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Human Monoclonal NPPB Primary Antibody for IHC, ELISA - ABIN965690
Pfister, Erdmann, Schneider: [Natriuretic peptides BNP and NT-pro-BNP--the "new troponins" for estimation of heart failure?]. in Deutsche medizinische Wochenschrift (1946) 2003
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Mouse (Murine) Polyclonal NPPB Primary Antibody for IF (p), IHC (p) - ABIN678623
Rigazio, Hernández, Corral: Cardiopathogenic mediators generated by GATA4 signaling upon co-activation with endothelin-1 and Trypanosoma cruzi infection. in Microbial pathogenesis 2014
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Human Monoclonal NPPB Primary Antibody for IA, ELISA - ABIN253336
Bu, Zhou, Tang, Jing, Zhang: Expression and purification of a novel therapeutic single-chain variable fragment antibody against BNP from inclusion bodies of Escherichia coli. in Protein expression and purification 2013
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Human Monoclonal NPPB Primary Antibody for IHC, ELISA - ABIN1724676
Dawson, Struthers: Screening for treatable left ventricular abnormalities in diabetic patients. in Expert opinion on biological therapy 2003
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Human Monoclonal NPPB Primary Antibody for ELISA, WB - ABIN191149
Crimmins, Kao: A glycosylated form of the human cardiac hormone pro B-type natriuretic peptide is an intrinsically unstructured monomeric protein. in Archives of biochemistry and biophysics 2008
CNP (show NPPC Antibodies) and BNP (show BNC2 Antibodies) serve as oocyte maturation inhibitor to inhibit oocyte maturation in mammals
ANP (show NPPA Antibodies) and BNP (show BNC2 Antibodies) gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.
Up-regulation of BNP (show BNC2 Antibodies) and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase (show CA-P60A Antibodies) 2a in failing myocardium are observed in a porcine model of tachycardia.
Data show that all five molecules, BNP (show BNC2 Antibodies), ICAM-1 (show ICAM1 Antibodies), TNF-alpha (show TNF Antibodies), VCAM-1 (show VCAM1 Antibodies) and IL-6 (show IL6 Antibodies), quickly and reliably signaled adverse interactions.
identification and characterization of a novel alternatively spliced variant of porcine and human NPPB (show BNP Antibodies) resulting from exon 2 skipping
Arial natriuretic factor (ANF (show HESX1 Antibodies)) and N-terminal proANF may be better suited than brain natriuretic peptide (show BNP Antibodies) as markers of cardiac preload during the development and treatment of acute heart failure.
Regional differences in BNP (show BNC2 Antibodies) and CNP (show NPPC Antibodies) expression were observed in myocardium with sustained left ventricular dyssynchronous contraction.
In patients with a recent acute coronary syndrome and type 2 diabetes mellitus, BNP (show BNC2 Antibodies) and NT-proBNP were powerful predictors of cardiovascular outcomes beyond heart failure and death, ie, were also predictive of MI and stroke.
Data suggest that subjects with morbid obesity exhibit down-regulation of serum levels of BNP (show BNC2 Antibodies) compared to control subjects. This study was conducted in Italy.
Increased baseline values of BNP (show BNC2 Antibodies) (and BigET-1) after acute myocardial infarction, were independently predictive of future cardiovascular events.
Patients with acute exacerbation of COPD (show ARCN1 Antibodies) and elevated NT-pro-BNP (show BNC2 Antibodies) levels had increased hospital length of stay and need for intensive care.
BNP (show BNC2 Antibodies) blood level is a biomarker for the the severity of hand-foot-mouth disease
Plasma levels of NT-pro-BNP (show BNC2 Antibodies) were significantly higher in hypertrophic cardiomyopathy (HCM) patients with more advanced disease. Elevated NT-pro-BNP (show BNC2 Antibodies) not only reflects the diastolic impairment of the LV, but it might also be the result of cardiac ischemia in patients with HCM.
Increased preoperative brain natriuretic peptide (show BNP Antibodies) concentration was associated with risk of serious adverse events in pediatric cardiac surgery patients.
In summary, there were no significant differences in ANP (show NPPA Antibodies) and BNP (show BNC2 Antibodies) levels after percutaneous epicardial left atrial appendage suture ligation using LARIAT device 3 months after procedure.
NPPB (show BNP Antibodies) SNPs were not associated with insulin (show INS Antibodies) resistance in older adults.
In people with Type 2 diabetes, high baseline B-type natriuretic peptide (show BNP Antibodies) levels were significantly associated with future atrial fibrillation development.
The present study provides a novel insight into the potential underlying mechanisms of telmisartan-induced inhibition of cardiomyocyte hypertrophy, which involves inhibition of NFAT (show NFATC1 Antibodies) activation, nuclear translocation and the ANP (show NPPA Antibodies)/BNP (show BNC2 Antibodies) cascade.
Data suggest that the negative inhibition of P2X3R activity by the BNP (show BNC2 Antibodies)/NPR-A (show NPR1 Antibodies) pathway results in a decreased P2X3R-mediated excitability of trigeminal neurons in wildetype cultures. In familial hemiplegic migraine type-1 model cultures, however, lack of efficient P2X3Rs downregulation contributes to the neuronal hyperexcitability phenotype.
we have validated a novel gene, Myh14, as a negative regulator of ISO-induced left ventricular mass hypertrophy in an in vivo mouse model and demonstrated the up-regulation of immediate early gene Myc, fetal gene Nppb, and fibrosis gene Lgals3 in ISO-treated Myh14 deficient hearts compared to controls.
P2X3 (show P2RX3 Antibodies) receptors on mouse trigeminal ganglion neurons are subjected to contrasting modulation by inhibitory brain natriuretic peptide (show BNP Antibodies) and facilitatory calcitonin gene-related peptide (show CALCA Antibodies) that both operate via complex intracellular signaling.
mouse trigeminal neurons endogenous BNP (show BNC2 Antibodies) acts on NPR-A (show NPR1 Antibodies) receptors to determine constitutive depression of P2X3 (show P2RX3 Antibodies) receptor function. Tonic inhibition of P2X3 (show P2RX3 Antibodies) receptor activity by BNP (show BNC2 Antibodies)/NPR-A (show NPR1 Antibodies)/PKG (show PRKG1 Antibodies) pathways occurs via two distinct mechanisms: P2X3 (show P2RX3 Antibodies) serine phosphorylation and receptor redistribution to non-raft membrane compartments
The findings demonstrated a substantial role of mitochondrial dysfunction in mediating the downregulation of NKCC2 (show SLC12A1 Antibodies) and ENaCalpha (show SCNN1A Antibodies) in obstructive kidney disease, possibly via iNOS (show NOS2 Antibodies)-derived nitric oxide and BNP (show BNC2 Antibodies).
Data (including data from studies in knockout mice) suggest expression of ANP (show NPPA Antibodies)/BNP (show BNC2 Antibodies)/GC-A (show GUCA1A Antibodies) (atrial natriuretic peptide (show NPPA Antibodies), brain natriuretic peptide (show BNP Antibodies), and guanylyl cyclase-A (show NPR1 Antibodies)) is important in embryonic neovascularization and organogenesis.
Goalpha plays a role in ANF (show HESX1 Antibodies) sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF (show HESX1 Antibodies) and BNP (show BNC2 Antibodies).
The RNA-Seq demonstrates that NPPB (show BNP Antibodies) which well activates natriuretic peptide receptor 1 (NPR1 (show NPR1 Antibodies)), is well expressed in mouse dorsal root ganglian.
a slow modulatory action by BNP (show BNC2 Antibodies) on TRPV1 (show TRPV1 Antibodies) and P2X3 (show P2RX3 Antibodies) receptors outlining the role of this peptide as a negative regulator of trigeminal sensory neuron excitability to nociceptive stimuli
This gene is a member of the natriuretic peptide family and encodes a secreted protein which functions as a cardiac hormone. The protein undergoes two cleavage events, one within the cell and a second after secretion into the blood. The protein's biological actions include natriuresis, diuresis, vasorelaxation, inhibition of renin and aldosterone secretion, and a key role in cardiovascular homeostasis. A high concentration of this protein in the bloodstream is indicative of heart failure. Mutations in this gene have been associated with postmenopausal osteoporosis.
gamma-brain natriuretic peptide
, natriuretic peptides B
, brain type natriuretic peptide
, natriuretic peptide precursor B
, natriuretic protein
, brain natriuretic peptide
, Brain natriuretic factor
, natriuretic peptide precursor type B
, Gamma-brain natriuretic peptide