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anti-Human ATG5 Antibodies:
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Cow (Bovine) Polyclonal ATG5 Primary Antibody for ICC, IF - ABIN258387
Wang, Singh, Massey, Kane, Kaushik, Grant, Xiang, Cuervo, Czaja: Loss of macroautophagy promotes or prevents fibroblast apoptosis depending on the death stimulus. in The Journal of biological chemistry 2008
Show all 137 Pubmed References
Human Polyclonal ATG5 Primary Antibody for IHC (p), WB - ABIN388519
Baehrecke: Autophagy: dual roles in life and death? in Nature reviews. Molecular cell biology 2005
Show all 27 Pubmed References
Human Polyclonal ATG5 Primary Antibody for IF, IHC (p) - ABIN388518
Lum, DeBerardinis, Thompson: Autophagy in metazoans: cell survival in the land of plenty. in Nature reviews. Molecular cell biology 2005
Show all 25 Pubmed References
Human Polyclonal ATG5 Primary Antibody for IF (p), IHC (p) - ABIN752413
Tang, Li, Wei, Zhang, Chiu, Hasenmayer, Zhang, Zhang: Resveratrol-induced apoptosis is enhanced by inhibition of autophagy in esophageal squamous cell carcinoma. in Cancer letters 2013
Show all 4 Pubmed References
Cow (Bovine) Polyclonal ATG5 Primary Antibody for ICC, IF - ABIN4282111
Reed, Morris, Jang, Mukherjee, Yue, Lukacs: Autophagy-inducing protein beclin-1 in dendritic cells regulates CD4 T cell responses and disease severity during respiratory syncytial virus infection. in Journal of immunology (Baltimore, Md. : 1950) 2013
Show all 2 Pubmed References
Human Polyclonal ATG5 Primary Antibody for IHC, ELISA - ABIN1001834
Kamada, Funakoshi, Shintani, Nagano, Ohsumi, Ohsumi: Tor-mediated induction of autophagy via an Apg1 protein kinase complex. in The Journal of cell biology 2000
Show all 4 Pubmed References
Human Monoclonal ATG5 Primary Antibody for ELISA, WB - ABIN522953
Song, Zhang, Guo, Sun, Han, Li, Zhao, Deng, Xie, Zhang, Wu, Wei: Autophagy contributes to the survival of CD133+ liver cancer stem cells in the hypoxic and nutrient-deprived tumor microenvironment. in Cancer letters 2013
Human Monoclonal ATG5 Primary Antibody for ELISA, WB - ABIN522956
Tsai, Lai, Sheu, Lin, Chang, Jan, Chen, Hsu, Huang, Huang, Sheen, Chen, Chang, Chang, Hsu: WWOX suppresses autophagy for inducing apoptosis in methotrexate-treated human squamous cell carcinoma. in Cell death & disease 2013
Cow (Bovine) Polyclonal ATG5 Primary Antibody for IHC, WB - ABIN2785620
Ewing, Chu, Elisma, Li, Taylor, Climie, McBroom-Cerajewski, Robinson, OConnor, Li, Taylor, Dharsee, Ho, Heilbut, Moore, Zhang, Ornatsky, Bukhman, Ethier, Sheng, Vasilescu, Abu-Farha, Lambert, Duewel et al.: Large-scale mapping of human protein-protein interactions by mass spectrometry. ... in Molecular systems biology 2007
Human Polyclonal ATG5 Primary Antibody for IF, IHC (p) - ABIN650657
Shintani, Klionsky: Autophagy in health and disease: a double-edged sword. in Science (New York, N.Y.) 2004
Show all 7 Pubmed References
miR-216b enhances the efficacy of vemurafenib and reverses drug resistance by targeting Beclin-1, UVRAG and ATG5 in melanoma.
ATG5 levels were significantly different between chronic total coronary occlusion, acute total coronary occlusion (myocardial infarction) and control groups.
Data show that miR-181a binds to the 3' untranslated regions (3'-UTR) of autophagy-related protein 5 (Atg5).
Our results suggest that these three genes that are critical components of the autophagy pathway (ATG16L, ATG2B, ATG5) are not significant risk factors among Spanish patients with either pseudoexfoliation syndrome or pseudoexfoliation glaucoma.
HBV gained access to Atg5-12/16L1 via interaction of its core protein with the Atg12 moiety of the complex. In contrast, subsequent autophagosome maturation and closure events were unnecessary for HBV replication, as evidenced by inhibition of Atg8/LC3 conjugation. Interfering with the HBV/Atg12 cross talk may be a tool for virus control.
The results support the association of SNPs in ATG5 and CRP (show CRP Antibodies) genes with response to adalimumab therapy in Crohn's disease.
the ATG5-ATG7 (show ATG7 Antibodies)-NCOA4 (show NCOA4 Antibodies) autophagic pathway has a role in ferroptosis
Atg5-mediated autophagy in proximal epithelial cells is a critical host-defense mechanism that prevents renal fibrosis by blocking G2/M arrest
knockdown of autophagy genes ATG5 or ATG7 (show ATG7 Antibodies) resulted in reduced hematopoietic stem/progenitor cell frequencies in vitro as well as in vivo.
Data suggest that Atg5 expression level in dopaminergic (DA) neurons is decreased and autophagy flux is blocked in zebrafish MPTP model of Parkinson's disease (PD); gene therapy in embryo/larva resulting in human ATG5 overexpression reverses PD pathological features, rescues DA neurons, and restores autophagy flux in DA neurons; thus, ATG5 plays role in neuroprotection.
Studies show that the TOR signaling pathway and ATG5 and ATG7 (show ATG7 Antibodies) are crucial for the regulation of cellular processes and can play various functions in different cell types in the brain.
Atg5 expression in Paneth cells is essential for tissue protection against cytokine-mediated immunopathology during acute gastrointestinal infection.
With increasing age, expression of the autophagy proteins ATG5 and LC3 (show MAP1LC3A Antibodies) in meniscus and articular cartilage was significantly reduced by 24 months.
Investigation of the autophagy-mediated regulation of renal lipid metabolism during prolonged starvation using proximal tubular cells-specific Atg5-deficient (atg5-TSKO) mice and an in vitro serum starvation model.
Atg5- and Atg7 (show ATG7 Antibodies)-dependent autophagy of dopaminergic neurons contributed to cellular and behavioral responses to morphine and may have implications for the future treatment of drug addiction.
Intracerebral hemorrhage contributes to microglia autophagic activation through the ATG5 (and BECN1 (show BECN1 Antibodies)) pathway.
Autophagy deficiency induced by RPE (show RPE Antibodies)-specific deletion of Atg5 or Atg7 (show ATG7 Antibodies) predisposes but does not necessarily drive the development of AMD (show AMD1 Antibodies)-like phenotypes or retinal degeneration.
ATG proteins ATG5 and ATG7 (show ATG7 Antibodies) may be required for phagosome maturation under some conditions, but are not universally required for this process
Atg5, an essential component of the autophagy machinery, is required for the development of mature natural killer (NK) cells and group 1, 2, and 3 innate innate lymphoid cell.
Atg5-dependent autophagy contributes to the development of acute myeloid leukemia (show BCL11A Antibodies) in an MLL (show MLL Antibodies)-AF9 (show MLLT3 Antibodies)-driven mouse model.
Data suggest that atg5 expression level in dopaminergic (DA) neurons is decreased and autophagy flux is blocked in zebrafish MPTP model of Parkinson's disease (PD); gene therapy in embryo/larva resulting in human ATG5 overexpression reverses PD pathological features, rescues DA neurons, and restores autophagy flux in DA neurons; thus, ATG5 plays role in neuroprotection.
The important role of atg5 gene in zebrafish neurogenesis and organogenesis, is reported.
ATG5 defines a phagophore domain connected to the endoplasmic reticulum during autophagosome formation in plants.
Required for autophagy. Conjugates to ATG12 and associates with isolation membrane to form cup-shaped isolation membrane and autophagosome. The conjugate detaches from the membrane immediately before or after autophagosome formation is completed (By similarity).
ATG5 autophagy related 5 homolog
, apoptosis specific protein
, apoptosis-specific protein
, autophagy protein 5
, autophagy-related 5
, autophagy 5-like protein
, APG5 autophagy 5-like
, potential preautophagosome nucleating protein Atg5