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Down-regulation of mitochondrial ATPase (show ATP5E ELISA Kits) by hypermethylation mechanism in chronic myeloid leukemia (show BCL11A ELISA Kits) is associated with multidrug resistance.
Nitration of tyrosine residues 368 and 345 in the beta-subunit (show POLG ELISA Kits) elicits FoF1-ATPase (show DNAH8 ELISA Kits) activity loss.
This gene encodes a subunit of mitochondrial ATP synthase. Mitochondrial ATP synthase catalyzes ATP synthesis, utilizing an electrochemical gradient of protons across the inner membrane during oxidative phosphorylation. ATP synthase is composed of two linked multi-subunit complexes: the soluble catalytic core, F1, and the membrane-spanning component, Fo, comprising the proton channel. The catalytic portion of mitochondrial ATP synthase consists of 5 different subunits (alpha, beta, gamma, delta, and epsilon) assembled with a stoichiometry of 3 alpha, 3 beta, and a single representative of the other 3. The proton channel seems to have nine subunits (a, b, c, d, e, f, g, F6 and 8). This gene encodes the b subunit of the proton channel.
ATP synthase subunit B
, ATP synthase, H+ transporting, mitochondrial F0 complex, subunit B1
, ATP synthase H+ transporting mitochondrial F0
, ATP synthase subunit b, mitochondrial
, ATP synthase, H+ transporting, mitochondrial F0 complex, subunit b
, ATPase subunit b
, ATP synthase B chain, mitochondrial
, H+-ATP synthase subunit b
, cell proliferation-inducing protein 47
, ATP synthase b-subunit