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anti-Mouse (Murine) NCOR1 Antibodies:
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Human Polyclonal NCOR1 Primary Antibody for ChIP, IP - ABIN188815
Dai, Hussain: NR2F1 disrupts synergistic activation of the MTTP gene transcription by HNF-4α and HNF-1α. in Journal of lipid research 2012
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Human Monoclonal NCOR1 Primary Antibody for IHC, ELISA - ABIN969310
Zhang, Yoon, Wong: JMJD2A is a novel N-CoR-interacting protein and is involved in repression of the human transcription factor achaete scute-like homologue 2 (ASCL2/Hash2). in Molecular and cellular biology 2005
Show all 2 Pubmed References
Human Monoclonal NCOR1 Primary Antibody for IHC, ELISA - ABIN966640
Atsumi, Tomita, Kiyoi, Naoe: Histone deacetylase 3 (HDAC3) is recruited to target promoters by PML-RARalpha as a component of the N-CoR co-repressor complex to repress transcription in vivo. in Biochemical and biophysical research communications 2006
Show all 2 Pubmed References
Human Polyclonal NCOR1 Primary Antibody for ICC, IF - ABIN407956
Nazha, Granner, Maloney, Odashiro, Antecka, Burnier: Aberrant nuclear repressor coreceptor 1 localization in human retinoblastoma. in Ophthalmic research 2013
Ncor1 and Ncor2 play essential but distinct roles in zebrafish primitive myelopoiesis
Liver-Specific Ablation of NCoR1 Induces Hepatic Steatosis and Does Not Prevent Hypothyroidism-Induced Hypercholesterolemia.
the nuclear receptor NCoR1 suppresses Bim1 to inhibit negative selection and promote thymocyte survival.
Loss of NCOR1 is associated with Thyroid Cancer.
These studies support a model in which NCOR1/2 mediates direct Retinoic acid-dependent repression of Fgf8 (show FGF8 Antibodies) in caudal (show CAD Antibodies) progenitors in order to control somitogenesis.
demonstrate that the binding of HDAC3 (show HDAC3 Antibodies) to IR nGREs in vivo is mediated through interaction with SMRT/NCoR1
GR SUMOylation site is mandatory for the formation of a GR-small ubiquitin-related modifiers (SUMOs)-SMRT/NCoR1-HDAC3 (show HDAC3 Antibodies) repressing complex, which is indispensable for NF-kappaB (show NFKB1 Antibodies)/AP1 (show JUN Antibodies)-mediated GC-induced tethered indirect transrepression in vitro
dexamethasone, a powerful regulator of metabolism and of adipocyte differentiation, confers this change in NCoR mRNA splicing in cultured adipocytes.
Phosphorylation-mediated recruitment switch of NCoR1 between nuclear receptor subsets provides a mechanism by which corepressors can selectively modulate liver energy metabolism during the fasting-feeding transition
NCOR1 protein levels were significantly reduced.
corepressor specificity exists in vivo and that nuclear receptor corepressor1 is the principal regulator of thyroid hormone (show PTH Antibodies) action
verexpression of COPS5 (show COPS5 Antibodies), through its isopeptidase activity, leads to ubiquitination and proteasome-mediated degradation of NCoR, a key corepressor for ERalpha (show ESR1 Antibodies) and tamoxifen-mediated suppression of ERalpha (show ESR1 Antibodies) target genes.
the NCOR/HDAC3 (show HDAC3 Antibodies) complex is a major suppressor of differentiation in rhabdomyosarcoma
Authors previously shown that Nuclear Receptor Corepressor 1 (NCoR) and the thyroid hormone receptor (show THRA Antibodies) beta1 (TRbeta (show TXNRD2 Antibodies)) inhibit tumor invasion. Here they show that these molecules repress VEGF-C (show VEGFC Antibodies) and VEGF-D (show Figf Antibodies) gene transcription in breast cancer cells, reducing lymphatic vessel density and sentinel lymph node invasion in tumor xenografts.
Nuclear Receptor Corepressor 1 is an important transcriptional regulator that interacts with nuclear receptors and other transcription factors. Recent results have shown the presence of inactivating mutations or deletions of the nuclear receptor corepressor 1 gene in human tumors.
NCOR1 function declines with prostate cancer progression. Reduction in NCOR1 levels causes bicalutamide resistance in LNCaP cells and compromises response to bicalutamide in mouse prostate in vivo
USP44 (show USP44 Antibodies) contributes to N-CoR functions in regulating gene expression and is required for efficient invasiveness of triple-negative breast cancer cells.
PDCD2 (show PDCD2 Antibodies) and NCoR1 may act as tumor suppressors in Gastrointestinal stromal tumors cells through the Smad (show SMAD1 Antibodies) signaling pathway.
Data suggest that complexes of HDAC3-H1.3 with NCOR1 and NCOR2/SMRT accumulate on chromatin in synchronized HeLa cells in late G2 phase and mitosis; deacetylation activity of HDAC3 is activated via phosphorylation of Ser-424 by CK2 only in mitosis.
NCoR depletion enhances cancer cell invasion and increases tumor growth and metastatic potential.
loss of nuclear NCoR results in upregulation of a specific cancer-related genetic signature, and is significantly associated with malignant melanoma progression.
Data provide in vivo evidence for targeted recruitment of N-CoR/SMRT-TBLR1 (show TBL1XR1 Antibodies) complexes by unliganded thyroid hormone (show PTH Antibodies) receptors in transcriptional repression during vertebrate development
This gene encodes a protein that mediates ligand-independent transcription repression of thyroid-hormone and retinoic-acid receptors by promoting chromatin condensation and preventing access of the transcription machinery. It is part of a complex which also includes histone deacetylases and transcriptional regulators similar to the yeast protein Sin3p. This gene is located between the Charcot-Marie-Tooth and Smith-Magenis syndrome critical regions on chromosome 17. Alternate splicing results in multiple transcript variants. Pseudogenes of this gene are found on chromosomes 17 and 20.
nuclear receptor corepressor 1
, nuclear receptor co-repressor 1
, retinoid X receptor interacting protein 13
, retinoid X receptor-interacting protein 13
, N-Cor/SMRT corepressor Rip13
, N-Cor/SMRT corepressor, Rip13
, thyroid hormone- and retinoic acid receptor-associated corepressor 1