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a putative membrane glycerophosphodiester phosphodiesterase\; may mediate lipid metabolism and G protein signaling [RGD, Feb 2006].. Additionally we are shipping GDE1 Antibodies (58) and GDE1 Kits (7) and many more products for this protein.
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MiR15a/16 inhibited the components of TGF-beta (show TGFB1 Proteins) signaling pathways.
Our outcomes suggest that miR (show MLXIP Proteins)-15a/16 maintain the retinal endothelial cell barrier by reducing TGFbeta3/VEGF (show VEGFA Proteins) signaling and increasing levels of key tight junction proteins.
The mRNA of TYRP1 (show TYRP1 Proteins) is now found to sequester the tumour suppressor miR-16.
we identified cyclin J and far upstream element-binding protein 1 (FUBP1 (show FUBP1 Proteins)) as novel miR-16 targets, which mediate miR-16 antiproliferative effects.
miR (show MLXIP Proteins)-15a/16 may function as a tumor suppressor in MM through multiple regulatory mechanisms
The expression levels of two target genes, Myb (show MYB Proteins) and VEGFR2 (show KDR Proteins), were affected significantly by miR-16, while glucose administration inhibited miR-16 expression and enhanced tumor cell proliferation and migration.
Quercetin decreases claudin-2 (show CLDN2 Proteins) expression mediated by up-regulation of miR-16 expression and instability of claudin-2 (show CLDN2 Proteins) mRNA in lung adenocarcinoma cells.
Our data indicate differential concentrations of plasma miR-16, miR (show MLXIP Proteins)-107, miR (show MLXIP Proteins)-130a and miR (show MLXIP Proteins)-146a in different breast cancer subtypes, suggesting a potential role of these miRs in breast cancer biology and tumor progression.
YAP1 (show YAP1 Proteins) was confirmed to be a functional target of miR (show MLXIP Proteins)-15a and miR-16-1 in GAC (show GLS Proteins).
miR-16 and miR (show MLXIP Proteins)-26a target checkpoint kinases Wee1 (show WEE1 Proteins) and Chk1 (show CHEK1 Proteins) in response to p53 (show TP53 Proteins) activation by genotoxic stress.
The authors confirmed the role of Gde1 in NAFLD by in vivo hepatic over-expression and shRNA knockdown studies.
findings designate GroPSer as a previously unappreciated reservoir for free serine in the nervous system and suggest that GDE1, through recycling serine from GroPSer, may impact D-serine-dependent neural signaling processes in vivo
regulation by by stimulation of G protein-coupled receptors
a multistep pathway for the production of anandamide in the nervous system by the sequential actions of Abh4 (show ABHD4 Proteins) and GDE1.
a putative membrane glycerophosphodiester phosphodiesterase\; may mediate lipid metabolism and G protein signaling
RGS16-interacting membrane protein
, membrane interacting protein of RGS16
, membrane-interacting protein of RGS16