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anti-Human IL5 Antibodies:
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Human Monoclonal IL5 Primary Antibody for CyTOF, ELISA (Capture) - ABIN4900785
Xavier-Elsas, Santos-Maximiano, Queto, Mendonça-Sales, Joseph, Gaspar-Elsas, Vargaftig: Ectopic lung transplantation induces the accumulation of eosinophil progenitors in the recipients' lungs through an allergen- and interleukin-5-dependent mechanism. in Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology 2007
Show all 13 Pubmed References
Mouse (Murine) Monoclonal IL5 Primary Antibody for ICS, Neut - ABIN1176999
Abrams: Immunoenzymetric assay of mouse and human cytokines using NIP-labeled anti-cytokine antibodies. in Current protocols in immunology / edited by John E. Coligan ... [et al.] 2008
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Human Monoclonal IL5 Primary Antibody for Inhibition, ELISA - ABIN2689772
Abrams, Roncarolo, Yssel, Andersson, Gleich, Silver: Strategies of anti-cytokine monoclonal antibody development: immunoassay of IL-10 and IL-5 in clinical samples. in Immunological reviews 1992
Show all 8 Pubmed References
Human Monoclonal IL5 Primary Antibody for Inhibition, ELISA - ABIN2689771
Butterfield, Leiferman, Abrams, Silver, Bower, Gonchoroff, Gleich: Elevated serum levels of interleukin-5 in patients with the syndrome of episodic angioedema and eosinophilia. in Blood 1992
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Human Monoclonal IL5 Primary Antibody for CyTOF, FACS - ABIN4900611
Sen, Chunsong, Baojun, Linjie, Qun, San, Qiuping, Junyan, Zhang, Jinquan: Aberration of CCR7 CD8 memory T cells from patients with systemic lupus erythematosus: an inducer of T helper type 2 bias of CD4 T cells. in Immunology 2004
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Mouse (Murine) Monoclonal IL5 Primary Antibody for ELISA - ABIN2689774
Sander, Andersson, Andersson: Assessment of cytokines by immunofluorescence and the paraformaldehyde-saponin procedure. in Immunological reviews 1991
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Human Monoclonal IL5 Primary Antibody for ELISA - ABIN2689775
Denburg, Silver, Abrams: Interleukin-5 is a human basophilopoietin: induction of histamine content and basophilic differentiation of HL-60 cells and of peripheral blood basophil-eosinophil progenitors. in Blood 1991
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Human Monoclonal IL5 Primary Antibody for FACS - ABIN4898071
Alisa, Boswell, Pathan, Ayaru, Williams, Behboudi: Human CD4(+) T cells recognize an epitope within alpha-fetoprotein sequence and develop into TGF-beta-producing CD4(+) T cells. in Journal of immunology (Baltimore, Md. : 1950) 2008
Show all 2 Pubmed References
Guinea Pig Polyclonal IL5 Primary Antibody for WB - ABIN6746379
Beckhelling, Chang, Chevalier, Ford, Houliston: Pre-M phase-promoting factor associates with annulate lamellae in Xenopus oocytes and egg extracts. in Molecular biology of the cell 2003
Knockdown (KD) of STAT5 abolished the direct anti-angiogenic effect of IL-5 on VEGF-induced endothelial cell proliferation, migration and tube formation.
These experiments indicate a direct role of S. aureus in the regulation of IL-5 production in nasal polyps and may suggest the involvement of bacterial proteins detected in the tissues.
The luteal phase of the cycle in perimenstrual asthma patients is associated with increased serum estradiol levels with concurrent higher sputum concentration of IL-5 and IL-8.
The concentration of Th2-related cytokines (IL-5 and IL-13) in asthmatic children with Rhinovirus(+) was significantly higher than those with Rhinovirus(-).
IL-5 is expressed intrathecally in tick-borne encephalitis, but its pathogenetic role remains unclear.
eosinophilia in FE is secondary to dysregulation of IL-5 production in PBMC (and their component subsets).
Data indicate that interleukin-5 (IL-5) was that only serum cytokines show statistical significance between severe chronic obstructive pulmonary disease (COPD) and controls.
Serum IL-5 and IL-13 are reliable biomarkers for the blood eosinophilia asthma phenotype.
Longitudinal co-variations between inflammatory cytokines, lung function and patient reported outcomes in patients with asthma
Obesity alters the lung neutrophil infiltration to enhance breast cancer metastasis through IL5 and GM-CSF.
simvastatin was demonstrated to inhibit IL-5-induced CCR3 expression and chemotaxis of eosinophils mediated via the mevalonate pathway.
Interleukin-5/-targeted treatments offer promises to patients with eosinophilic respiratory disorders; review
Data indicate that interleukin 5 (IL-5)-associated single nucleotide polymorphisms (SNPs) were not associated with carotid intima-media thickness.
Post-liver transplant patients with food allergy showed a unique cytokine profile in response to various stimuli, with extremely elevated IL-5, low IL-10 secretion.
high levels of IL-33 and a high IL-33/soluble ST2 ratio correlates with elevated levels of IFN-gamma, TNF-alpha and IL-17alpha as well as IL-5, demonstrating that IL-33 has pleiotropic effects.
Production of IL-5 in response to both extract and the Bet v1-derived peptide mix strongly suggested that T cells were a major source of IL-5.
TRPV1 expression level, IL-4 level, and rs4790522 site mutation are the main risk factors inducing bronchial asthma in children
It has been shown in this study that the level of IL-5, one of the markers of eosinophilic inflammation, is higher in EBC of children with atopic asthma than in those with nonatopic asthma.
IL-5 may be part of protective mechanisms operating in early atherosclerosis, at least in women
Results showed that PAX2 expression is significantly overexpressed in esophageal squamous cell carcinoma tissues and IL-5 is identified as PAX2's effector for metastasis.
These data indicate that mice deficient for IL-4R(-/-)/IL-5(-/-) have the highest susceptibility for Litomosoides sigmodontis infection.
we have studied the gene expression changes associated with IL-5 up-regulation in eosinophils
binding of IL-5 to IL-5Ralpha receptors enhances angiogenic responses by stimulating the expression of HSP70-1 via the eNOS signaling pathway.
Both pre- and post-transcriptional processes may be involved in the AR modulation of ILC2 IL-5 and IL-13 production.
IL-33 acts directly on bone marrow ILC2s, making them an early source of IL-5 and part of a process that is central in IL-33-driven eosinophilia.
these studies establish a basal defect in eosinophilopoiesis in IL-33- and ST2-deficient mice and a mechanism whereby IL-33 supports eosinophils by driving both systemic IL-5 production and the expansion of IL-5Ralpha-expressing precursor cells
Increased production of IL-5 from Peyer's patch cells and the restored Th1-type immune response might cause the production of abnormal IgA and might induce the glomerular deposition of IgA in IGA nephropathy.
E. granulosus infection remarkably reduces the severity of OVA-induced airway inflammation likely through enhancing IL-10 and down-regulation of IL-5 and IL-17A.
selective proliferation of IgM rheumatoid factor-secreting B-1a cells is induced by co-stimulation by the specific pathogen antigen and IL-5 in the development of MC in Capillaria hepatica-infected mice
IL5, a cytokine involved in allergic and infectious diseases, facilitates metastatic colonization through recruitment of sentinel eosinophils and regulation of other inflammatory/immune cells in the microenvironment of the distal lung.
In mice, low-dose IL-2-anti-IL-2 antibody complexes drove group 2 innate lymphoid cells (ILC2) to produce IL-5 and proliferate.
Data (including data from knockout mice) suggest that up-regulation of IL5 production in lungs during influenza virus infection is due to infiltration of natural killer cells and alveolar macrophages into infected lung tissue.
A decrease in the levels of IL-5, IL-9, and IL-6R in the BALF.
eosinophils express CAR4 following IL-5 or allergen exposure, and that CAR4 is involved in regulating the lung transcriptome associated with allergic airway inflammation
Loa loa exhibits a differential survival and development in different strains of cytokine or cytokine receptor gene knockout mice with IL-4R and IL-5 playing critical roles in the host resistance to L. loa infection.
In mice 8 hours afterthe single administration of Immunovac-VP-4 the levels of IL-1beta, IL-6, IL- 12, IL-5 increased significantly. However their concentration differed depending on the method of administration.
Protection of montelukast on OVA-induced eosinophilic gastroenteritis via modulating IL-5, eotaxin-1 and MBP expression.
Together, these studies support the conclusion that surfactant protein D increases susceptibility to Cryptococcus neoformans infection by promoting Cryptococcus neoformans-driven pulmonary IL-5 and eosinophil infiltration.
IL5 induced eosinophils and cysteinyl leukotrienes are involved in the pathology of mite antigen-induced chronic asthma model.
This study showed that pronounced lung eosinophilia in horses can be transient, abate without specific treatment, and in this instance, lack correlation to upregulation of expression of either IL-4 or IL-5.
The protein encoded by this gene is a cytokine that acts as a growth and differentiation factor for both B cells and eosinophils. This cytokine is a main regulator of eosinopoiesis, eosinophil maturation and activation. The elevated production of this cytokine is reported to be related to asthma or hypereosinophilic syndromes. The receptor of this cytokine is a heterodimer, whose beta subunit is shared with the receptors for interleukine 3 (IL3) and colony stimulating factor 2 (CSF2/GM-CSF). This gene, together with those for interleukin 4 (IL4), interleukin 13 (IL13), and CSF2, form a cytokine gene cluster on chromosome 5. This cytokine, IL4, and IL13 are found to be regulated coordinately by long-range regulatory elements spread over 120 kilobases on chromosome 5q31.
B-cell differentiation factor I
, T-cell replacing factor
, eosinophil differentiation factor
, B-cell growth factor II
, cytotoxic T-lymphocyte inducer
, Interleukin 5 (colony-stimulating factor eosinophil)
, Interleukin 5 (colony-stimulating factor, eosinophil)
, colony-stimulating factor, eosinophil
, interleukin 5
, interleukin 5 (colony-stimulating factor, eosinophil)