Browse our anti-Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) Antibodies

Full name:: anti-Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 Antibodies (SLC1A4)

On www.antibodies-online.com are 60 Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) Antibodies from 19 different suppliers available. Additionally we are shipping Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 Proteins (4) and and many more products for this protein. A total of 70 Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 products are currently listed.

Synonyms:: ASCT-1, ASCT1, AW045657, SATT, si:zc101n13.5, zgc:92817

list all antibodies	Gene Name	GeneID	UniProt
	SLC1A4	6509	P43007
	SLC1A4	55963	O35874
	SLC1A4	305540
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Most Popular Reactivities for anti-Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) Antibodies

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anti-Human Antibodies:

anti-Mouse (Murine) Antibodies:

26 WB Antibodies

anti-Rat (Rattus) Antibodies:

19 WB Antibodies

All available anti-Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 Antibodies

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Relevant Pathways for anti-Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 Antibodies

Dicarboxylic Acid Transport

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More Antibodies against Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 Interaction Partners

Human Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) interaction partners

Results suggest that ASCT1/2 may play an important role in regulating extracellular d-serine and NMDA receptor-mediated physiological effects and that ASCT1/2 inhibitors have the potential for therapeutic benefit.
ANKRD50 simultaneously engages multiple parts of the SNX27 (show SNX27 Antibodies)-retromer-WASH complex machinery in a direct and co-operative interaction network that is needed to efficiently recycle the nutrient transporters
SLC1A4 deficiency should not be considered a population-specific disorder, and all patients with unexplained severe neurodevelopmental delay and the features outlined should be investigated regardless of ethnicity, as there are no known metabolic markers of this potentially treatable condition.
SLC1A4 is the disease causing gene of a novel neurologic disorder manifesting with significant intellectual disability, severe postnatal microcephaly, spasticity and thin corpus callosum.
ASCT1 is essential in brain serine transport.
SLC1A4 disruption may impair brain development and function by decreasing the levels of L-serine in neurons. The identification of additional families with mutations in SLC1A4 would be necessary to confirm its involvement in intellectual disability.
Na+ interactions with the neutral amino acid transporter ASCT1.
ASCT1 is able to mediate a concentrative transport of alanine, which is Na+-dependent but not coupled to the Na+ gradient
used as receptor by HERV-W Env glycoprotein (show ERVW-1 Antibodies)
results strongly suggest that combinations of amino acid sequence changes and N-linked oligosaccharides in a critical carboxyl-terminal region of extracellular loop 2 (ECL2) control retroviral utilization of both the ASCT1 and ASCT2 (show SLC1A5 Antibodies) receptors

Mouse (Murine) Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) interaction partners

used as receptor by HERV-W Env glycoprotein (show ERVW-1 Antibodies) when their sites for N-glycosylation are eliminated by mutagenesis
ASCT1 expression was gradually downregulated in neuronal populations during late embryonic and neonatal periods, whereas its high expression was transmitted to radial glial cells and then to astrocytes; ASCT1 appears regulated to meet metabolic demands
results strongly suggest that combinations of amino acid sequence changes and N-linked oligosaccharides in a critical carboxyl-terminal region of extracellular loop 2 (ECL2) control retroviral utilization of both the ASCT1 and ASCT2 (show SLC1A5 Antibodies) receptors
either Phgdh (show PHGDH Antibodies) or ASCT1 is provided to each segment of renal tubules, suggesting that metabolic interplay mediated by L-serine biosynthesis and supply may exist in the kidney
Results demonstrate injury-induced changes in Phgdh (show PHGDH Antibodies) and ASCT1 expression.

Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4) Antigen Profile

Antigen Summary

Transporter for alanine, serine, cysteine, and threonine. Exhibits sodium dependence.

Alternative names and synonyms associated with Solute Carrier Family 1 (Glutamate/Neutral Amino Acid Transporter), Member 4 (SLC1A4)

solute carrier family 1 member 4 (SLC1A4) antibody
solute carrier family 1 (glutamate/neutral amino acid transporter), member 4 (SLC1A4) antibody
solute carrier family 1 (glutamate/neutral amino acid transporter), member 4 (slc1a4) antibody
solute carrier family 1 (glutamate/neutral amino acid transporter), member 4 (Slc1a4) antibody
solute carrier family 1 member 4 (Slc1a4) antibody

ASCT-1 antibody
ASCT1 antibody
AW045657 antibody
SATT antibody
si:zc101n13.5 antibody
zgc:92817 antibody

Protein level used designations for SLC1A4

solute carrier family 1 (glutamate/neutral amino acid transporter), member 4 , solute carrier family 1, member 4 , neutral amino acid transporter A-like , neutral amino acid transporter A , ASCT-1 , alanine/serine/cysteine/threonine transporter 1 , glutamate/neutral amino acid transporter , solute carrier family 1 member 4 , SATT , neutral amino acid transporter ASCT1

GENE ID	SPECIES
459278	Pan troglodytes
481391	Canis lupus familiaris
693400	Macaca mulatta
100124992	Xenopus (Silurana) tropicalis
100397568	Callithrix jacchus
100447643	Pongo abelii
100587032	Nomascus leucogenys
368885	Danio rerio
769612	Gallus gallus
6509	Homo sapiens
326577	Bos taurus
55963	Mus musculus
305540	Rattus norvegicus

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