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CLCN3 encodes a member of the voltage-gated chloride channel (ClC) family. Additionally we are shipping Chloride Channel 3 Proteins (7) and Chloride Channel 3 Kits (1) and many more products for this protein.
Showing 10 out of 106 products:
Mammalian Monoclonal CLCN3 Primary Antibody for WB - ABIN1304600
Gaurav, Bewtra, Agrawal: Novel CLC3 transcript variants in blood eosinophils and increased CLC3 expression in nasal lavage and blood eosinophils of asthmatics. in Immunity, inflammation and disease 2015
Show all 3 Pubmed References
Cow (Bovine) Polyclonal CLCN3 Primary Antibody for IHC, WB - ABIN2776327
Salazar, Love, Styers, Werner, Peden, Rodriguez, Gearing, Wainer, Faundez: AP-3-dependent mechanisms control the targeting of a chloride channel (ClC-3) in neuronal and non-neuronal cells. in The Journal of biological chemistry 2004
Show all 2 Pubmed References
Rat (Rattus) Polyclonal CLCN3 Primary Antibody for WB - ABIN1742509
Eckenstaler, Lessmann, Brigadski: CAPS1 effects on intragranular pH and regulation of BDNF release from secretory granules in hippocampal neurons. in Journal of cell science 2016
Human Polyclonal CLCN3 Primary Antibody for IF (p), IHC (p) - ABIN1386131
Larrouture, Nelson, Robinson, Liu, Tourkova, Schlesinger, Blair: Chloride-hydrogen antiporters ClC-3 and ClC-5 drive osteoblast mineralization and regulate fine-structure bone patterning in vitro. in Physiological reports 2015
ClC-3 is a potential target of 17beta-estradiol and is modulated by the ERalpha (show ESR1 Antibodies) in breast cancer cells.
SDPR (show SDPR Antibodies) variants were not associated with major depressive disorder.
Overall, these data suggest that Cavin-2 (show SDPR Antibodies) represents a useful marker for discriminating the degree of differentiation in LPS (show IRF6 Antibodies) tumors.
Study provided novel and compelling evidence for the functional role of the unique CLC-3, which are significantly upregulated during ischemia, in the protection of the heart under stress
Data found that CVN2 expression was signi fi cantly down-regulated in oral squamous cell carcinoma (OSCC) in vitro and in vivo and that CVN2 overexpression led to decreased proliferative activities through the CVN2/caveolin-1 (show CAV1 Antibodies)/ERK (show EPHB2 Antibodies) path- way. Furthermore, high tumoral progression occurred in CVN2-negative patients with OSCC.
Hepatocellular carcinoma patients with lower cavin-2 (show SDPR Antibodies) expression have a relatively poor prognosis.
ClC-3 promotes endometriotic cell migration and invasion.
identification of a novel metastasis suppressor gene, serum deprivation response (SDPR (show SDPR Antibodies)), localized to 2q32-33, a region reported to be associated with significant loss of heterozygosity in breast cancer, is reported.
Cavin-1 (show PTRF Antibodies) and cavin-2 (show SDPR Antibodies) are strongly expressed within caveolae-like structures within liver sinusoidal endothelial cells of the hepatitis C-related cirrhotic liver and cavin-1 (show PTRF Antibodies) would play a critical role in regulating aspects of caveolin-1 (show CAV1 Antibodies).
these results demonstrated that ClC-3 is involved in the proliferation and migration of osteosarcoma cell
Results have shown that the expression of ClC-3 was reduced in hypertrophic H9c2 cells, primary rat neonatal cardiomyocytes and myocardium of C57/BL/6 mice and that ClC-3 played an important role in beta-adrenergic cardiac hypertrophy.
we conclude that the expression of ClC-3 chloride channels in osteoblasts helps them respond to PTH (show PTH Antibodies) stimulation, which mediates osteogenic differentiation.
our findings show that Cl(-) channels can be activated by estrogen via ERa on the cell membrane and suggest that the ClC-3 Cl(-) channel may be one of the targets of estrogen in the regulation of osteoblast activity.
ClC-3 is an endogenous inhibitor of neuropathic pain development and down-regulation of ClC-3 contributes to mechanical hypersensitivity.
This study provides a new mechanism by which endophilin A2 (show SH3GL1 Antibodies) regulates ClC-3 channel activity, and sheds light on how ClC-3 is transported to cell membranes to play its critical role as a chloride channel (show CLCA1 Antibodies) in VSMCs function
roles of AQP-3 (show AQP3 Antibodies) in AQP-3 (show AQP3 Antibodies) aquaglyceroporin and ClC-3 chloride channels complex
ClC-3 plays a major role in hyperglycemia induced hippocampal neuronal apoptosis.
Threonine532 phosphorylation in ClC-3 channels is required for angiotensin II-induced Cl(-) current and migration in cultured vascular smooth muscle cells
Data suggest that ClC3/Clcn3 expression is up-regulated by mechanical stimulation (persistent static compression here) in osteoblastic cell line and appears to participate in mechanically sensitive osteogenesis and gene expression regulation.
Our findings demonstrated that ClC-3 deficiency inhibits atherosclerotic lesion development, possibly via suppression of JNK (show MAPK8 Antibodies)/p38 MAPK (show MAPK14 Antibodies) dependent SR-A (show MSR1 Antibodies) expression and foam cell formation.
ClC-3 is specialized in mainly performing incomplete capacitive nontransporting cycles in intracellular membranes.
A local enhancement of CIC (show CIC Antibodies)-3 expression at the leading edge of the wounded epidermis was found to be specific to closing wounds.
This gene encodes a calcium-independent phospholipid-binding protein whose expression increases in serum-starved cells. This protein is a substrate for protein kinase C (PKC) phosphorylation and recruits polymerase I and transcript release factor (PTRF) to caveolae. Removal of this protein causes caveolae loss and its over-expression results in caveolae deformation and membrane tubulation.
H(+)/Cl(-) exchange transporter 3
, chloride channel 3
, chloride channel protein 3
, chloride transporter ClC-3
, protein kinase C-regulated chloride channel
, chloride channel Clc-3
, Chloride channel protein 3
, H(+)/Cl(-) exchange transporter 3-like
, phosphatidylserine binding protein
, phosphatidylserine-binding protein
, serum deprivation-response protein
, putative chloride channel ClC-3