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High-affinity epithelial cell surface receptor for APF. Additionally we are shipping CKAP4 Proteins (5) and CKAP4 Kits (2) and many more products for this protein.
Showing 10 out of 92 products:
Human Polyclonal CKAP4 Primary Antibody for IHC, IHC (p) - ABIN4298845
Ek, Andréasson, Hober, Kampf, Pontén, Uhlén, Merz, Borrebaeck: From gene expression analysis to tissue microarrays: a rational approach to identify therapeutic and diagnostic targets in lymphoid malignancies. in Molecular & cellular proteomics : MCP 2006
Human Polyclonal CKAP4 Primary Antibody for ICC, IF - ABIN439792
Agemy, Kotamraju, Friedmann-Morvinski, Sharma, Sugahara, Ruoslahti: Proapoptotic peptide-mediated cancer therapy targeted to cell surface p32. in Molecular therapy : the journal of the American Society of Gene Therapy 2013
Human Polyclonal CKAP4 Primary Antibody for ICC, IF - ABIN439793
Mandal, Mandal, Park: Global quantitative proteomics reveal up-regulation of endoplasmic reticulum stress response proteins upon depletion of eIF5A in HeLa cells. in Scientific reports 2016
Cow (Bovine) Polyclonal CKAP4 Primary Antibody for IHC, WB - ABIN2782739
Ko, McNiff, Glusac: Squamous cell carcinomas with single cell infiltration: a potential diagnostic pitfall and the utility of MNF116 and p63. in Journal of cutaneous pathology 2008
Show all 2 Pubmed References
Both DKK1 (show DKK1 Antibodies) and CKAP4 are frequently expressed in pancreatic and lung tumours, and their simultaneous expression is negatively correlated with prognosis.
CKAP4 has a role as a Dickkopf1 (show DKK1 Antibodies) receptor and in pancreatic and lung tumor progression
Human Prostate Basal cell hyperplasia is an expansion of p63 (show RPE65 Antibodies).
APF binds specifically to sites within the cytoskeleton-associated protein 4 (CKAP4) extracellular domain
CLIMP-63 (also known as CKAP4), is the partner of triadin (show TRDN Antibodies), is responsible for this association of triads and microtubules.
The still rudimentary information of how CLIMP-63 fulfills these different roles, what these are exactly and how post-translational modifications control them, will be discussed.
Although VIMP (show SELS Antibodies) can interact with CLIMP-63 and Syn5L, it does not interact with MT-binding ER proteins (such as Reep1 (show REEP1 Antibodies)) that shape the tubular smooth ER
revealed that CKAP4 could associate with EGFR (show EGFR Antibodies) at basal status and the complex was reduced upon EGF (show EGF Antibodies) stimulation, leading to release EGFR (show EGFR Antibodies) into cytoplasm
Single nucleotide polymorphisms in p63 (show RPE65 Antibodies) are implicated in the etiology of nonsyndromic bladder-exstrophy-epispadias complex.
Here we report that the combination of p63 (show RPE65 Antibodies), a master regulator of epidermal development and differentiation, and KLF4 (show KLF4 Antibodies), a regulator of epidermal differentiation, is sufficient to convert dermal fibroblasts to a keratinocyte phenotype.
p63 positively regulates desmosome adhesion by directly controlling the expression of several desmosome genes, including Dsp (show DSP Antibodies), Dsc3 (show DSC3 Antibodies) and Dsg1 (show DSG1 Antibodies).
Thus p63 closely interacts with SP-A (show SFTPA1 Antibodies) and may play a role in the trafficking or the biological function of the surfactant protein.
High-affinity epithelial cell surface receptor for APF. Mediates the anchoring of the endoplasmic reticulum to microtubules.
cytoskeleton-associated protein 4
, 63 kDa membrane protein
, 63-kDa cytoskeleton-linking membrane protein
, transmembrane protein (63kD), endoplasmic reticulum/Golgi intermediate compartment
, type-II transmembrane protein p63
, late passage cDNA-1