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Bone morphogenetic proteins (e.g., BMP9\; MIM 605120) are members of the transforming growth factor-beta (see TGFB1\; MIM 190180) superfamily and regulate tissue differentiation and maintenance. Additionally we are shipping GDF15 Kits (90) and GDF15 Proteins (37) and many more products for this protein.
Showing 10 out of 334 products:
Human Polyclonal GDF15 Primary Antibody for IHC, IHC (p) - ABIN4313803
Tabrizi, Kalloger, Köbel, Cipollone, Roskelley, Mehl, Gilks: Primary ovarian mucinous carcinoma of intestinal type: significance of pattern of invasion and immunohistochemical expression profile in a series of 31 cases. in International journal of gynecological pathology : official journal of the International Society of Gynecological Pathologists 2010
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Mouse (Murine) Polyclonal GDF15 Primary Antibody for IHC - ABIN966198
Iguchi, Chrysovergis, Lee, Baek, Langenbach, Eling: A reciprocal relationship exists between non-steroidal anti-inflammatory drug-activated gene-1 (NAG-1) and cyclooxygenase-2. in Cancer letters 2009
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Mouse (Murine) Polyclonal GDF15 Primary Antibody for IHC - ABIN966197
Cekanova, Lee, Donnell, Sukhthankar, Eling, Fischer, Baek: Nonsteroidal anti-inflammatory drug-activated gene-1 expression inhibits urethane-induced pulmonary tumorigenesis in transgenic mice. in Cancer prevention research (Philadelphia, Pa.) 2009
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Mouse (Murine) Polyclonal GDF15 Primary Antibody for IHC - ABIN966196
Ding, Mracek, Gonzalez-Muniesa, Kos, Wilding, Trayhurn, Bing: Identification of macrophage inhibitory cytokine-1 in adipose tissue and its secretion as an adipokine by human adipocytes. in Endocrinology 2009
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Human Polyclonal GDF15 Primary Antibody for IF (p), IHC (p) - ABIN704561
Ruan, Li, Nie, Zhou, Zou: Differential expression of glycoprotein non-metastatic melanoma protein B (GPNMB) involved in trichostatin A-induced apoptosis in gastric cancer. in International journal of clinical and experimental medicine 2015
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Human Polyclonal GDF15 Primary Antibody for ELISA, WB - ABIN269826
Comitato, Nesaretnam, Leoni, Ambra, Canali, Bolli, Marino, Virgili: A novel mechanism of natural vitamin E tocotrienol activity: involvement of ERbeta signal transduction. in American journal of physiology. Endocrinology and metabolism 2009
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Mouse (Murine) Polyclonal GDF15 Primary Antibody for ELISA, WB - ABIN1043801
Baek, Eling: Changes in gene expression contribute to cancer prevention by COX inhibitors. in Progress in lipid research 2006
GDF-15 in blood is more accurate in differentiating pancreatic mass due to chronic pancreatitis from pancreatic adenocarcinoma than Ca19-9. the combination of GDF-15 with Ca125 (show MUC16 Antibodies) is remarkably useful in differentiating pancreatic mass.
Our results show GDF-15 as an effective biomarker in patients with metastatic colorectal cancer
GDF-15 and TRAIL-R2 (show TNFRSF10B Antibodies) were the most powerful Proximity Extension Assay chip biomarkers in predicting long-term all-cause mortality in patients with acute myocardial infarction.
This study found that the mean serum GDF15 levels in the Parkinson Disease patients were significantly higher than those of the healthy control group. Secondly, in the Parkinson Disease patients, the GDF15 levels were significantly higher in the men than in the women.
Elevated GDF-15 correlates with lack of reverse remodeling and increased mortality after TAVR and improves risk prediction of mortality.
Initial serum GDF15 level showed an inverse correlation with serum glomerular filtration rate and was associated with worse renal outcome in immunoglobulin A nephropathy.
data reveal that YAP (show YAP1 Antibodies) promotes metastasis of breast cancer cells by repressing GDF15 transcription and present a novel molecular mechanism underlying the pro-metastasis function of YAP (show YAP1 Antibodies) oncoprotein, with the implication of a therapeutic avenue for breast cancer treatment.
GDF15 was significantly correlated with mitochondrial disease severity and the proportion of ragged (show SOX18 Antibodies)-red fibers identified in skeletal muscle.
GDF-15 serum levels were elevated in patients with Systemic sclerosis and correlated with the extent of skin fibrosis, and it was found to be higher in Systemic sclerosis patients with interstitial pulmonary fibrosis
Enhanced radiosensitivity and decreased migration in cells caused by CDP138 knockdown are partially dependent on GDF15 inhibition.
GDF15 is a myokine secreted by aging-related stress and may control aging phenotype
Membrane-protein extraction and use of an intracellular protein (show CKAP2 Antibodies)-transport inhibitor showed that GDF-15 promoted CaV3.1 (show CACNA1G Antibodies) and CaV3.3 (show CACNA1I Antibodies) alpha-subunit (show POLG Antibodies) expression by trafficking to the membrane.
CHOP (show DDIT3 Antibodies) activates Gdf15 transcription via binding to its promoter upon endoplasmic reticulum stress.
The ANT1 (show SLC25A4 Antibodies)-deficient muscle mitochondria produce excess reactive oxygen species (ROS (show ROS1 Antibodies)) and are partially uncoupled. Hence, the muscle respiration under nonphosphorylating conditions is increased. Muscle transcriptome analysis revealed the induction of mitochondrial biogenesis, down-regulation of diabetes-related genes, and increased expression of the genes encoding the myokines FGF21 (show FGF21 Antibodies) and GDF15.
isolating GFRAL as the receptor for GDF15-induced anorexia and weight loss, the identification of a mechanistic basis for the non-homeostatic regulation of neural circuitry by a peripheral signal associated with tissue damage and stress; findings provide opportunities to develop therapeutic agents for the treatment of disorders with altered energy demand
These data suggest that GDF15 is required for IL-13 (show IL13 Antibodies)-induced improvement of glucose intolerance in mice fed an HFD.
HIF-1alpha (show HIF1A Antibodies) and GDF15 expression are inversely related under normoxia and hyperoxia.
This is the first study defining the role of Mic-1/Gdf15 in a number of behavioural domains.
epithelial restitution was promoted by enhanced NAG-1 expression and subsequent enterocyte locomotion during the early wound-healing process
both GDF-15 and TGF-beta1 (show TGFB1 Antibodies) counteract chemokine (show CCL1 Antibodies)-induced integrin activation on neutrophils via the ALK-5 (show TGFBR1 Antibodies)/TGF-betaRII heterodimer.
Bone morphogenetic proteins (e.g., BMP9\; MIM 605120) are members of the transforming growth factor-beta (see TGFB1\; MIM 190180) superfamily and regulate tissue differentiation and maintenance. They are synthesized as precursor molecules that are processed at a dibasic cleavage site to release C-terminal domains containing a characteristic motif of 7 conserved cysteines in the mature protein.
growth differentiation factor 15
, NSAID (nonsteroidal anti-inflammatory drug)-activated protein 1
, NSAID-activated gene 1 protein
, NSAID-regulated gene 1 protein
, growth/differentiation factor 15
, macrophage inhibitory cytokine 1
, placental TGF-beta
, placental bone morphogenetic protein
, prostate differentiation factor
, macrophage inhibiting cytokine-1