene on chromosome 17 have been found for plasma apolipoprotein H levels in middle-aged and older adults.
Prevalence and pathogenicity of IgA (show IgA ELISA Kits) aB2GPI antibodies may vary from a population of ESRD patients to another.
Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.
, apolipoprotein H (beta-2-glycoprotein I)
, APC inhibitor
, activated protein C-binding protein
, beta-2-glycoprotein 1
, beta-2-glycoprotein I
, anticardiolipin cofactor