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anti-Human FSHR Antibodies:
anti-Mouse (Murine) FSHR Antibodies:
anti-Rat (Rattus) FSHR Antibodies:
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Human Polyclonal FSHR Primary Antibody for IF (p), IHC (p) - ABIN729508
Luo, Yin, Zhang, Yuan, Zhao, Luan, Zhang: Role of SDF-1/CXCR4 and cytokines in the development of ovary injury in chemotherapy drug induced premature ovarian failure mice. in Life sciences 2017
Show all 2 Pubmed References
Human Polyclonal FSHR Primary Antibody for WB - ABIN4909704
Wang, Wang, Zhu, Zhang, Sheng, Zhang, Han, Yuan, Weng: Seasonal expression of luteinizing hormone receptor and follicle stimulating hormone receptor in testes of the wild ground squirrels (Citellus dauricus Brandt). in Acta histochemica 2018
Human Polyclonal FSHR Primary Antibody for ELISA, WB - ABIN560952
Ritter, Thuering, Saint Mezard, Luong-Nguyen, Seltenmeyer, Junker, Fournier, Susa, Morvan: Follicle-stimulating hormone does not impact male bone mass in vivo or human male osteoclasts in vitro. in Calcified tissue international 2008
the novel homozygous FSHR variant observed in 2 siblings with Hypergonadotropic hypogonadism (show LHCGR Antibodies) can expand the spectrum of FSHR mutations in humans.
Follicle-stimulating hormone receptor single nucleotide polymorphisms are not associated with male infertility.
the expression levels of placental FSHR mRNA and protein were significantly decreased in pregnancies complicated by preeclampsia.
Polymorphisms of the FSH receptor is associated with normal morphology and genetic maturation (metaphase II) oocytes in dependence of genotypic variation polymorphisms.
T cells redirected against FSHR(+) tumor cells with full-length FSH (show BRD2 Antibodies) represent a promising therapeutic alternative against a broad range of ovarian malignancies, with negligible toxicity even in the presence of cognate targets in tumor-free ovaries
Findings of this study suggest a significant association between FSHR gene p. Thr307Ala or p. Asn680Ser coding sequence change and PCOS. The variant homozygote genotype results in a higher risk of PCOS.
The evaluation of sperm DNA fragmentation as a surrogate marker of sperm quality, and of the FSHR SNP rs6166 (p.N680S), might be useful to predict the response to FSH (show BRD2 Antibodies) treatment in men with idiopathic infertility
Mouse chondrocytes and human articular cartilage express functional FSHR. Moreover, FSH (show BRD2 Antibodies) can act on chondrocytes and cause genetic changes.
The mutation p.R59X in FSHR is causative for primary ovarian insufficiency by means of arresting folliculogenesis.
two mutations, V(221)G and T(449)N, in the extracellular domain and transmembrane helix 3, of FSHR, respectively, are reported.
The amount of dopamine d1 receptor (show DRD1 Antibodies), dopamine D2 receptor (show DRD2 Antibodies), and follicle stimulating hormone receptor (FSHr) mRNA were quantified in ovarian tissues in anestrous and mares expressing estrus during the breeding season are reported.
Activated TGF-beta (show TGFB1 Antibodies) signaling rescued miR (show MYLIP Antibodies)-143-reduced FSHR and intracellular signaling molecules, and miR (show MYLIP Antibodies)-143-induced porcine granulosa cell apoptosis.
The results showed that polymorphisms in exon 10 of the FSHR gene had a significant effect on litter size traits of Wannan Black and Berkshire pigs. These results can be applied for marker-assisted selection in the 2 swine breeds
These results showed that an increased FSHR gene expression level was accompanied with an increase in histone H3K9 acetylation levels, suggesting that histone H3K9 acetylation could regulate the expression of the porcine FSHR gene.
248 F(2) animals from a Duroc and Meishan cross were genotyped for three FSHR SNPs at positions 74, 532 and 1166, and these were correlated with the phenotypes of litter size and corpus luteum number
findings suggest the FSH receptor may be involved in the early follicle formation in pigs, which begins during prenatal life.
These data highlight an important interdependency between the potent pro-inflammatory cytokine IL1A (show IL1A Antibodies) and Fshr expression.
Study demonstrates expression of follicle-stimulating hormone receptor (FSHR) and a direct action of follicle-stimulating hormone on testicular stem/germ cells possibly mediated via alternatively spliced growth factor type 1 receptor FSHR3 in mice.
FSHR and LHR (show LHCGR Antibodies) proteins are significantly upregulated in CCs (show CCS Antibodies) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (show ESR1 Antibodies), LHR (show LHCGR Antibodies), and FSHR
Brca1 (show BRCA1 Antibodies)(GC-/-) models reveal that specific intra-follicular Brca1 (show BRCA1 Antibodies) loss alone, or combined with cancer-promoting genetic (Trp53 (show TP53 Antibodies) loss) and endocrine (high serum follicle-stimulating hormone) changes, was not sufficient to cause ovarian tumors.
Data (including date from knockout mice) suggest that Fshr is expressed early in pregnany in placenta and other extragonadal tissues of fetoplacental unit; expression is particularly strong at term.
Sertoli cell-specific expression of MTA2 (show MTA2 Antibodies) is required for transcriptional regulation of FSHR gene during spermatogenesis.
By day 20 and in adult animals total AR or FSHR ablation significantly reduced Leydig cell numbers but Sertoli cell specific AR ablation had no effect.
The results demonstrate that gain-of-function mutations of the FSHR in mice bring about distinct and clear changes in ovarian function
INVESTIGATION OF STAT5A (show STAT5A Antibodies), FSHR AND LHR (show LHCGR Antibodies) GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
The expression of FSHR mRNA in granulosa cells was highest in small antral follicles, then decreased significantly as follicles increased in size, and was lowest in cysts.
transfer. We conclude that variation at these loci of the FSHR gene has no significant effect on pregnancy rates in Luxi cattle.
Specific alleles of the bovine FSHR gene are associated with variations in embryo yield and in the number of unfertilised oocytes.
This study evaluated the relationships among aromatase (show CYP19A1 Antibodies), IGF-1 (show IGF1 Antibodies), IGF2R (show IGF2R Antibodies), and follicle stimulating hormone (FSH) receptor levels expressed in ovarian follicles of cattle selected for twin pregnancies.
granulosa cell clustering is accompanied by marked increases in FSHr, IGF-1r (show IGF1R Antibodies), and p450 arom (show CYP19A1 Antibodies) expression, and precedes induction and subsequent peak E2 production
Dominant follicles experience a reduction in FSH (show BRD2 Antibodies) dependence (diminished expression of FSHR), but acquire increased LH dependence (enhanced expression of LHCGR (show LHCGR Antibodies)) as they grow during the low FSH (show BRD2 Antibodies) milieu of follicular waves.
FSHR is specifically regulated through androgen receptor (show AR Antibodies) in granulosa cells
Heterozygous heifers showed a higher pregnancy rate (67 and 66% for LHR (show LHCGR Antibodies) and FSHR genes, respectively), but no significant effects were observed for the genes studied (
Data show that double mutation of follicle-stimulating hormone receptor (fshr) and luteinizing hormone receptor (lhcgr (show LHCGR Antibodies)) resulted in infertile males.
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr and lhcgr (show LHCGR Antibodies).
Characterization of the first functional zebrafish (Danio rerio) gonadotropic hormone I receptor (follicle stimulating hormone receptor).
FSHR is expressed in the hamster ovary starting from the fetal life to account for FSH (show BRD2 Antibodies)-induced primordial follicle formation and cyclic AMP (show TMPRSS5 Antibodies) production.
The protein encoded by this gene belongs to family 1 of G-protein coupled receptors. It is the receptor for follicle stimulating hormone and functions in gonad development. Mutations in this gene cause ovarian dysgenesis type 1, and also ovarian hyperstimulation syndrome. Alternative splicing results in multiple transcript variants.
, follicle-stimulating hormone receptor
, follitropin receptor
, follicle stimulating hormone receptor
, gonadotropic hormone I
, follicle-stimulating hormone receptor-like