Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) (FUT6) ELISA Kits

The protein encoded by FUT6 is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Additionally we are shipping Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Antibodies (75) and Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Proteins (4) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
FUT6 2528 P51993
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Top Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) ELISA Kits at antibodies-online.com

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Catalog No. Reactivity Sensitivity Range Images Quantity Delivery Price Details
Human 0.59 ng/mL 1.56 ng/mL - 100 ng/mL 96 Tests 13 to 16 Days

More ELISA Kits for Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Interaction Partners

Human Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) (FUT6) interaction partners

  1. Overexpressed N-fucosylation on cell surface promotes the migration and invasion of metastatic pancreatic cancer cells. Overexpressed N-fucosylation is driven by gene FUT3, 5, and 6 in metastatic pancreatic cancer cells.

  2. findings indicated that HOTAIR/miR-326/FUT6 axis mediated colorectal cancer (CRC) procession through alpha1, 3-fucosylated CD44 via PI3K/AKT/mTOR pathway. This work rendered new therapeutic targets for CRC.

  3. we identified a novel association of a common loss-of-function, missense variant in Fucosyltransferase 6 (FUT6; rs17855739,p.Glu274Lys, P = 9.02 x 10-24) with higher soluble E-selectin levels. This variant is considerably more common in populations of African ancestry compared to non-African ancestry populations.

  4. data suggest that both FUT5 and FUT6 can promote the development of colorectal cancer (CRC) via the PI3K/Akt signalling pathway, which is regulated by miR-125a-3p. miR-125a-3p may serve as a predictive biomarker and a potential therapeutic target in CRC treatment.

  5. meta-analysis identified new variants, rs3760775 (P = 1.2 x 10-23) and rs78060698 (P = 8.3 x 10-17) in FUT6 to be associated with circulating B12 concentrations.

  6. ur results strongly showed that the low expression of FUT6 regulated by miR-106b contributed to cell migration, invasion, and proliferation in human breast cancer.

  7. There were significant differences between the type 2 diabetes mellitus patients and controls in the risk allele distributions of rs3792267 (CAPN10) (P = 0.002), rs1501299 (APM1) (P = 0.017), and rs3760776 (FUT6) (P = 0.031).

  8. The T allele of rs3760776 might repress the transcription of the FUT6 gene.

  9. In normal colon samples a significant relationship between sLe(x) expression and the ratio between FUT6/B4GALNT2 activities exists, demonstrating for the first time a role for B4GALNT2 in sLe(x) inhibition in vivo

  10. Results suggest that FUT4-, FUT6- or FUT8-mediated MDR in human HCC is associated with the activation of the PI3K/Akt pathway and the expression of MRP1.

  11. these results suggest that FUT6 plays an important role in hepatocellular carcinoma growth by regulating the PI3K/Akt signaling pathway.

  12. The biosynthesis of the selectin-ligand sialyl Lewis x in colorectal cancer tissues is regulated by fucosyltransferase VI and can be inhibited by an RNA interference-based approach.

  13. GALNT14 and FUT3/6 H-scores were significantly higher in non-small cell lung cancer cell lines sensitive to dulanermin and drozitumab versus resistant cell lines

  14. endo-beta-galactosidase-sensitive and mAb FH6-reactive carbohydrate chains are generated under the control of expression levels of FUT6 and involved in the adhesion of colon carcinoma cells to liver sections.

  15. 5'UT-exons A or C have higher expression of FUT8 transcripts and higher alpha6-fucosyltransferase activity

  16. IL-1 beta-induced sLeX expression on HuH-7 cells was suppressed by transfection of gene-specific small interference RNAs against FUT VI and ST3Gal IV but not against FUT IV and ST3Gal III.

  17. two defined regions in the 5'-flanking region of the FUT VI transcription start site are critical for FUT VI transcription in HepG2 cells

  18. results suggested that PKR is the primary target for HSV-1 early RNA during induction of FUT3, FUT5, and FUT6

Arabidopsis thaliana Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) (FUT6) interaction partners

  1. characterisation of FUT4 and FUT6 alpha-(1 --> 2)-fucosyltransferases

  2. FUT6 contributed to fucosylation of root arabinogalactan-proteins.

  3. AtFUT4 and AtFUT6 are the first enzymes to be characterized for AGP glycosylation

Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) (FUT6) Antigen Profile

Antigen Summary

The protein encoded by this gene is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Mutations in this gene are a cause of fucosyltransferase-6 deficiency. Two transcript variants encoding the same protein have been found for this gene.

Gene names and symbols associated with FUT6

  • fucosyltransferase 6 (FUT6) antibody
  • fucosyltransferase 6 S homeolog (fut6.S) antibody
  • ATFUT6 antibody
  • F7A19.16 antibody
  • F7A19_16 antibody
  • FCT3A antibody
  • FT1A antibody
  • Fuc-TVI antibody
  • fucosyltransferase 6 antibody
  • FucT-VI antibody
  • FUT antibody
  • FUT3 antibody
  • fut6 antibody
  • FUTB antibody

Protein level used designations for FUT6

alpha-(1,3)-fucosyltransferase , fucosyltransferase VI , galactoside 3-L-fucosyltransferase , fucosyltransferase 3 (galactoside 3(4)-L-fucosyltransferase, Lewis blood group) , alpha (1,3) fucosyltransferase , fucosyltransferase 3 (galactoside 3(4)-L-fucosyltransferase, Lewis blood group included) , alpha 1,3 fucosyltransferase , blood group Lewis alpha-4-fucosyltransferase , fucT-III , fucosyltransferase III , galactoside 3(4)-L-fucosyltransferase , lewis FT , alpha 1,3/4 fucosyltransferase Lewis 1 , fucosyltransferase 6 (alpha (1,3) fucosyltransferase) , fucosyltransferase 6 S homeolog

2528 Homo sapiens
428341 Gallus gallus
338077 Bos taurus
837968 Arabidopsis thaliana
399049 Xenopus laevis
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