Browse our anti-NFKB1 (NFKB1) Antibodies

Human Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. These data suggest that TLR2 activation is involved in the induction of IL-15 (show IL15 Antibodies) production by primary Sjogren's syndrome salivary gland epithelial cells and promotes inflammation through NF-kappaB activation.

2. Lipid oxidation product 4-hydroxy-2-nonenal is at the crossroads of NF-kappaB pathway and anti-apoptotic Bcl2 (show BCL2 Antibodies) expression. (Review)

3. NKILA is a vital determinant of tongue squamous cell carcinoma cell migration and invasion; the NF-kappaB signaling pathway mediates this effect

4. Data show that p50 (show CD40 Antibodies) is important as a unique mechanism of R-CHOP (show DDIT3 Antibodies)-resistance in activated B-cell-like diffuse large B-cell lymphoma and in patients without TP53 (show TP53 Antibodies) mutations.

5. GGN played a tumor-promoting role in bladder cancer through regulation of NFkappaB/caspase3-mediated apoptosis signaling.

6. Together, these data show that RIP2 (show ARHGEF28 Antibodies) promotes survival of breast cancer cells through NF-kappaB activation and that targeting RIP2 (show ARHGEF28 Antibodies) may be therapeutically beneficial for treatment of TNBC.

7. High NF-kappa-B expression is associated with lung cancer.

8. Studies indicate that NF-kappa B (NF-kappaB) is of particular interest to the space environment, as it is associated with many of the spaceflight-related health consequences [review].

9. In summary, we demonstrate that ADAMTS18 (show ADAMTS18 Antibodies) silencing in breast cancer is significantly correlated with promoter CpG methylation. ADAMTS18 (show ADAMTS18 Antibodies) acts as an antagonist of AKT (show AKT1 Antibodies) and NF-kappaB signaling, further suppressing EMT (show ITK Antibodies) and metastasis of breast cancer cells.

10. novel EGFR (show EGFR Antibodies)-NF-kappaB-FOXC1 (show FOXC1 Antibodies) signaling axis that is critical for BLBC cell function

Cow (Bovine) Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. These results indicated that docosahexaenoic acid may attenuate lipopolysaccharide-stimulated inflammatory response in bovine mammary epithelial cells by suppressing NF-kappaB activation through a mechanism partly dependent on PPARgamma (show PPARG Antibodies) activation.

2. Taken together, Staphylococcus aureus induces TGF-beta1 (show TGFB1 Antibodies) and bFGF (show FGF2 Antibodies) expression through the activation of AP-1 (show JUN Antibodies) and NF-kappaB in bovine mammary gland fibroblasts.

3. Data indicate the involvement of PKC-alpha (show PKCa Antibodies) in proMMP-2 activation and inhibition of TIMP-2 (show TIMP2 Antibodies) expression by NF-kappaB-MT1-MMP (show MMP14 Antibodies)-dependent and -independent pathway.

4. MMP-1 (show MMP1 Antibodies) promotes VEGFR2 (show KDR Antibodies) expression and proliferation of endothelial cells through stimulation of PAR-1 (show F2R Antibodies) and activation of NF-kappaB

5. dynamic compression stimulates cell proliferation and proteoglycan (show Vcan Antibodies) synthesis in the presence of IL-1beta (show IL1B Antibodies) and/or inhibitors of the MAPKs and NFkappaB and AP-1 (show JUN Antibodies) signalling pathways

Rhesus Monkey Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. TRIM5alpha (show TRIM5 Antibodies) represses HIV-1 LTR promoter activity by negatively regulating TAK1 (show NR2C2 Antibodies)/TAB1 (show TAB1 Antibodies)/TAB2 (show TAB2 Antibodies)/TAB3 (show TAB3 Antibodies)-complex-mediated NF-kappaB activation.

Pig (Porcine) Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. Porcine parvovirus virus infection significantly induced IL-6 (show IL6 Antibodies) expression and this induction depended on NF-kappaB activation and TLR9 (show TLR9 Antibodies) signaling pathways in PK-15 cell.

2. This study showed that porcine epidemic diarrhea virus inhibited NF-kappaB activity and nsp1 was a potent NF-kappaB antagonist for suppression of both interferon (show IFNA Antibodies) and early production of pro-inflammatory cytokines.

3. inhibition of NF-kappaB increases autophagy via JNK (show MAPK8 Antibodies) signaling, and promotes steroidogenesis in porcine granulosa cells

4. SIRT1 (show SIRT1 Antibodies), p53 (show TP53 Antibodies) and NF-kappaB are involved in the control of both the proliferation and the apoptosis of ovarian cells.

5. porcine CD74 (show CD74 Antibodies) significantly enhanced the inflammatory response by regulating the NF-kappaB signalling pathway during PCV2 infection, which suggests that porcine CD74 (show CD74 Antibodies) may be implicated in the pathogenesis of PCV2 infection.

6. These results suggest that trans-10, cis (show CISH Antibodies)-12-conjugated linoleic acid can modulate TNF-alpha (show TNF Antibodies) production and NF-kappa B expression by a PPARgamma (show PPARG Antibodies)-dependent pathway in porcine peripheral blood mononuclear cells.

Mouse (Murine) Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. miR-17 approximately 92 family clusters have a role in iNKT cell ontogenesis via modulation of TGF-beta (show TGFB1 Antibodies) signaling

2. NF-kappaB1-mediated signaling regulates the development of gastric mucosal pathology following TAM (show CCNA1 Antibodies) administration.

3. that TG2 (show TGM2 Antibodies) depletion increases nuclear factor-kappaB (NF-kappaB) signaling

4. LRRK1 physically interacted and potently synergized with CARMA1 (show CARD11 Antibodies) to enhance NF-kappaB activation.

5. NLRC5 (show NLRC5 Antibodies) knockout mice fed with high fat showed accelerated fibrosis and inflammation response by promoting alpha-SMA (show SMN1 Antibodies), Collagen I, Collagen III, TLR4 (show TLR4 Antibodies)/MyD88 (show MYD88 Antibodies), phosphorylated IKKalpha (show CHUK Antibodies), IkappaBalpha (show NFKBIA Antibodies) and NF-kappaB expression.

6. The results obtained in the present study demonstrate for the first time that S100A8 (show S100A8 Antibodies) as well as MyD88 (show MYD88 Antibodies) and NF-B are activated by T3 and that these molecules are directly involved in the thyroid hormone (show PTH Antibodies)-induced cardiac hypertrophic response. These data suggest that one of the mechanisms underlying T3-dependent cardiac hypertrophy/failure may involve the activation of an inflammatory pathway.

7. Our data suggest that rCC16 suppresses LPS (show TLR4 Antibodies)-mediated inflammatory mediator TNF-alpha (show TNF Antibodies), IL-6 (show IL6 Antibodies), and IL-8 (show IL8 Antibodies) production by inactivating NF-kappaB and p38 MAPK (show MAPK14 Antibodies) but not AP-1 (show JUN Antibodies) in RAW264.7 cells.

8. Our data indicate for the first time that the inflammasome is involved in the inflammatory response and cell death in hypoxia-induced beta cells through the ROS (show ROS1 Antibodies)-TXNIP (show TXNIP Antibodies)-NLRP3 (show NLRP3 Antibodies) axis in vitro. This provides new insight into the relationship between hypoxia and inflammation in T2D.

9. Chronic overnutrition and astrocytic IKKbeta (show IKBKB Antibodies)/NF-kappaB upregulation similarly impaired astrocytic plasticity, leading to sustained shortening of high-order processes. In physiology, astrocytic IKKbeta (show IKBKB Antibodies)/NF-kappaB upregulation resulted in early-onset effects, including glucose intolerance and blood pressure rise, and late-onset effects, including body weight and fat gain.

10. Overall, these results suggest that p53 (show TP53 Antibodies) is involved in improving insulin (show INS Antibodies) sensitivity of hepatic cells via inhibition of mitogen-activated protein kinases (MAPKs) and NF-kappaB pathways.

Zebrafish Nuclear Factor of kappa Light Polypeptide Gene Enhancer in B-Cells 1 (NFKB1) interaction partners

1. Study indicate an essential role of NF-kappaB in gonadal differentiation.