anti-Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 (LILRA3) Antibodies

Leukocyte Ig-like receptors (LIRs) are a family of immunoreceptors expressed predominantly on monocytes and B cells and at lower levels on dendritic cells and natural killer (NK) cells. Additionally we are shipping Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 Proteins (13) and Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 Kits (7) and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
LILRA3 11026 Q8N6C8
LILRA3    
LILRA3    
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Top anti-Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 Antibodies at antibodies-online.com

Showing 10 out of 33 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Delivery Price Details
Cow Rabbit Un-conjugated WB 100 μL 2 to 3 Days
$289.00
Details
Human Rabbit Un-conjugated WB 100 μL 2 to 3 Days
$289.00
Details
Human Mouse Un-conjugated ELISA, WB Western Blot analysis of LILRA3 expression in transfected 293T cell line by LILRA3 monoclonal antibody (M01), clone 2E9.Lane 1: LILRA3 transfected lysate(50.485 KDa).Lane 2: Non-transfected lysate. Western Blot detection against Immunogen (74.03 KDa) . 100 μg 11 to 12 Days
$440.00
Details
Human Rabbit Un-conjugated ICC, IHC, WB Used in DAB staining on fromalin fixed paraffin-embedded Liver tissue 100 μg 13 to 16 Days
$332.00
Details
Human Mouse Un-conjugated ELISA, FACS, WB Black line: Control Antigen (100 ng);Purple line: Antigen (10ng); Blue line: Antigen (50 ng); Red line:Antigen (100 ng) Western blot analysis using LILRA3 mAb against human LILRA3 (AA: 24-168) recombinant protein. (Expected MW is 41.7 kDa) 0.1 mg 3 to 4 Days
$430.10
Details
Human Rabbit Un-conjugated WB Western Blot analysis of LILRA3 expression in transfected 293T cell line by LILRA3 MaxPab polyclonal antibody.Lane 1: LILRA3 transfected lysate(47.50 KDa).Lane 2: Non-transfected lysate. 100 μg 11 to 12 Days
$425.71
Details
Human Mouse Un-conjugated WB Western Blot analysis of LILRA3 expression in transfected 293T cell line by LILRA3 MaxPab polyclonal antibody.Lane 1: LILRA3 transfected lysate(48.29 KDa).Lane 2: Non-transfected lysate. 50 μg 11 to 12 Days
$440.00
Details
Human Rabbit Un-conjugated WB   100 μL 11 to 14 Days
$390.50
Details
Human Rabbit Un-conjugated WB   100 μL 11 to 14 Days
$551.83
Details
Human Rabbit Un-conjugated ELISA   100 μL 14 to 16 Days
$183.12
Details

Top referenced anti-Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 Antibodies

  1. Human Monoclonal LILRA3 Primary Antibody for ELISA, WB - ABIN564777 : An, Chandra, Piraino, Borges, Geczy, McNeil, Bryant, Tedla et al.: Soluble LILRA3, a potential natural antiinflammatory protein, is increased in patients with rheumatoid arthritis and is tightly regulated by interleukin 10, tumor necrosis factor-alpha, and ... in The Journal of rheumatology 2010 (PubMed)
    Show all 5 Pubmed References

More Antibodies against Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 Interaction Partners

Human Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 (LILRA3) interaction partners

  1. LILRA3 polymorphism is associated with Takayasu arteritis.

  2. Mutations in the genes glucokinase regulatory protein (GCKR), RNase L (RNASEL), leukocyte immunoglobulin-like receptor 3 (LILRA3), and dynein axonemal heavy chain 10 (DNAH10) segregated with elevated HDLc levels in families, while no mutations associated with low HDLc.

  3. The homozygous LILRA3 deletion is associated with a higher susceptibility for HIV disease and with a faster disease progression.

  4. Letter: LILRA3 gene deletion is not involved in the giant cell arteritis and systemic sclerosis predisposition in Spanish patients.

  5. LILRA3 significantly reversed Nogo-66-mediated inhibition of neurite outgrowth and promoted synapse formation in primary cortical neurons through regulation of the ERK/MEK pathway.

  6. Experiments point towards a beneficial role for LILRA3 in virus infections, especially in ssRNA viruses, like HIV, that engage TLR8; however, the potentially beneficial role of LILRA3 is abrogated during a HIV infection.

  7. LILRA3 gene deletion was not associated with Multiple Sclerosis susceptibility and did not affect the age of disease onset, clinical subtype or disease severity.

  8. Evidence showed lack of significant association between LILRA3 deletion and multiple sclerosis pathogenesis. [meta-analysis]

  9. LILRA3 is a new susceptibility factor for systemic lupus erythematosus (SLE) and primary Sjogren's syndrome (pSS). It predisposes to certain phenotypes such as leucopenia, thrombocytopenia, autoantibody positivity and increased disease activity.

  10. The current study was conducted to investigate the association of rs103294 of LILRA3 with benign prostatic hyperplasia risk.

  11. ILT6 deletion polymorphism does not appear to be a lupus susceptibility gene in South Indian Tamils, but may behave as a genetic modifier of autoantibody phenotype by influencing the production of anti-Ro60 and anti-Ro52 autoantibodies

  12. LILRA3 is an immunostimulatory molecule, whose deficiency is associated with higher frequency of B-non-Hodgkin's lymphoma.

  13. functional LILRA3 is a novel genetic risk factor for rheumatoid arthritis (RA), especially in males. It appears to highly predispose to ACPA-positive RA and confers an increased risk of disease severity in patients with early RA.

  14. Data did not indicate any association of LILRA3 deletion with multiple sclerosis (MS) susceptibility, however, patients negative for the deletion may begin to suffer from MS significantly earlier than patients who are positive.

  15. LILRA3 binds both classical and non-classical HLA class I molecules but with reduced affinities compared to LILRB1/LILRB2

  16. LILRA1 and LILRA3 generally display a greater binding preference for binding to HLA-C free heavy chain, particularly following removal of beta2-microglobulin by acid treatment.

  17. These results suggest that LILRA3 may play a role in chronic inflammatory conditions such as rheumatoid arthritis.

  18. A deletion of LILRA3 gene was found in a minority of British population

  19. Six alleles characterised, of which four will encode unique peptides, one of which is likely to directly influence ligand binding.

  20. our results suggest that both the nonfunctional LILRA3 alleles and the low-expressing LILRB2 alleles identified in our study have increased in Northeast Asians because of natural selection.

Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 (LILRA3) Antigen Profile

Protein Summary

Leukocyte Ig-like receptors (LIRs) are a family of immunoreceptors expressed predominantly on monocytes and B cells and at lower levels on dendritic cells and natural killer (NK) cells. All LIRs in subfamily B have an inhibitory function (see, e.g., LILRB1, MIM 604811). LIRs in subfamily A, with short cytoplasmic domains lacking an immunoreceptor tyrosine-based inhibitory motif (ITIM) and with transmembrane regions containing a charged arginine residue, may initiate stimulatory cascades (see LILRA1, MIM 604810). One member of subfamily A (LILRA3) lacks a transmembrane region and is presumed to be a soluble receptor (Borges et al., 1997

Gene names and symbols associated with LILRA3

  • leukocyte immunoglobulin like receptor A3 (LILRA3) antibody
  • CD85E antibody
  • e3 antibody
  • HM31 antibody
  • HM43 antibody
  • ILT-6 antibody
  • ILT6 antibody
  • LIR-4 antibody
  • LIR4 antibody

Protein level used designations for LILRA3

CD85 antigen-like family member E , immunoglobulin-like transcript 6 , leucocyte immunoglobulin-like receptor , leukocyte immunoglobulin-like receptor 4 , leukocyte immunoglobulin-like receptor A3 , leukocyte immunoglobulin-like receptor subfamily A member 3 , monocyte inhibitory receptor HM43/HM31

GENE ID SPECIES
11026 Homo sapiens
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