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anti-Human BID Antibodies:
anti-Mouse (Murine) BID Antibodies:
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Human Polyclonal BID Primary Antibody for IP, IHC - ABIN222962
Chen, Huang, Wang: Deficiency of Bim in dendritic cells contributes to overactivation of lymphocytes and autoimmunity. in Blood 2007
Show all 6 Pubmed References
Dog (Canine) Polyclonal BID Primary Antibody for IHC, IHC (fro) - ABIN4284615
Tiao, Lin, Chen, Liou, Wang, Huang, Chou, Huang, Chuang: Dexamethasone decreases cholestatic liver injury via inhibition of intrinsic pathway with simultaneous enhancement of mitochondrial biogenesis. in Steroids 2011
Show all 4 Pubmed References
Dog (Canine) Polyclonal BID Primary Antibody for IHC (fro), IHC (p) - ABIN537391
Krajewska, Rosenthal, Mikolajczyk, Stennicke, Wiesenthal, Mai, Naito, Salvesen, Reed, Fiskum, Krajewski: Early processing of Bid and caspase-6, -8, -10, -14 in the canine brain during cardiac arrest and resuscitation. in Experimental neurology 2004
Show all 3 Pubmed References
Human Polyclonal BID Primary Antibody for IHC, IHC (p) - ABIN4284621
Spencer, Gaudet, Albeck, Burke, Sorger: Non-genetic origins of cell-to-cell variability in TRAIL-induced apoptosis. in Nature 2009
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Human Monoclonal BID Primary Antibody for ICC, FACS - ABIN968979
Wan, Liu, Xing, Chen: Bid is required in NPe6-PDT-induced apoptosis. in Photochemistry and photobiology 2008
Show all 2 Pubmed References
Human Polyclonal BID Primary Antibody for IHC (p), IHC - ABIN153045
Stracker, Morales, Couto, Hussein, Petrini: The carboxy terminus of NBS1 is required for induction of apoptosis by the MRE11 complex. in Nature 2007
Show all 2 Pubmed References
Human Monoclonal BID Primary Antibody for FACS, IF - ABIN965667
Wu, Xing, Chen, Wang: Bid is not required for Bax translocation during UV-induced apoptosis. in Cellular signalling 2007
Show all 2 Pubmed References
Human Polyclonal BID Primary Antibody for ELISA, WB - ABIN2477648
Yin: Bid, a BH3-only multi-functional molecule, is at the cross road of life and death. in Gene 2006
Human Polyclonal BID Primary Antibody for IF (p), IHC (p) - ABIN738441
Fauzee, Wang, Dong, Li, Wang, Mandarry, Xu, Pan: Novel hydrophilic docetaxel (CQMU-0519) analogue inhibits proliferation and induces apoptosis in human A549 lung, SKVO3 ovarian and MCF7 breast carcinoma cell lines. in Cell proliferation 2012
Mouse (Murine) Polyclonal BID Primary Antibody for WB - ABIN153056
Gorrini, Squatrito, Luise, Syed, Perna, Wark, Martinato, Sardella, Verrecchia, Bennett, Confalonieri, Cesaroni, Marchesi, Gasco, Scanziani, Capra, Mai, Nuciforo, Crook, Lough, Amati: Tip60 is a haplo-insufficient tumour suppressor required for an oncogene-induced DNA damage response. in Nature 2007
results suggest that histone modifications in the promoter region of miR (show MLXIP Antibodies)-500a may be responsible for the increased expression of miR (show MLXIP Antibodies)-500a in Hepatocellular carcinoma (HCC (show FAM126A Antibodies)), which promotes cancer progression by targeting BID, indicating that miR (show MLXIP Antibodies)-500a may be a potential prognostic predictor and therapeutic target for HCC (show FAM126A Antibodies) patients.
The caspase-8 (show CASP8 Antibodies)/Bid/cytochrome c (show CYCS Antibodies) axis links signals from death receptors to mitochondrial reactive oxygen species production.
administration of SMAC (show DIABLO Antibodies) or BH3 mimetics following short-term paclitaxel treatment could be an effective therapeutic strategy for TNBC, while only BH3-mimetics could effectively overcome long-term paclitaxel resistance
caspase-10 (show CASP10 Antibodies) was dispensable for enhancement of cisplatin/LA-12 and TRAIL combination-induced cell death and stimulation of Bid cleavage
an oligomeric arrangement of Bid, Bax (show BAX Antibodies), and possibly other members of the Bcl-2 (show BCL2 Antibodies) family of proteins that form a self-propagating network that permeabilizes the outer-mitochondrial membrane.
Combined therapy with Seliciclib((R)) and Belinostat((R)) results in eradication of non-small cell lung cancer via apoptosis induction and BID activation.
Results from genome-wide DNA methylation, functional network analysis and pyrosequencing, show selective CpG sites (NOS1AP, BID, and GABRB1) differentially methylated in smokers and chronic obstructive pulmonary disease patients compared to nonsmokers.
The adenovirus-mediated truncated Bid overexpression induced by the Cre/LoxP system can effectively eliminate CD133+ ovarian cancer stem cells, representing a novel therapeutic strategy for the treatment of ovarian cancer.
MiR (show MLXIP Antibodies)-20a-directed regulation of BID in colorectal cancer.
the results establish that cleavage by caspase 8 (show CASP8 Antibodies) and the subsequent association with the outer mitochondrial membrane are two critical events that activate Bid during death receptor-mediated apoptosis.
we show that BID is an important regulator downstream of FAS (show FAS Antibodies) in mouse neutrophils. Loss of BID significantly delays FASL (show FASL Antibodies)-cell death and partially counteracts the switch from apoptosis to necroptosis when caspases are blocked.
Study exposes BID as the molecular link between the previously separated pathways of oxidative stress signaling by merging the death pathways of ferroptosis and oxytosis at the level of mitochondria, i.e. the crucial point of decision for neuronal function and survival.
Cleaved Bid did not permeabilize the membrane, but lowered the membrane breakthrough force.
toxic reactive oxygen species levels in Mll5 (show MLL5 Antibodies)(-/-) mice are critically dependent on type 1 interferon (IFN-1) signaling, which triggers mitochondrial accumulation of full-length Bid.
In the present study, we identified the proapoptotic Bcl-2 (show BCL2 Antibodies) family protein Bid as a positive regulator of mutant SOD1 (show SOD1 Antibodies)-induced TLR-nuclear factor-kappaB (NF-kappaB (show NFKB1 Antibodies)) signaling in microglia.
ATM (show ATM Antibodies)-BID-MTCH2 (show MTCH2 Antibodies) pathway that we have identified plays a critical role in the DDR (show DDR1 Antibodies) via regulation of mitochondrial metabolism.
DRP1 (show CRMP1 Antibodies)-dependent apoptotic mitochondrial fission occurs independently of BAX (show BAX Antibodies), BAK (show BAK1 Antibodies) and APAF1 (show APAF1 Antibodies) to amplify cell death by BID and oxidative stress.
A phosphorylation-deficient mutant of BID, MTCH2 (show MTCH2 Antibodies)'s ligand, induces an increase in OXPHOS, but with higher ROS (show ROS1 Antibodies) and reduced ATP levels than Mtch2 (show MTCH2 Antibodies) loss, and is associated with hypersensitivity to irradiation.
Motifs of VDAC2 (show VDAC2 Antibodies) required for mitochondrial Bak (show BAK1 Antibodies) import and tBid-induced apoptosis.
BID protein suppresses p38 (show CRK Antibodies) activity and induce malignant cell transformation of hepatocytes.
Stage-specific expression of TNFalpha (show TNF Antibodies) regulates bad/bid-mediated apoptosis and RIP1 (show RALBP1 Antibodies)/ROS (show ROS1 Antibodies)-mediated secondary necrosis in Birnavirus-infected fish cells.
Bid and Bax (show BAX Antibodies) are signal transduction factors in granulosa cells and play proapoptotic roles.
Hyperosmotic Shock Engages Two Positive Feedback Loops through Caspase-3 (show CASP3 Antibodies)-dependent Proteolysis of JNK1 (show MAPK8 Antibodies)-2 and Bid.
tail regression at metamorphosis implicates an apoptotic pathway inducible by T(3) hormone in an organ autonomous manner and involving the cell death executioners BID and Caspases-2 and -8
Here, we identified and characterized the Xenopus homologs of caspase-10 (show CASP10 Antibodies) (xCaspase-10beta), a novel initiator caspase (show CASP3 Antibodies), and Bid (xBid), a BH3-only (show BBC3 Antibodies) molecule of the Bcl-2 (show BCL2 Antibodies) family involved in both the extrinsic and intrinsic pathways.
diubiquitylation is a specific feature of xBid.
This gene encodes a death agonist that heterodimerizes with either agonist BAX or antagonist BCL2. The encoded protein is a member of the BCL-2 family of cell death regulators. It is a mediator of mitochondrial damage induced by caspase-8 (CASP8)\; CASP8 cleaves this encoded protein, and the COOH-terminal part translocates to mitochondria where it triggers cytochrome c release. Multiple alternatively spliced transcript variants have been found, but the full-length nature of some variants has not been defined.
BH3-interacting domain death agonist
, BID isoform ES(1b)
, BID isoform L(2)
, BID isoform Si6
, Human BID coding sequence
, apoptic death agonist
, desmocollin type 4
, p22 BID
, apoptotic death agonist BID
, BH3 interacting domain death agonist