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anti-Human CLIP2 Antibodies:
anti-Rat (Rattus) CLIP2 Antibodies:
anti-Mouse (Murine) CLIP2 Antibodies:
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Human Monoclonal CLIP2 Primary Antibody for IF, WB - ABIN968391
De Zeeuw, Hoogenraad, Goedknegt, Hertzberg, Neubauer, Grosveld, Galjart: CLIP-115, a novel brain-specific cytoplasmic linker protein, mediates the localization of dendritic lamellar bodies. in Neuron 1998
Show all 2 Pubmed References
Human Polyclonal CLIP2 Primary Antibody for IHC, IHC (p) - ABIN4301707
Selmansberger, Feuchtinger, Zurnadzhy, Michna, Kaiser, Abend, Brenner, Bogdanova, Walch, Unger, Zitzelsberger, Hess: CLIP2 as radiation biomarker in papillary thyroid carcinoma. in Oncogene 2015
Cross checking with epidemiological estimates and model validation suggests that CLIP2 is a marker of high precision. CLIP2 leaves an imprint in the epidemiological incidence data which is typical for a driver gene.
This study aims to analyze the influences of single-nucleotide polymorphism in the NCAN (show NCAN Antibodies)-CILP2 (show Cilp2 Antibodies) region on non-alcoholic fatty liver disease and plasma lipid levels in the Asian and Pacific ethnic groups.
CLIP2 protein expression is elevated in papillary thyroid carcinomas from patients exposed to radioiodine fallout compared with a nonexposed control group.
A clear radiation dose-response relationship for the CLIP2 marker expression in post-Chernobyl papillary thyroid carcinomas
The minor T allele of CILP2 (show Cilp2 Antibodies) gene and I allele of ACE (show ACE Antibodies) gene have a protective effect.
CLIP2 haploinsufficiency by itself does not lead to the physical or cognitive characteristics of the Williams-Beuren syndrome; GTF2IRD1 and GTF2I (show GTF2I Antibodies) are the main genes causing the cognitive defects
Sex (male)-specific association of rs16996148 SNP in the NCAN/CILP2/PBX4 and serum lipid levels is observed both the Mulao and Han ethnic groups.
Cartilage intermediate layer protein 2 (CILP-2 (show Cilp2 Antibodies)) is expressed in articular and meniscal cartilage and down-regulated in experimental osteoarthritis.
Data show no differences in triglyceride or total cholesterol levels in relation to any allelic variants of ANGPTL3 (show ANGPTL3 Antibodies), CILP2 (show Cilp2 Antibodies), or TRIB1 (show TRIB1 Antibodies) SNPs.
Data show that gene CLIP2 was specifically overexpressed in the exposed cases.
evidence that mice with haploinsufficiency for Cyln2 have features reminiscent of Williams syndrome, including mild growth deficiency, brain abnormalities, hippocampal dysfunction and particular deficits in motor coordination
Williams Syndrome is caused by a variety of genes and that heterozygous deletion of CYLN2 is one of the major causes
May play a role in cartilage scaffolding (By similarity).
CAP-Gly domain-containing linker protein 2
, Williams-Beuren syndrome chromosome region 3
, Williams-Beuren syndrome chromosome region 4
, cytoplasmic linker 2
, cytoplasmic linker protein 115
, cytoplasmic linker protein 2
, williams-Beuren syndrome chromosomal region 3 protein
, williams-Beuren syndrome chromosomal region 4 protein
, cytoplasmic linker protein 1, 115 kDa
, CAP-GLY domain containing linker protein 2
, CAP-Gly domain-containing linker protein 2-like
, cartilage intermediate layer protein-like protein 2