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anti-Human TRIAP1 Antibodies:
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Inhibition of TRIAP1 in RPMI8226 cells increased the percentage of apoptotic cells, accompanied by increased expression of APAF1 and Caspase 9, and Caspase 9 and Caspase 3/7 activity.
TRIAP1 is regulated by miR-320b and has a role in progression in nasopharyngeal carcinoma
crystal structures of free TRIAP1 and TRIAP1-SLMO1 complex reveal how the PRELI domain is chaperoned during import into the intermembrane mitochondrial space; structural resemblance of PRELI-like domain of SLMO1 with that of mammalian phoshatidylinositol transfer proteins suggest they share similar lipid transfer mechanisms
Results describe the upregulation of TRIAP1 in drugresistant breast cancer cells. Its experimental modulation changed breast tumor cells sensitivity to doxorubicin, thus confirming its role in drug resistance.
Loss of TRIAP1 or PRELI impairs the accumulation of Cardiolipin, facilitates the release of cytochrome c, and renders cells vulnerable to apoptosis upon intrinsic and extrinsic stimulation.
TRIAP1 is Conserved Specific Coregulators of the p21:PUMA Expression Ratio.
HSPC132 is p53CSV, a novel p53-inducible gene involved in the p53-dependent cell-survival pathway
Upregulated in at least 50% of multiple myeloma cases tested.
Mediates cell survival by inhibiting activation of caspase-9 which prevents induction of apoptosis.
TP53-regulated inhibitor of apoptosis 1
, mitochondrial distribution and morphology 35 homolog
, p53-inducible cell-survival factor
, protein 15E1.1