Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. Additionally we are shipping HAVCR1 Kits (84) and HAVCR1 Proteins (36) and many more products for this protein.
Showing 10 out of 160 products:
Human Monoclonal HAVCR1 Primary Antibody for CyTOF, FACS - ABIN4899580
Ivie, Fennessey, Sheng, Rubin, McClain: Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin. in PLoS ONE 2011
Show all 4 Pubmed References
Human Monoclonal HAVCR1 Primary Antibody for CyTOF, FACS - ABIN4899579
Kuroda, Fujikura, Noyori, Kajihara, Maruyama, Miyamoto, Yoshida, Takada: A polymorphism of the TIM-1 IgV domain: implications for the susceptibility to filovirus infection. in Biochemical and biophysical research communications 2014
Show all 4 Pubmed References
Mouse (Murine) Polyclonal HAVCR1 Primary Antibody for ICC, IHC - ABIN1077715
Schindler, Bondeva, Schindler, Claus, Franke, Wolf: Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2016
Data suggest that T-cell immunoglobulin domain and mucin (show SLC13A2 Antibodies) domain containing protein 1 (TIM1 (show TIMELESS Antibodies)) to be under positive natural selection in primates.
Cisplatin enhances kidney injury molecule-1 (Kim-1) gene expression in kidney S3 cells.
By preventing ERK1/2 (show MAPK1/3 Antibodies) phosphorylation following renal injury, STAT3 (show STAT3 Antibodies) phosphorylation is decreased, leading to less phosphorylated STAT3 (show STAT3 Antibodies) within the nucleus, and subsequently less KIM-1 mRNA increases post injury
Study used a previously described highly mobile membrane mimetic membrane in combination with a conventional lipid bilayer model to generate a membrane-bound configuration of Tim1 (show ARHGEF5 Antibodies) in silico, identified two possible states for a membrane-bound form of Tim1 (show ARHGEF5 Antibodies).
Our data reveal a previously unknown role for Galpha12 (show GNA12 Antibodies) in regulating efferocytosis and that renal tubular epithelial cells require KIM-1 to mediate this process.
Urinary L-FABP (show FABP1 Antibodies), NGAL (show LCN2 Antibodies), Kim-1 and albumin (show ALB Antibodies) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (show HAND1 Antibodies)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (show LDLR Antibodies) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
data suggest that TIM-1 signaling plays a direct role in Breg maintenance and induction both under physiological conditions (in response to ACs (show Acsl1 Antibodies)) and in response to therapy through TIM-1 ligation
Deletion of the mucin (show SLC13A2 Antibodies) domain impaired KIM-1-mediated phagocytic function, resulting in increased proinflammatory cytokine production, decreased antiinflammatory growth factor secretion by proximal epithelial cells, and an increase in tissue macrophages.
Tim-1 is critical for maintaining self-tolerance by regulating IL-10 (show IL10 Antibodies) production in Bregs
Quantification of TIM-3 (show HAVCR2 Antibodies) and KIM-1 mRNA expressions, along with KIM-1 protein measurements in urine and blood could be employed as promising tools for noninvasive diagnosis of allograft dysfunction.
Report detection of drug-induced acute kidney injury in humans by combining the sensitivity of urinary KIM-1 along with urinary miR (show MLXIP Antibodies)-21, -200c, and -423.
measurement of urinary and renal KIM-1 level may be helpful to evaluate severity of renal pathological damage and prognosis in adult Henoch-Schonlein purpura patients with nephritis
Urine KIM-1 level in acute kidney injury patients was significantly higher than that in the healthy controls.
PCNSL is characterized by frequent Tim-1 (show ARHGEF5 Antibodies) expression, and its soluble form in CSF (show CSF2 Antibodies) may become a useful biomarker for PCNSL.
TIM-1 (show ARHGEF5 Antibodies) is a unique marker for the identification of a human IL-10 (show IL10 Antibodies)(+) Breg subpopulation which is partially superimposed with transitional B cells
in the current meta-analysis, based on ten prospective studies involving 29366 participants, we evaluated the role of urinary tubular injury markers (NGAL (show LCN2 Antibodies), KIM-1 and NAG (show NAGLU Antibodies)) in predicting clinical outcomes including CKD stage 3, end stage renal disease and mortality.
Urinary KIM-1 levels in the acute kidney injury (AKI) preterm infant group were not significantly higher than in no-AKI group on day of life 1, 3 and 7.
We conclude that KIM-1 might serve as a sensitive biomarker to screen children for kidney damage induced by environmental toxic agents, such as chromium and arsenic
These results suggest that a 6-amino acid deletion polymorphism in the mucin (show SLC13A2 Antibodies) domain of TIM-1 (show ARHGEF5 Antibodies) protects from HIV-1 infection with a recessive effect.
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, hepatitis A virus cellular receptor 1 homolog
, kidney injury molecule 1
, t cell immunoglobulin and mucin domain-containing protein 1
, t cell membrane protein 1
, T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, rho guanine nucleotide exchange factor 5