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anti-Human GRIP1 Antibodies:
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Dog (Canine) Polyclonal GRIP1 Primary Antibody for WB - ABIN2779681
Sugatani, Nishitani, Yamakawa, Yoshinari, Sueyoshi, Negishi, Miwa: Transcriptional regulation of human UGT1A1 gene expression: activated glucocorticoid receptor enhances constitutive androstane receptor/pregnane X receptor-mediated UDP-glucuronosyltransferase 1A1 regulation with glucocorticoid receptor-interacting protei in Molecular pharmacology 2005
GRIP1 (show NCOA2 Antibodies) is phosphorylated at an N-terminal serine cluster by cyclin-dependent kinase-9 (CDK9 (show CDK9 Antibodies)), which is recruited into GC-induced GR:GRIP1:CDK9 (show CDK9 Antibodies) hetero-complexes, producing distinct GRE-specific GRIP1 (show NCOA2 Antibodies) phospho-isoforms.
Data suggest that molecules in the erythropoietin (show EPO Antibodies)-producing hepatocellular receptor B family (EPHB) / ephrinB (EFNB) signalling pathways, specifically ephrin B3 (show EFNB3 Antibodies) and GRIP1 (show NCOA2 Antibodies), are involved blood pressure regulation.
GRIP1 (show NCOA2 Antibodies) was identified as one of the most important differentially expressed, topologically significant proteins in the protein-protein interaction network in Alzheimer's disease.
the Trip6-GRIP1-myosin VI interaction and its regulation on F-actin network play a significant role in dendritic morphogenesis
In 15 of 590 families, we identified recessive mutations in the genes FRAS1 (show FRAS1 Antibodies), FREM2 (show FREM2 Antibodies), GRIP1 (show NCOA2 Antibodies), FREM1 (show FREM1 Antibodies), ITGA8, and GREM1 (show GREM1 Antibodies), all of which function in the interaction of the ureteric bud and the metanephric mesenchyme.
In three unrelated families with parental consanguinity, GRIP1 (show NCOA2 Antibodies) mutations were found to segregate with Fraser syndrome in an autosomal recessive manner.
Gain-of-function glutamate receptor interacting protein 1 variants alter GluA2 (show GRIA2 Antibodies) recycling and surface distribution in patients with autism
GRIP1 (show NCOA2 Antibodies) splice forms interact with gephyrin (show GPHN Antibodies) and play a role in synaptic function at GABAergic and glycinergic synapses in cultured hippocampal neurons.
after regulated endocytosis, binding to GRIP/ABP (show ABP1 Antibodies) stabilizes the internalized receptors in an intracellular pool and prevents them from being recycled back to the plasma membrane or entering a degradative pathway.
study showed neither single marker nor haplotype analysis revealed an association between variants at GRIP1 (show NCOA2 Antibodies) locus & schizophrenia; suggests it is unlikely that the GRIP1 (show NCOA2 Antibodies) polymorphisms investigated play a substantial role in schizophrenia susceptibility
GRIP1 (show NCOA2 Antibodies) is a critical regulator of immunometabolism, which engages distinct transcriptional mechanisms to coordinate the balance between macrophage populations and ultimately promote metabolic homeostasis.
GRIP1 (show NCOA2 Antibodies) binds to apoER2 (show LRP8 Antibodies) and EphrinB2 (show EFNB2 Antibodies) to induce activity-dependent AMPA (show GRIA3 Antibodies) receptor insertion at the synapse.
Study performed social behavioral testing on neuron-specific Grip1/2-double knockout (DKO) and wild type (WT) mice and determined the expression profile of key signaling proteins in AMPAR, mGluR, mTOR, and GABA pathways in brain. Data Grip1/2-mediated AMPA signaling in regulating social behaviors and disturbance of glutamate- and GABA-signaling in specialized brain regions in autism-related social behavioral deficits.
subcellular redistribution of GRIP1 (show NCOA2 Antibodies) and a change in the binding of GRIP1 (show NCOA2 Antibodies) to GluA2 (show GRIA2 Antibodies) during synaptic scaling, was observed.
This study demonistrated that deletion of accumbal GRIP1 increases vulnerability to cue-induced cocaine relapse without altering the reinforcing properties of the drug or the response to natural rewards
a regulatory role for GRIP1 (show NCOA2 Antibodies) during microtubule-based transport and suggest a crucial function for 14-3-3 (show YWHAQ Antibodies) proteins in controlling kinesin-1 motor attachment during neuronal development
Data indicate that glucocorticoid receptor (GR (show NR3C1 Antibodies)):GRIP1 (show NCOA2 Antibodies) use distinct mechanisms to repress inflammatory genes at different stages of the transcription cycle.
GRIP1 (show NCOA2 Antibodies) and 2 regulate activity-dependent AMPA (show GRIA3 Antibodies) receptor recycling via exocyst complex interactions.
This gene encodes a member of the glutamate receptor interacting protein family. The encoded scaffold protein binds to and mediates the trafficking and membrane organization of a number of transmembrane proteins. Alternatively spliced transcript variants encoding different isoforms have been described.
glutamate receptor-interacting protein 1
, eye blebs
, AMPA receptor-interacting protein GRIP1
, glutamate receptor interacting protein 1
, glutamate receptor-interacting protein 1-like