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anti-Mouse (Murine) Caspase 9 Antibodies:
anti-Rat (Rattus) Caspase 9 Antibodies:
anti-Human Caspase 9 Antibodies:
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Human Polyclonal Caspase 9 Primary Antibody for IHC (p), WB - ABIN3044458
Jiang, Li, Zhou, Wang, Zhang, Wang: Colistin-induced apoptosis in PC12 cells: involvement of the mitochondrial apoptotic and death receptor pathways. in International journal of molecular medicine 2014
Show all 15 Pubmed References
Dog (Canine) Polyclonal Caspase 9 Primary Antibody for FACS, IHC - ABIN4288194
Cowan, Thai, Krajewski, Reed, Nicholson, Kaufmann, Roskams: Caspases 3 and 9 send a pro-apoptotic signal from synapse to cell body in olfactory receptor neurons. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2001
Show all 15 Pubmed References
Human Polyclonal Caspase 9 Primary Antibody for WB - ABIN3043748
Ge, Yu, Petitte, Zhang: Epidermal growth factor-induced proliferation of chicken primordial germ cells: involvement of calcium/protein kinase C and NFKB1. in Biology of reproduction 2009
Show all 14 Pubmed References
Dog (Canine) Polyclonal Caspase 9 Primary Antibody for IHC, IHC (fro) - ABIN4288190
Krajewski, Krajewska, Ellerby, Welsh, Xie, Deveraux, Salvesen, Bredesen, Rosenthal, Fiskum, Reed: Release of caspase-9 from mitochondria during neuronal apoptosis and cerebral ischemia. in Proceedings of the National Academy of Sciences of the United States of America 1999
Show all 7 Pubmed References
Dog (Canine) Polyclonal Caspase 9 Primary Antibody for FACS, IF (p) - ABIN724160
Wang, Han, Zhou, Wu, Wang, Jia, Lv, Cheng, Pan, Liu, Zhou, Sun: Inhibition of hydrogen sulfide synthesis provides protection for severe acute pancreatitis rats via apoptosis pathway. in Apoptosis : an international journal on programmed cell death 2013
Show all 6 Pubmed References
Human Polyclonal Caspase 9 Primary Antibody for FACS, IHC (p) - ABIN1881137
Martin, Allan, Lickrish, Sampson, Morrice, Clarke: Protein kinase A regulates caspase-9 activation by Apaf-1 downstream of cytochrome c. in The Journal of biological chemistry 2005
Show all 5 Pubmed References
Human Polyclonal Caspase 9 Primary Antibody for ELISA, ICC - ABIN6260485
He, Cao, Guo, Li, Shang, Liu, Xie, Xu, Liu: Quercetin induces autophagy via FOXO1-dependent pathways and autophagy suppression enhances quercetin-induced apoptosis in PASMCs in hypoxia. in Free radical biology & medicine 2017
Show all 5 Pubmed References
Human Polyclonal Caspase 9 Primary Antibody for IP, WB - ABIN222956
Scarabelli, Stephanou, Pasini, Comini, Raddino, Knight, Latchman: Different signaling pathways induce apoptosis in endothelial cells and cardiac myocytes during ischemia/reperfusion injury. in Circulation research 2002
Show all 4 Pubmed References
Human Monoclonal Caspase 9 Primary Antibody for ICC, IF - ABIN153015
Fearnhead, Rodriguez, Govek, Guo, Kobayashi, Hannon, Lazebnik: Oncogene-dependent apoptosis is mediated by caspase-9. in Proceedings of the National Academy of Sciences of the United States of America 1998
Show all 5 Pubmed References
Human Polyclonal Caspase 9 Primary Antibody for IF (p), IHC (p) - ABIN724175
Gao, Liu, Li, Wu, Peng, Jing: Hispidulin induces mitochondrial apoptosis in acute myeloid leukemia cells by targeting extracellular matrix metalloproteinase inducer. in American journal of translational research 2016
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Inhibition of caspase-9 aggravates acute liver injury through suppression of cytoprotective autophagy.
inducible caspase-9 (iC9) safety switch can eliminate CD19 (show CD19 Antibodies).CAR-Ts in a dose-dependent manner, allowing either a selective containment of CD19 (show CD19 Antibodies).CAR-T expansion in case of CRS (show CARS Antibodies) or complete deletion on demand granting normal B cell reconstitution.
Cepharanthine treatment reduced mitochondrial membrane potential and upregulated the level of cleaved caspases, including caspase-9 and caspase-3 (show CASP3 Antibodies)/7, in a dose-dependent fashion.
Bcl-2 (show BCL2 Antibodies)-caspase-9 apoptosis pathway was clearly activated in the testis of asthmatic mice with the increased expression of apoptosis-related proteins.
Inhibition of Caspase-9 restricted, while Apaf-1 promoted, Chlamydia pneumoniae infection in HEp-2, HeLa, and mouse epithelial fibroblast (MEF) cells.
The study demonstrates sublethal caspase (show CASP3 Antibodies)-activation has an important role in cardiomyocyte differentiation and may have significant implications for promoting cardiac regeneration after myocardial injury involving exogenous or endogenous cell sources.
Casp6 (show CASP6 Antibodies) knockout in YAC128 mice does not abolish the presence of the mutant HTT (show HTT Antibodies) protein-586 fragment but ameliorates depression, body weight gain, IGF-1 (show IGF1 Antibodies) and BDNF (show BDNF Antibodies) levels in YAC128 mice.
study demonstrates that MMP-3 (show MMP3 Antibodies) leads to caspase-9 activation and suggests that this occurs indirectly via a cytosolic protein, possibly involving Apaf-1 (show APAF1 Antibodies)
caspase-9 mediates Puma (show BBC3 Antibodies) activation to determine the threshold for overcoming chemoresistance in cancer cells.
Casp9, the initiator caspase (show CASP3 Antibodies) of the intrinsic apoptotic cascade, has a role in murine fetal and adult hematopoiesis
Low CASP9 expression is associated with Colorectal Cancer.
the results of the present study suggest that miR (show MLXIP Antibodies)-96-5p, which is frequently upregulated in hepatocellular carcinoma (HCC (show FAM126A Antibodies)), inhibits apoptosis by targeting CASP9. Therefore, miR (show MLXIP Antibodies)-96-5p may be a potential therapeutic target for HCC (show FAM126A Antibodies)
CASP9 germline mutations may have played a role at least in part to the susceptibility of development of gliomas in a Li-Fraumeni-like family lacking a TP53 (show TP53 Antibodies) germline mutation.
The caspase 9 level was significantly lower and related with oxidant status in patients with polycystic ovary syndrome, while the circulating levels of caspases 3 and 7 were statistically similar in both PCOS and control groups.
Results indicate that the apoptotic protease-activating factor 1 (Apaf-1 (show APAF1 Antibodies)) apoptosome activates caspase-9 in part through sequestration of the inhibitory caspase (show CASP3 Antibodies) recruitment domains (CARDs) domain.
1,4-BQ evidently induced mitochondria-mediated apoptosis and increased pro-apoptotic genes (Caspase-9 and Caspase-3 (show CASP3 Antibodies)) expression in a dose-dependent manner.
DES1 plays a key role in palmitic acid-mediated caspase 9 and caspase 3 (show CASP3 Antibodies) activation.
CASP9 expression associated with inhibition of miR (show MLXIP Antibodies)-182.
CASP-9 polymorphism is associated with Primary Brain Tumors.
Fisetin inhibited Triple-Negative Breast Cancer Cells cell division and induced apoptosis, which was associated with mitochondrial membrane permeabilization and the activation of caspase-9 and caspase-8 (show CASP8 Antibodies), as well as the cleavage of poly(ADP-ribose) polymerase-1 (show PARP1 Antibodies).
Suppression of caspase-9 resulted in significant upregulation of cell proliferation-, adhesion-, growth-, development- and division-regulating genes, and the downregulation of cell death program- and stress response-regulating genes.
Endothelial cell apoptosis by H. somnus-activated platelets required activation of both caspase-8 (show CASP8 Antibodies) and caspase-9.
The ATP and cytochrome c (show CYCS Antibodies) dependent activation of caspase-9 is increased during hypoxia. The ATP and cytochrome c (show CYCS Antibodies) sites of Apaf-1 (show APAF1 Antibodies) that mediate caspase-9 activation are modified during hypoxia.
Results suggest that zearalenone induces apoptosis and necrosis of porcine granulosa cells in a dose-dependent manner via a caspase-3 (show CASP3 Antibodies)- and caspase-9-dependent mitochondrial pathway.
We conclude that the hypoxia-induced activation of caspase-9 is mediated by Src kinase (show CSK Antibodies).
This gene encodes a member of the cysteine-aspartic acid protease (caspase) family. Sequential activation of caspases plays a central role in the execution-phase of cell apoptosis. Caspases exist as inactive proenzymes which undergo proteolytic processing at conserved aspartic residues to produce two subunits, large and small, that dimerize to form the active enzyme. This protein is processed by caspase APAF1\; this step is thought to be one of the earliest in the caspase activation cascade. Alternative splicing results in two transcript variants which encode different isoforms.
, ICE-like apoptotic protease 6
, apoptotic protease Mch-6
, apoptotic protease-activating factor 3
, 25 kDa caspase-9 dominant negative protein
, Ice-like apoptotic protease 6
, apoptotic protease activating factor 3
, caspase-9 dominant negative form
, caspase-9-carboxyl-terminal divergent
, apoptotic protease MCH-6
, protein phosphatase 1, regulatory subunit 56
, caspase 9, apoptosis-related cysteine protease