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anti-Human MAVS Antibodies:
anti-Mouse (Murine) MAVS Antibodies:
anti-Rat (Rattus) MAVS Antibodies:
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Human Monoclonal MAVS Primary Antibody for ICC, IP - ABIN1169132
Michallet, Meylan, Ermolaeva, Vazquez, Rebsamen, Curran, Poeck, Bscheider, Hartmann, König, Kalinke, Pasparakis, Tschopp: TRADD protein is an essential component of the RIG-like helicase antiviral pathway. in Immunity 2008
Show all 3 Pubmed References
Human Polyclonal MAVS Primary Antibody for IHC (p), WB - ABIN2477103
Neubauer, Newell, Ingwall: Metabolic consequences and predictability of ventricular fibrillation in hypoxia. A 31P- and 23Na-nuclear magnetic resonance study of the isolated rat heart. in Circulation 1992
Show all 3 Pubmed References
Mouse (Murine) Polyclonal MAVS Primary Antibody for WB - ABIN1881530
Ichinohe, Pang, Iwasaki: Influenza virus activates inflammasomes via its intracellular M2 ion channel. in Nature immunology 2010
Human Polyclonal MAVS Primary Antibody for ELISA, IF - ABIN4332859
Klarquist, Hennies, Lehn, Reboulet, Feau, Janssen: STING-mediated DNA sensing promotes antitumor and autoimmune responses to dying cells. in Journal of immunology (Baltimore, Md. : 1950) 2014
Using MAVS as a platform, NLRP11 (show NLRP11 Antibodies) degrades TRAF6 (show TRAF6 Antibodies) to attenuate the production of type I IFNs as well as virus-induced apoptosis. Our findings reveal the regulatory role of NLRP11 (show NLRP11 Antibodies) in antiviral immunity by disrupting MAVS signalosome.
Low MAVS expression is associated with RNA virus infections.
MCCC1 (show MCCC1 Antibodies) plays an essential role in virus-triggered, MAVS-mediated activation of NF-kappaB (show NFKB1 Antibodies) signaling.
The down regulation of TRIF (show TRIM69 Antibodies), TLR3 (show TLR3 Antibodies), and mitochondrial antiviral signaling protein (MAVS) expressions in chronic hepatitis C correlates with the disease severity and the outcome of hepatitis C virus infection
Taken together, these findings reveal an essential role of CypA (show PPIA Antibodies) in boosting RIG-I (show DDX58 Antibodies)-mediated antiviral immune responses by controlling the ubiquitination of RIG-I (show DDX58 Antibodies) and MAVS.
Data show that human cytomegalovirus (HCMV; human betaherpesvirus 5) glycoprotein US9 inhibits the IFN-beta (show IFNB1 Antibodies) response by targeting the mitochondrial antiviral-signaling protein (MAVS) and stimulator of interferon (show IFNA Antibodies) genes (STING)-mediated signaling pathways.
our results demonstrate that miR (show MLXIP Antibodies)-22 negatively regulates poly(I:C)-induced production of type I interferon (show IFNA Antibodies) and inflammatory cytokines via targeting MAVS.
this analysis did not indicate the association of the MAVS locus with susceptibility to Addison's disease and type 1 diabetes
In the late phase of RNA viral infection, iRhom2 (show RHBDF2 Antibodies) mediates proteasome-dependent degradation of the E3 ubiquitin ligase (show MUL1 Antibodies) MARCH5 (show MARCH5 Antibodies) and impairs mitochondria-associated degradation (MAD) of VISA.
findings suggest that oxidative stress-induced (show SQSTM1 Antibodies) MAVS oligomerization in SLE patients may contribute to the type I IFN signature that is characteristic of this syndrome.
MAVS was identified as a main player in HDV detection and adaptive immunity was found to be involved in the amplification of the innate immune response.
MAVS is essential for spontaneous high basal expression of IFN-beta (show IFNB1 Antibodies) in cardiac myocytes and the heart.
this study shows that keratinocytes are an important source of IFN-beta (show IFNB1 Antibodies) and MAVS is critical to this function, and demonstrates how the epidermis triggers unwanted skin inflammation under disease conditions
RIPK3 (show RIPK3 Antibodies) regulates type I IFN both transcriptionally, by interacting with MAVS and limiting RIPK1 (show RIPK1 Antibodies) interaction with MAVS, and post-transcriptionally.
Data suggest that activation of either RIG-I (show DDX58 Antibodies)/MAVS or STING pathways during acute intestinal tissue injury in mice resulted in IFN-I signaling that maintained gut (show GUSB Antibodies) epithelial barrier integrity and reduced GVHD severity.
this paper identifies TRIM31 (show TRIM31 Antibodies), an E3 ubiquitin ligase (show MUL1 Antibodies) of the TRIM (show TRAT1 Antibodies) family of proteins, as a regulator of MAVS aggregation
Taken together, these results suggest that MAVS is essential for boosting optimal primary CD4 (show CD4 Antibodies)(+) T cell responses upon NS4B-P38G West Nile virus vaccination and yet is dispensable for host protection and recall T cell responses during secondary wild-type West Nile virus infection.
this study demonstrates that the capacity of hepatitis A virus to evade MAVS-mediated type I interferon (show IFNA Antibodies) responses defines its host species range.
Hepatitis C virus NS3-4A inhibits the peroxisomal MAVS-dependent antiviral signaling response.
The authors determined that porcine reproductive and respiratory syndrome virus 3C protease cleaved MAVS at Glu268.
Real-time quantitative PCR analysis indicated that Tibetan porcine IPS-1 (show ISYNA1 Antibodies) mRNA was most abundant in the liver and kidney.
Functions of the two zebrafish MAVS variants are opposite in the induction of IFN1 by targeting IRF7 (show IRF7 Antibodies)
Data show that mitochondrial antiviral signaling protein (MAVS) splicing variant 1 (MAVS_tv1) cooperated with DEAD (Asp-Glu-Ala-Asp) box polypeptide 58 RIG-I (show DDX58 Antibodies) in the accumulation of RIG-I (show DDX58 Antibodies) transcript in a positive feedback loop.
This gene encodes an intermediary protein necessary in the virus-triggered beta interferon signaling pathways. It is required for activation of transcription factors which regulate expression of beta interferon and contributes to antiviral immunity. Multiple transcript variants encoding different isoforms have been found for this gene.
CARD adapter inducing interferon beta
, CARD adaptor inducing IFN-beta
, IFN-B promoter stimulator 1
, interferon beta promoter stimulator protein 1
, mitochondrial antiviral-signaling protein
, putative NF-kappa-B-activating protein 031N
, virus-induced signaling adaptor
, virus-induced-signaling adapter
, virus-induced signaling adapter
, IFN-beta promoter stimulator-1
, mitochondrial anti-viral signaling protein
, mitochondrial IFN-beta promoter stimulator 1
, interferon-beta promoter stimulator protein 1