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anti-Human B4GALT3 Antibodies:
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Data suggest that allosteric communication between heterodimeric AT1R and PTGFR is mediated through GNAQ and may also involve proximal phospholipase C but not distal protein kinase C signaling partners; PTGFR activation has negligible effects on AT1R-based conformational biosensors. (AT1R = angiotensin II receptor, type 1; PTGFR = prostaglandin F2alpha receptor; GNAQ = GTP-binding protein G[q] subunit alpha)
The mutations c.1456C < T (p.L486F) in MYOC (show MYOC Antibodies) and c.322G < A (p.V108I) in B4GALT3 are likely responsible for the pathogenesis of Primary Open-angle Glaucoma in this family.
The genotype frequencies of six SNPs in AFAP1, GMDS and PTGFR genes were conformed to Hardy-Weinberg equilibrium (HWE).
our findings evidenced that B4GALT3 upregulated by miR (show MLXIP Antibodies)-27a contributes to the tumorigenic activities by b1-integrin pathway and might provide potential biomarkers for cervical cancer.
these results indicated that the actions of AKR1C3 (show AKR1C3 Antibodies) can produce FP receptor (show PTGFR Antibodies) ligands whose activation results in carcinoma cell survival in breast cancer.
Data suggest that expression of B4GALT3 in placenta is up-regulated in third trimester; over-expression of B4GALT3 in cultured trophoblasts suppresses cell migration; thus, B4GALT3 appears to regulate trophoblast invasion in late stages of pregnancy.
The results indicated that the rs12731181 G allele of the Prostaglandin F2alpha Receptor (show PTGFR Antibodies) Gene was associated with susceptibility to essential hypertension.
Influence of PTGS1, PTGFR, and MRP4 genetic variants on intraocular pressure response to latanoprost in Chinese primary open-angle glaucoma patients
The SNPs of the PTGFR (show PTGFR Antibodies) and MMP-1 (show MMP1 Antibodies) genes may determine the latanoprost response in a white European Spanish population.
beta-1,4-Galactosyltransferase III suppresses beta1 integrin-mediated invasive phenotypes and negatively correlates with metastasis in colorectal cancer.
The protein encoded by this gene is member of the G-protein coupled receptor family. This protein is a receptor for prostaglandin F2-alpha (PGF2-alpha), which is known to be a potent luteolytic agent, and may also be involved in modulating intraocular pressure and smooth muscle contraction in uterus. Knockout studies in mice suggest that the interaction of PGF2-alpha with this receptor may initiate parturition in ovarian luteal cells and thus induce luteolysis. Two transcript variants encoding different isoforms have been found for this gene.
UDP-Gal:betaGlcNAc beta 1,4- galactosyltransferase 3
, beta-1,4-galactosyltransferase 3
, UDP-Gal:beta-GlcNAc beta-1,4-galactosyltransferase 3
, UDP-galactose:beta-N-acetylglucosamine beta-1,4-galactosyltransferase 3
, beta-1,4-GalTase 3
, beta-N-acetylglucosaminyl-glycolipid beta-1,4-galactosyltransferase 3
, N-acetyllactosamine synthase
, UDP-Gal:betaGlcNAc beta 1,4-galactosyltransferase, polypeptide 3; beta-1,4-galactosyltransferase III; beta4GalT-III; expressed sequence tag mouse EST 6
, beta-1,4-galactosyltransferase III
, PGF receptor
, PGF2 alpha receptor
, PGF2-alpha receptor
, prostaglandin F2 alpha receptor
, prostaglandin F2-alpha receptor
, prostaglandin receptor (2-alpha)
, prostanoid FP receptor
, Beta-1,4-GalTase 3
, UDP-Gal:betaGlcNAc beta 1,4- galactosyltransferase, polypeptide 3