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Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. Additionally we are shipping KLF2 Antibodies (117) and KLF2 Proteins (8) and many more products for this protein.
Showing 4 out of 11 products:
LPS exposure resulted in hypermethylation in KLF2 promoter in HUVECs, which subsequently led to downregulation of the KLF2 expression.
The expression of SH3PXD2A-AS1 (show PTGDR ELISA Kits) was inversely correlated with the expression of P57 (show CDKN1C ELISA Kits) and KLF2 in Colorectal Cancer tissue samples.
Suppression of miR (show MLXIP ELISA Kits)-150 led to up-regulation of KLF2, which in turn protected ATDC5 cells against IL-1 (show IL1A ELISA Kits)-induced injury.
Suberanilohydroxamic acid has KLF2-dependent anti-inflammatory effects, repressing vascular inflammation and atherosclerosis.
we focused on emphasizing the involvement of KLF2 in health and disease states and how they interact with transcriptional master regulator NF-kappaB (show NFKB1 ELISA Kits).
GBP5 and KLF2 may be useful as a diagnostic tool for active tuberculosis, also the two-gene set may serve as surrogate biomarkers for monitoring tuberculosis therapy.
Studies indicate that the physiological changes in miR (show MLXIP ELISA Kits)-200b levels during acute hypoxia may actually have a proangiogenic effect through Klf2 downregulation and subsequent stabilization of HIF-1 (show HIF1A ELISA Kits) signaling.
The flow-mediated miR (show MLXIP ELISA Kits)-10a expression is regulated by Kruppel-like factor 2 through modulation in RARalpha (show RARA ELISA Kits)-RARE binding, with the consequent regulation in GATA6 (show GATA6 ELISA Kits)/VCAM-1 (show VCAM1 ELISA Kits) in endothelial cells.
the effects of omentin (show ITLN1 ELISA Kits)-1 on KLF2 expression are mediated by p53 (show TP53 ELISA Kits).
ZFAS1 over-expression confers an oncogenic function in gastric cancer and this function is partly dependent on repressing KLF2 and NKD2
miRNA-92a may function through regulating KLF2.
the data revealed that puerarin inhibited the monocyte adhesion in vitro and in vivo and thus reduced atherosclerotic lesions in apoE (show APOE ELISA Kits)-/- mice; the protective effects were mediated by activation of ERK5/KLF2 signaling pathway.
any one of Klf2, Klf4 (show KLF4 ELISA Kits) and Klf5 (show KLF5 ELISA Kits) was sufficient to support self-renewal of mouse embryonic stem cells, whereas the removal of all three compromised it.
KLF2 induces an antiproliferative secretome, including activin A (show INHBA ELISA Kits), which attenuates liver regeneration.
miRNA92a targets KLF2 and PTEN signaling to promote human T follicular helper precursors in type 1 diabetes mellitus islet autoimmunity.
peripheral tolerance is enhanced or diminished through modulation of Treg trafficking tosecondary lymphoid organs (SLOs (show DHCR7 ELISA Kits)), a process that can be controlled by adjusting KLF2 protein levels.
KLF2 and KLF4 (show KLF4 ELISA Kits) serve as important regulators that promote hemoglobin alpha (show HBA-A1 ELISA Kits) expression in the endothelium.
Mouse embryos deficient in Orai1, Klf2, or Klf4 (show KLF4 ELISA Kits) showed a significantly reduced lymphatic density and impaired lymphatic development.
this study shows that KLF2 governs the degree of dendritic cells activation and hence the intensity of proatherogenic T cell responses
KLF2 and ERG (show ERG ELISA Kits) associate in a physical complex and the two proteins synergistically activate transcription of Flk1 (show KDR ELISA Kits).
miR (show MYLIP ELISA Kits)-92a coregulates KLF4 (show KLF4 ELISA Kits) and KLF2 expression in arterial endothelium and contributes to phenotype heterogeneity associated with regional atherosusceptibility and protection in vivo.
reveals a beta1 integrin-Klf2-Egfl7 (show EGFL7 ELISA Kits)-signaling pathway that is tightly regulated by cerebral cavernous malformation proteins
used microarray expression profiles of klf2a, klf2b and klf17 (show KLF17 ELISA Kits) knockdown and overexpression embryos to identify Klf target genes, which reveals that Klfs participate in specification of the extraembryonic enveloping layer
Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. KLF2 regulates T-cell trafficking by promoting expression of the lipid-binding receptor S1P1 (S1PR1\; MIM 601974) and the selectin CD62L (SELL\; MIM 153240) (summary by Weinreich et al., 2009
Krueppel-like factor 2
, Kruppel-like factor LKLF
, lung Kruppel-like zinc finger transcription factor
, lung krueppel-like factor
, lung Kruppel-like factor
, Kruppel-like factor 2 (lung)
, LOW QUALITY PROTEIN: Krueppel-like factor 2
, Kruppel-like factor 2b