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The protein encoded by MAOB belongs to the flavin monoamine oxidase family.
Showing 8 out of 28 products:
Mouse (Murine) Monoamine Oxidase B ELISA Kit for Sandwich ELISA - ABIN425827
Palencia, Garcia, Osorio-Rico, Trejo-Solís, Escamilla-Ramírez, Sotelo: Neuroprotective effect of thalidomide on MPTP-induced toxicity. in Neurotoxicology 2015
Zebrafish MAO (zMAO) exhibits functional properties similar to human hMAO A as well as exhibits its own unique behavior.
expression, purification & characterization of zebrafish monoamine oxidase (zMAO) using Pichia pastoris expression system is described; zMAO possesses little differential sensitivity to acetylenic inhibitors than exhibited by either human MAO A & MAO (show MAOA ELISA Kits) B
this study confirm the presence of functionally active MAO in zebrafish brain and other tissues and characterize the neural systems that express MAO and areas of intense activity in the brain.
This study shows that MAO-B levels are increased not only in astrocytes but also in pyramidal neurons in Alzheimer disease brain and also suggests that MAO-B regulates Abeta (show APP ELISA Kits) production in neurons via gamma-secretase
The findings may suggest that MAO-B is an important sensor in iron-stressed neuronal cells.
Childhood trauma interacts with haplotypes in COMT (show COMT ELISA Kits), MAOA (show MAOA ELISA Kits) and MAOB, increasing risk for OCD.
brain protein levels of MAOB are normal or elevated in the three parkinsonian conditions-with MAOB increase generally associated with elevations in levels of astrocyte markers. Brain MAOA (show MAOA ELISA Kits) concentrations were, somewhat surprisingly, not decreased in Parkinson's disease, Progressive Supranuclear palsy or multiple system atrophy with the exception of the atrophic putamen in MSA (show TPO ELISA Kits), despite loss of dopamine neurons.
MAOA (show MAOA ELISA Kits) and MAOB variants may contribute to the etiology of Attention deficit hyperactivity disorder (ADHD) in the Indo (show IDO1 ELISA Kits)-Caucasoid population and could be responsible for higher occurrence of ADHD in the boys.
MAOB gene variants are contributing to the etiology of ADHD in the Indo (show IDO1 ELISA Kits)-Caucasoid population
This study suggests that MAOB increases ASD (show ARSD ELISA Kits) risk in males, possibly through its sex-specific regulatory effect on 5-HT (show DDC ELISA Kits) metabolism and behavior.
These findings suggest an association of platelet MAO-B activity, but a lack of association of MAOB rs1799836 and MAOA (show MAOA ELISA Kits)-uVNTR, with selected psychotic symptoms in ethnically homogenous veterans with PTSD.
No significant association was found between severe agitation and MAOA (show MAOA ELISA Kits) uVNTR or MAOB rs1799836 polymorphism, revealing that these individual polymorphisms in MAO (show MAO ELISA Kits) genes are not related to severe agitation in subjects with schizophrenia and conduct disorder. Higher platelet MAO-B activity was found in subjects with severe agitation vs. non-agitated subjects. Platelet MAO-B activity was not associated with MAOB rs1799836
we performed a meta-analysis in the Chinese population to provide comprehensive data on the association between the MAOB polymorphism and Parkinson's disease. Overall, no significant association was found between the MAOB A644G polymorphism and Parkinson's disease risk in the Chinese population.
cloning and characterization of monoamine oxidase A (show MAOA ELISA Kits) and B genes in pig
Hydralazine (Hdz) abrogated the degradation of tele-methylhistamine (tMH) catalyzed by monoamine oxidase B (MAO-B) in vitro.
Huntington disease (show HTT ELISA Kits) neural cells exhibit increased Monoamine oxidase-A (show MAOA ELISA Kits) and Monoamine oxidases-B expression and activity
Activity of monoamine oxidase B in the striatum is reduced during the presymptomatic stage, but returns to the control level during the symptomatic stage of Parkinsonism in mice.
These findings demonstrate that regulation of monoamine levels by Mao (show MAO ELISA Kits) activity in beta cells is pivotal for physiological insulin (show INS ELISA Kits) secretion and that loss of MaoB expression may contribute to the beta cell dysfunction in type 2 diabetes.
Monoamine oxidase B deficiency (along with monoamine oxidase A (show MAOA ELISA Kits)) resulted in an array of abnormalities similar to autism-spectrum disorder.
chronic elevations of monoamines, because of the absence of MAO A (show MAOA ELISA Kits) and MAO B, cause functional alterations that are accompanied with changes in the cellular mechanisms underlying learning and memory.
MAO-B is involved in the acute neurotoxicity of MPTP (show PTPN2 ELISA Kits) in embryonic and newborn mice
Data suggest that both IR1 (imidazoline receptor 1 (show NISCH ELISA Kits)) and IR2/Maob play roles in acquisition of behavioral responses in alcoholism; activation of IR1 (show NISCH ELISA Kits) and IR2/Maob may serve as molecular targets in prevention (and possibly treatment) of alcoholism.
The results of our study further confirms that the dox-inducible astrocytic MAO-B transgenic mouse displays similar age-related behavioral and neuropathological features to other models of Parkinson's disease
MAO-B seems to exert a critical role in the development of postoperative and neuropathic pain
The protein encoded by this gene belongs to the flavin monoamine oxidase family. It is a enzyme located in the mitochondrial outer membrane. It catalyzes the oxidative deamination of biogenic and xenobiotic amines and plays an important role in the metabolism of neuroactive and vasoactive amines in the central nervous sysytem and peripheral tissues. This protein preferentially degrades benzylamine and phenylethylamine.
amine oxidase [flavin-containing] B
, monoamine oxidase B
, amine oxidase
, amine oxidase [flavin-containing]
, MAO, brain
, MAO, platelet
, adrenalin oxidase
, monoamine oxidase type B
, tyramine oxidase
, monoamine oxidase A
, monoamine oxidase-B