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The protein encoded by KRT16 is a member of the keratin gene family. Additionally we are shipping Keratin 16 Antibodies (138) and Keratin 16 Proteins (8) and many more products for this protein.
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inflammatory cytokines promoted Nrf2 (show GABPA ELISA Kits) nuclear translocation in psoriatic epidermis, which led to elevated expression of K6, K16, and K17 (show KRT17 ELISA Kits), thus promoting keratinocyte proliferation and contributing to the pathogenesis of psoriasis.
The wider spectrum of KRT16 mutations suggests that changes in codons 125, 127, and 132 are most commonly responsible for PC-1 (show PCSK1 ELISA Kits) and that proline substitution mutations at codons 127 or 128 may produce more severe disease
analysis of a new p.Leu421Pro (c.1262T>C) mutation in the highly conserved helix termination motif of K16 in a large Spanish family that may have a role in pachyonychia congenita
Keratin 16 regulates innate immunity in response to epidermal barrier breach.
Data established a seven-gene (AR, ESR2 (show ESR2 ELISA Kits), GATA3 (show GATA3 ELISA Kits), GBX2, KRT16, MMP28 (show MMP28 ELISA Kits) and WNT11 (show WNT11 ELISA Kits)) prognostic signature to define a subset of triple-negative breast cancer (TNBC).
Genotype-phenotype correlations among PC patients with codon-125 mutation in KRT16 were established, while the phenotypes caused by the IVS8-2A>C mutation in KRT6A (show KRT6A ELISA Kits) need further studies to confirm the rare feature of fissured tongue
We found that the KRT16 mutation carriers developed plantar keratoderma at a similar age to KRT6A (show KRT6A ELISA Kits) carriers wheras KRT6B and KRT17 (show KRT17 ELISA Kits) carriers were significantly more likely to report later onset.
Phenotypic differences exist between KRT6A (show KRT6A ELISA Kits) and KRT16 mutations support adoption of a new classification system.
Patients with p.Asn125Asp and p.Arg127Pro mutations in KRT16 exhibited more severe disease than patients carrying p.Asn125Ser and p.Arg127Cys mutations in terms of age of onset of symptoms, extent of nail (show CD244 ELISA Kits) involvement, and impact on daily quality of life.
Data suggest that an immune response to trichohyalin (show TCHH ELISA Kits) and K16 may have a role in the pathogenesis of the enigmatic disorder.
we show that the loss of Krt16 function in mice causes the development of prominent calluses on the plantar side of front and hind paws, which significantly compromise mobility.
Keratin 6 and keratin 16 may play a role in wrinkle formation.
The protein encoded by this gene is a member of the keratin gene family. The keratins are intermediate filament proteins responsible for the structural integrity of epithelial cells and are subdivided into cytokeratins and hair keratins. Most of the type I cytokeratins consist of acidic proteins which are arranged in pairs of heterotypic keratin chains and are clustered in a region of chromosome 17q12-q21. This keratin has been coexpressed with keratin 14 in a number of epithelial tissues, including esophagus, tongue, and hair follicles. Mutations in this gene are associated with type 1 pachyonychia congenita, non-epidermolytic palmoplantar keratoderma and unilateral palmoplantar verrucous nevus.
, keratin 16
, Keratin, type I cytoskeletal 17
, cytokeratin 16
, focal non-epidermolytic palmoplantar keratoderma
, keratin, type I cytoskeletal 16
, keratin complex 1, acidic, gene 16
, keratin intermediate filament 16a
, keratin intermediate filament 16b
, keratin 14
, keratin 16 (focal non-epidermolytic palmoplantar keratoderma)
, type I keratin KA16