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found endogenous expression of the long form of the enzyme in human tissue, predominantly in the trans-Golgi network of endothelial cells that form human high endothelial venules.
Epitope expressing changes with suppression or over-expression of the Gal6ST (Gal 6-O-sulfotransferase) gene, suggesting that Gal6ST is involved in the biosynthesis of the unique epitopes of KL-6/mAb.
C4ST-1 and C6ST-1 differ from each other in the recognition of uronic acid residues adjacent to the targeted GalNAc residue
The first to identify upregulated CHST1 protein gene expression correlating with the motility of Schwann cells that guide growing axons through both developmental and injured environments.
sulfated keratan sulfate is produced by beta3GNT7, beta4GalT4, CGn6ST, and KSG6ST
A new point mutation in C6ST-1 is associated with spondyloepiphyseal dysplasia, Omani type.
CHST1 does not contribute to the synthesis of glycan ligands for Siglec-F in the airways, although its absence results in exaggerated accumulation of airway macrophages and lymphocytes.
KSGal6ST is essential for C-6 sulfation of Gal within keratan sulfate in early postnatal brains.
neither KSGal6ST nor C6ST-1 is required for Siglec-F ligand synthesis in leukocytes or lung tissue
This locus encodes a member of the keratin sulfotransferase family of proteins. The encoded enzyme catalyzes the sulfation of the proteoglycan keratin.
carbohydrate (chondroitin 6/keratan) sulfotransferase 1
, carbohydrate sulfotransferase 1
, galactose/N-acetylglucosamine/N-acetylglucosamine 6-O-sulfotransferase 1
, N-acetylglucosamine-6-O-sulfotransferase 1
, keratan sulfate Gal-6 sulfotransferase