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Human beta-1,3-Glucuronyltransferase 1 (Glucuronosyltransferase P) (B3GAT1) interaction partners

1. Cellular levels of HNK-1 carrier glycoforms are decreased in the brain of Alzheimer's disease subjects.

2. CD4(+) T cells that coexpress CD57 and CD154, which are exclusively present in cytomegalovirus-positive individuals.

3. greater HIV RNA transcription was associated with lower CD57 expression on CD8 T cells. Although causality cannot be inferred from this retrospective study, these results suggest that asymptomatic CMV replication and residual cellular HIV transcription may contribute to persistent immune dysregulation during suppressive ART.

4. TRAF4 expression is higher in mastocytosis patients with food hypersensitivity in their medical history, while B3GAT1 expression is lower in mastocytosis patients with insect venom allergy in history

5. analysis of natural killer cell markers in renal transplantation that have roles in acute rejection reveals that CD56 and CD57 are increased in the interstitial compartment in donor-specific antibody (DSA)-negative biopsies from patients with acute antibody-mediated rejection without C4d, and CD16 is increased in the glomerular compartment in DSA-positive biopsies

6. Case Report: chronic lymphocytic leukemia with aberrant CD56 and CD57 expression.

7. Among CD8+ T-lymphocytes, CD28+CD57+ cells represent a subset with some senescent features that are distinct from the CD28-CD57+ cells.

8. analysis of CD57 in prostate neoplasms and benign prostatic tissue

9. The frequency of CD57-expressing antigen specific CD4+ T cells was higher in active compared to latent tuberculosis.

10. glioblastoma stem cells are killed by CD133-specific CAR T cells but induce the T cell aging marker CD57

11. polyfunctional CD57+ cell expansion has a role in CD8+ T cell response to Staphylococcal Enterotoxin B during CMV latent infection

12. Our results suggest that an increase in the population of CD57(+) T-cells is a potent prognostic marker and may also influence the systemic immunity of patients with OSCC.

13. Abnormally low proportions of CD28(-)CD8(+) T cells expressing CD57 predict increased mortality during treated HIV infection and may be reversed with early ART initiation.

14. NKG2C(hi)CD57+ NK cells respond specifically to acute infection with cytomegalovirus and not Epstein-Barr virus.

15. CD56dim CD57int NK cells represent an intermediate functional phenotype in response to vaccine-induced and receptor-mediated stimuli.

16. T cells in unaffected skin from psoriasis patients exhibit a phenotype compatible with replicative inability. As they have a lower replicative capacity, CD57+ T cells are less frequent in lesional tissue due to the high cellular turnover.

17. NKG2Chi CD57hi natural killer cells are highly responsive to human cytomegalovirus-infected macrophages only in the presence of HCMV-specific antibodies, whereas they are functionally poor effectors of natural cytotoxicity.

18. Analysis identified CD57(bright) expression as a reliable phenotype of granule marker-containing cytotoxic T-lymphocytes.

19. Strong expression of CD57 correlates with aggressive attributes of U-NB1 and SK-N-BE(2)-C neuroblastoma cells and is linked with undifferentiated neuroblastoma cells in patients.

20. HIV infected subjects had higher percentage of T cell subsets expressing CD57.

Mouse (Murine) beta-1,3-Glucuronyltransferase 1 (Glucuronosyltransferase P) (B3GAT1) interaction partners

1. We then examined several HNK-1-related antibodies, including the Cat-315 antibody, for reactions with HNK-1-capped core M2 glycans. Finally, we confirmed the increased HNK-1 epitope expression in demyelinated brains of cuprizone-fed mice.

2. Results could lead to discovery of new roles of the human natural killer-1 (HNK-1) epitope in neural plasticity.

3. Alpha-Klotho expression is associated with expression of the non-sulfated HNK-1 epitope.

4. A linear HNK-1 mimetic peptide demonstrates potential as an efficient, feasible, and safe adjunct treatment for nerve injuries requiring surgical repair in cynomolgus monkeys.

5. the specific enzyme complex of beta4GalT-II with GlcAT-P plays an important role in the biosynthesis of HNK-1 carbohydrate.

6. HNK-1 epitope-carrying tenascin-C spliced variant regulates the proliferation of mouse embryonic neural stem cells.

7. A 207 bp fragment of the 5'-upstream region of mouse GlcAT-P gene acts as a strong promoter in PC-12 cells, which express the HNK-1 epitope.

8. HNK-1 deficient mice exhibit impaired synaptic plasticity and spatial learning. (HNK-1, CD57)

9. In development, HNK-1-positive-cells and NCAM-positive cells were seen after P-21. These results suggest that the Schwann cells from the motor and the sensory fascicles have different subtypes.

10. the localization of HNK-1 carbohydrate was restricted partially in the brain of GlcAT-P-deficient mice

11. TonEBP activation by calcium controls {beta}1,3-glucuronosyltransferase-I expression, a key regulator of glycosaminoglycan synthesis in cells of the intervertebral disc

12. HNK-1 epitope on GluR2 regulates cell surface stability of GluR2 by modulating the interaction with N-cadherin