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anti-Human FAS Antibodies:
anti-Mouse (Murine) FAS Antibodies:
anti-Rat (Rattus) FAS Antibodies:
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Mouse (Murine) Polyclonal FAS Primary Antibody for IHC (fro), IHC (p) - ABIN3044338
Jiang, Li, Zhou, Wang, Zhang, Wang: Colistin-induced apoptosis in PC12 cells: involvement of the mitochondrial apoptotic and death receptor pathways. in International journal of molecular medicine 2014
Show all 14 Pubmed References
Rat (Rattus) Polyclonal FAS Primary Antibody for WB - ABIN3042386
Liu, Shan, Dong, Liu, Ma, Liu: Combined early fluid resuscitation and hydrogen inhalation attenuates lung and intestine injury. in World journal of gastroenterology 2013
Show all 13 Pubmed References
Human Polyclonal FAS Primary Antibody for IHC (p), WB - ABIN3042387
Qin, Ma, Yang, Hu, Zhou, Fu, Tian, Liu, Xu, Shen: A Triterpenoid Inhibited Hormone-Induced Adipocyte Differentiation and Alleviated Dexamethasone-Induced Insulin Resistance in 3T3-L1 adipocytes. in Natural products and bioprospecting 2015
Show all 13 Pubmed References
Human Monoclonal FAS Primary Antibody for IHC (fro), IF - ABIN1106610
Vega, Jazirehi, Huerta-Yepez, Bonavida: Rituximab-induced inhibition of YY1 and Bcl-xL expression in Ramos non-Hodgkin's lymphoma cell line via inhibition of NF-kappa B activity: role of YY1 and Bcl-xL in Fas resistance and chemoresistance, respectively. in Journal of immunology (Baltimore, Md. : 1950) 2005
Show all 16 Pubmed References
Human Monoclonal FAS Primary Antibody for FACS - ABIN5541252
Hata, Matsuzaki, Takeya, Yoshida, Sonoki, Nagasaki, Kuribayashi, Kawano, Takatsuki: Expression of Fas/Apo-1 (CD95) and apoptosis in tumor cells from patients with plasma cell disorders. in Blood 1995
Show all 12 Pubmed References
Human Monoclonal FAS Primary Antibody for FACS - ABIN5541251
Boirivant, Pica, DeMaria, Testi, Pallone, Strober: Stimulated human lamina propria T cells manifest enhanced Fas-mediated apoptosis. in The Journal of clinical investigation 1997
Show all 12 Pubmed References
Mouse (Murine) Monoclonal FAS Primary Antibody for CyTox, FACS - ABIN1177304
Enari, Hug, Nagata: Involvement of an ICE-like protease in Fas-mediated apoptosis. in Nature 1995
Show all 9 Pubmed References
Mouse (Murine) Monoclonal FAS Primary Antibody for FACS - ABIN2689463
Hiromatsu, Aoki, Makino, Matsumoto, Mizuochi, Gotoh, Nomoto, Ogasawara, Nagata, Yoshikai: Increased Fas antigen expression in murine retrovirus-induced immunodeficiency syndrome, MAIDS. in European journal of immunology 1994
Show all 9 Pubmed References
Human Polyclonal FAS Primary Antibody for IHC, IHC (p) - ABIN448412
Horuz, Göktaş, Çetinel, Akça, Aydın, Ekici, Albayrak, Sarıca: Role of TNF-associated cytokines in renal tubular cell apoptosis induced by hyperoxaluria. in Urolithiasis 2013
Show all 5 Pubmed References
Mouse (Murine) Monoclonal FAS Primary Antibody for CyTox, FACS - ABIN2689461
Kägi, Vignaux, Ledermann, Bürki, Depraetere, Nagata, Hengartner, Golstein: Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity. in Science (New York, N.Y.) 1994
Show all 5 Pubmed References
These results demonstrated that overexpression of ING4 (show ING4 Antibodies) can induce the apoptosis of melanoma cells and CD3 (show CD3 Antibodies)+ T cells through signaling pathways such as the Fas/FasL (show FASL Antibodies) pathway, and that ING4 (show ING4 Antibodies) gene therapy for melanoma treatment is a novel approach.
Tag7 (show PGLYRP1 Antibodies) activates lymphocytes capable of Fasl (show FASL Antibodies)-Fas-dependent contact killing of virus-infected cells.
These results indicated that cMyc (show MYC Antibodies) and Fas regulated the sensitivity of A549 cells to irradiation by regulating caspase8-mediated Bid (show BID Antibodies) activation and the subsequent association with the mitochondrial pathway of apoptosis.
this study characterized in juvenile systemic lupus erythematosus a distinct profile from adult SLE that comprises increased sFas, sTRAIL, and reduced sFasL (show FASL Antibodies), notably in patients with active disease and with nephritis.
results from the current study showed that the association of FAS-670A/G SNP with idiopathic azoospermia was not found in a population of men with idiopathic infertility.
The Btk (show BTK Antibodies)-dependent PIP5K1gamma lipid kinase activation by Fas counteracts FasL (show FASL Antibodies)-induced cell death.
Study identify genes highly expressed in Kras knockout lung cancer cells, including the Fas receptor gene. Antibodies that activate Fas receptor selectively induced apoptosis in Kras-independent lung cancer cells suggesting that FAS is involved in KRAS-related drug resistance.
The authors observed differential expression of CD95(Fas) in CD27 (show CD27 Antibodies)(+) B-cells from cirrhotic patients that was inversely correlated with peripheral CD27 (show CD27 Antibodies)(+) B-cell frequency. They conclude that peripheral CD27 (show CD27 Antibodies)(+) memory B-cells in cirrhosis exhibit increased sensitivity to Fas-induced apoptosis in an activation-dependent manner to which endotoxin contributes, associated with reduced frequency of circulating memory B-cells.
the analysis of mRNA level showed that disease progression is associated with significantly increased expression of FasR and/or FasL (show FASL Antibodies). In conclusion, our observation seems to confirm that spherical model of cancer lines is more reliable for some sophisticated analysis because of their greater resemblance to the CSCs from human CRC (show CALR Antibodies) samples in comparison to commonly used adherent cells
study indicates FAS-FASL (show FASL Antibodies) promoter SNPs may promote the production of cross-reactive anti-ganglioside antibodies in GBS (show GNB5 Antibodies)
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (show PTGES Antibodies)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF (show TNF Antibodies)) and Fas were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
Results did not support that K8/K18 (show KRT18 Antibodies) filaments influence the expression of Fas on the surface of luteal cells.
activation of the Fas pathway and presence of FSH (show BRD2 Antibodies) during in vitro maturation increased the incidence of apoptosis in cumulus cells
demonstrated for the first time that normal ejaculated spermatozoa express Fas antigen (show FASL Antibodies); data showed that a large percentage of normal ejaculated spermatozoa of fertile bulls are immunocytochemically positive for Fas
identification of 14-3-3zeta (show YWHAZ Antibodies) as a novel phosphocofilin binding protein involved in the maintenance of the cellular phosphocofilin pool
Both Sharpin (show SHARPIN Antibodies)/Fas and Sharpin (show SHARPIN Antibodies)/Fasl (show FASL Antibodies) compound mutant mice developed an auto-inflammatory phenotype similar to that seen in Sharpin (show SHARPIN Antibodies) null mice, indicating that initiation of apoptosis by FAS signalling is likely not involved in the pathogenesis of this disease.
leucine deprivation induces the expression of miR (show MLXIP Antibodies)-212-5p in a GCN2 (show EIF2AK4 Antibodies)/ATF4 (show ATF4 Antibodies)-dependent manner. miR (show MLXIP Antibodies)-212-5p suppresses lipid accumulation in liver by targeting FAS and SCD1 (show SCD Antibodies) under both normal diet and high-fat diet conditions.
Our data show that loss of Fas activity strongly affects the early development of atopic dermatitis (AD) by leading to Th2-dominant inflammation characterized by dermal infiltration of CD4 (show CD4 Antibodies)+ T cells, neutrophils and increased skin expression of Th2 cytokines.However, Fas/FasL (show FASL Antibodies)-apoptotic pathway is also involved in restricting tissue remodelling and dermal fibrosis during AD.
Hrd1 (show SYVN1 Antibodies)-null B cells exhibited high Fas expression during activation and rapidly underwent Fas-mediated apoptosis, which could be largely inhibited by FasL (show FASL Antibodies) neutralization. Fas mutation in Hrd1 (show SYVN1 Antibodies) KO mice abrogated the increase in B-cell AICD. We identified Hrd1 (show SYVN1 Antibodies) as the first E3 ubiquitin ligase (show MUL1 Antibodies) of the death receptor Fas and Hrd1 (show SYVN1 Antibodies)-mediated Fas destruction as a molecular mechanism in regulating B-cell immunity.
FAS contributes to mitochondrial dysfunction, steatosis development, and insulin (show INS Antibodies) resistance under high fat diet.
anti-apoptotic molecules BclxL (show BCL2L1 Antibodies) and Bcl-2 (show BCL2 Antibodies) and the pro-apoptotic factors BAD and BID (show BID Antibodies) cooperate to promote migration of triple-negative breast cancer cells stimulated with cl-CD95L (show FASL Antibodies).
These findings reveal a role for MOAP-1 (show MOAP1 Antibodies) in Fas signaling in the liver by promoting MTCH2 (show MTCH2 Antibodies)-mediated tBid recruitment to mitochondria.
The in vivo delivery of CRISPR/Cas9 could maintain liver homeostasis and protect hepatocytes from Fas-mediated cell apoptosis in the fulminant hepatic failure model.
This study demonstrated that Ischemic neurons release sFasL (show FASL Antibodies), which contributes to M1-microglial polarization.
Fas was expressed on fetal pig pancreatic cells, both beta and non-beta cells, and the level of expression could be upregulated by exposure to interleukin 1beta
The expression of FAS and FAS ligand (show FASL Antibodies) in splenic macrophages co-infected with porcine circovirus 2 and porcine reproductive and respiratory syndrome virus is reported
The present results may provide some insights to understand the role of Fas/FasL (show FASL Antibodies) in the spinal cord but not motor cortex with neuronal apoptosis and neuroplasticity in monkeys subjected to hemisection spinal cord injury.
Ligustrazine has notable protective effects on pulmonary ischemia/reperfusion injury inhibiting Fas/FasL (show FASL Antibodies) mRNA express in lung tissue and decreasing apoptosis.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor contains a death domain. It has been shown to play a central role in the physiological regulation of programmed cell death, and has been implicated in the pathogenesis of various malignancies and diseases of the immune system. The interaction of this receptor with its ligand allows the formation of a death-inducing signaling complex that includes Fas-associated death domain protein (FADD), caspase 8, and caspase 10. The autoproteolytic processing of the caspases in the complex triggers a downstream caspase cascade, and leads to apoptosis. This receptor has been also shown to activate NF-kappaB, MAPK3/ERK1, and MAPK8/JNK, and is found to be involved in transducing the proliferating signals in normal diploid fibroblast and T cells. Several alternatively spliced transcript variants have been described, some of which are candidates for nonsense-mediated mRNA decay (NMD). The isoforms lacking the transmembrane domain may negatively regulate the apoptosis mediated by the full length isoform.
APO-1 cell surface antigen
, CD95 antigen
, Delta Fas/APO-1/CD95
, FAS 827dupA
, FASLG receptor
, Fas (TNF receptor superfamily, member 6)
, Fas AMA
, TNF receptor superfamily member 6
, apoptosis antigen 1
, apoptosis-mediating surface antigen FAS
, tumor necrosis factor receptor superfamily member 6
, tumor necrosis factor receptor superfamily, member 6
, Fas antigen
, 14-3-3 protein zeta/delta
, factor activating exoenzyme S
, protein kinase C inhibitor protein 1
, Fas (TNF receptor superfamily member)
, apo-1 antigen
, Fas antigen (ATP1)
, Fas receptor
, Tumor necrosis factor receptor superfamily, member 6
, apoptosis (APO-1) antigen 1 ( FAS ), member 6
, Fas receptor CD95