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The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. Additionally we are shipping HAVCR1 Kits (109) and HAVCR1 Proteins (45) and many more products for this protein.
Showing 10 out of 274 products:
Human Polyclonal HAVCR1 Primary Antibody for ELISA, ICC - ABIN4359752
Lim, Chan, Lai, Tang, Chow, Lam, Leung: Distinct role of matrix metalloproteinase-3 in kidney injury molecule-1 shedding by kidney proximal tubular epithelial cells. in The international journal of biochemistry & cell biology 2012
Show all 7 Pubmed References
Human Monoclonal HAVCR1 Primary Antibody for CyTOF, FACS - ABIN4899579
Ivie, Fennessey, Sheng, Rubin, McClain: Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin. in PLoS ONE 2011
Show all 4 Pubmed References
Human Polyclonal HAVCR1 Primary Antibody for WB - ABIN4359750
Xu, Cheng, Wen, Oh, Mou, Shi, Xu, Li: Inverse association between T-cell immunoglobulin and mucin domain-1 and T-bet in a mouse model of allergic rhinitis. in The Laryngoscope 2007
Show all 2 Pubmed References
Mouse (Murine) Polyclonal HAVCR1 Primary Antibody for ICC, IHC - ABIN1077715
Schindler, Bondeva, Schindler, Claus, Franke, Wolf: Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2016
neither the inflammatory biomarker calprotectin (show S100A8 Antibodies), nor the tubular biomarkers NGAL (show LCN2 Antibodies) and KIM-1, provide robust prognostic information on the loss of renal function in a heterogeneous CKD population. All of them, however, are candidate prognostic biomarkers in primarily inflammatory renal diseases.
High KIM1 expression is associated with Clear Cell Renal Cell Carcinoma (show MOK Antibodies).
Human oligodendrocytes undergo apoptosis when exposed to Sema4A (show Sema4a Antibodies) and take up H-ferritin for meeting iron requirements and that these functions are mediated via the Tim-1 (show ARHGEF5 Antibodies) receptor. H-ferritin can block Sema4A (show Sema4a Antibodies)-mediated cytotoxicity. Sema4A (show Sema4a Antibodies) is detectable in the CSF (show CSF2 Antibodies) of multiple sclerosis patients and HIV-seropositive persons and can induce oligodendrocyte cell death.
Elevated plasma NT-proBNP and KIM-1 in patients with stage 5 chronic kidney disease (CKD5) undergoing maintenance hemodialysis (HD) and HF (+) (heart failure)CKD5-HD patients suggest that natriuretic peptides and KIM-1 may contribute to the pathogenesis of HF in CKD5-HD patients.
These findings suggest that the TIMD4 (show TIMD4 Antibodies)-HAVCR1 variants may be the genetic risk factors for coronary heart disease and ischemic strok
These studies provide evidence that, in the absence of a functional alpha-dystroglycan, TIM-1 (show ARHGEF5 Antibodies) mediates the entry of Lassa virus pseudoviral particles through interactions of virions with the IgV PtdSer-binding pocket of TIM-1 (show ARHGEF5 Antibodies).
total flavone of Desmodium styracifolium inhibited HK-2 (show HK2 Antibodies) cell apoptosis and autophagy by regulating KIM-1 via p38/MAPK (show MAPK14 Antibodies) pathway.
Polymorphism +4259A>C in exon 3 of the TIM-3 (show HAVCR2 Antibodies) gene is associated with susceptibility to multiple sclerosis but polymorphism -1637C>T in the promoter region of TIM-1 (show ARHGEF5 Antibodies) is not.
High KIM1 expression is associated with decreased proximal tubular epithelial cell function.
findings show that TIM1 (show ARHGEF5 Antibodies) is not an essential cellular receptor for hepatitis A virus entry into cultured cells
Data suggest that T-cell immunoglobulin domain and mucin (show SLC13A2 Antibodies) domain containing protein 1 (TIM1 (show TIMELESS Antibodies)) to be under positive natural selection in primates.
findings show that TIM1 (show ARHGEF5 Antibodies) is not required for hepatitis A virus (HAV) replication and pathogenesis in permissive strains of mice, although it may facilitate early stages of infection by binding phosphatidylserine on the quasi-enveloped HAV surface
the interaction of TIM4TIM1 was found to promote Th2cell proliferation through enhancing SIRT1 (show SIRT1 Antibodies) expression in mice with nasal allergic rhinitis.
findings provide evidence for the role of Tim-1 in the induction of a cytokine storm phenomenon and the pathogenesis of Ebola virus disease
Cisplatin enhances kidney injury molecule-1 (Kim-1) gene expression in kidney S3 cells.
By preventing ERK1/2 phosphorylation following renal injury, STAT3 (show STAT3 Antibodies) phosphorylation is decreased, leading to less phosphorylated STAT3 (show STAT3 Antibodies) within the nucleus, and subsequently less KIM-1 mRNA increases post injury
Study used a previously described highly mobile membrane mimetic membrane in combination with a conventional lipid bilayer model to generate a membrane-bound configuration of Tim1 (show ARHGEF5 Antibodies) in silico, identified two possible states for a membrane-bound form of Tim1 (show ARHGEF5 Antibodies).
Our data reveal a previously unknown role for Galpha12 (show GNA12 Antibodies) in regulating efferocytosis and that renal tubular epithelial cells require KIM-1 to mediate this process.
Urinary L-FABP (show FABP1 Antibodies), NGAL (show LCN2 Antibodies), Kim-1 and albumin (show ALB Antibodies) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (show HAND1 Antibodies)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (show LDLR Antibodies) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, kidney injury molecule 1
, hepatitis A virus cellular receptor 1 homolog
, t cell immunoglobulin and mucin domain-containing protein 1
, hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, t cell membrane protein 1
, T-cell immunoglobulin and mucin domain-containing protein 1