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Human Polyclonal LHCGR Primary Antibody for ELISA, WB - ABIN4229674
Yung, Maman, Ophir, Rubinstein, Barzilay, Yerushalmi, Hourvitz: Progesterone antagonist, RU486, represses LHCGR expression and LH/hCG signaling in cultured luteinized human mural granulosa cells. in Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology 2013
Show all 4 Pubmed References
Human Polyclonal LHCGR Primary Antibody for IHC (p) - ABIN270629
Yung, Aviel-Ronen, Maman, Rubinstein, Avivi, Orvieto, Hourvitz: Localization of luteinizing hormone receptor protein in the human ovary. in Molecular human reproduction 2014
Show all 3 Pubmed References
Human Polyclonal LHCGR Primary Antibody for IF (p), IHC (p) - ABIN873328
Yang, Dou, Zhang, Gu, Lv, DU, Ba, Mu, Lu: Increased 3β-hydroxysteroid dehydrogenase 2 and 17α-hydroxylase activities in a virilized adolescent female with adrenal adenoma: A case report. in Experimental and therapeutic medicine 2016
Show all 2 Pubmed References
Human Monoclonal LHCGR Primary Antibody for FACS, IF - ABIN5582545
Pidoux, Gerbaud, Tsatsaris, Marpeau, Ferreira, Meduri, Guibourdenche, Badet, Evain-Brion, Frendo: Biochemical characterization and modulation of LH/CG-receptor during human trophoblast differentiation. in Journal of cellular physiology 2007
Mouse (Murine) Polyclonal LHCGR Primary Antibody for IF (p), IHC (p) - ABIN730558
Umehara, Kawai, Kawashima, Tanaka, Okuda, Kitasaka, Richards, Shimada: The acceleration of reproductive aging in Nrg1flox/flox;Cyp19-Cre female mice. in Aging cell 2017
Rat (Rattus) Polyclonal LHCGR Primary Antibody for WB - ABIN336206
Berndt, Perrier dHauterive, Blacher, Péqueux, Lorquet, Munaut, Applanat, Hervé, Lamandé, Corvol, van den Brûle, Frankenne, Poutanen, Huhtaniemi, Geenen, Noël, Foidart: Angiogenic activity of human chorionic gonadotropin through LH receptor activation on endothelial and epithelial cells of the endometrium. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006
genetic association/pharmacogenetic studies in population in India: Data suggest that need for (and dose required of) fertility agent recombinant LH in addition to fertility agent recombinant FSH (show BRD2 Antibodies) is associated with SNP in LHCGR in women undergoing ovarian stimulation in conjunction with in vitro fertilization. (LH = luteinizing hormone; FSH (show BRD2 Antibodies) = follicle stimulating hormone)
hCG (show CGA Antibodies) and its receptor, LH/CGR, are expressed in numerous sites of the reproductive tract, both in gonadal and extra-goanadal tissues, promoting oocyte maturation, fertilization, implantation and early embryo development
The reduced fertilisation and pregnancy rate was associated with a lower LH receptor density and a lack of essential down-regulation of the FSH (show BRD2 Antibodies) and LH receptor.
This work demonstrates that the expression of FSHR (show FSHR Antibodies) and LHCGR can be induced in hGL5 cells but that the FSHR (show FSHR Antibodies)-dependent cAMP/PKA pathway is constitutively silenced, possibly to protect cells from FSHR (show FSHR Antibodies)-cAMP-PKA-induced apoptosis.
Inactivating mutations of the LHCGR gene may be a common cause of 46,XY primary amenorrhea.
data demonstrate that the majority of LHR mutations lead to intracellular retention and highlight the potential for novel pharmacological chaperone therapeutics that can "rescue" expression/function of retained mutant GPCRs.
expression and activation of LHCGR and ARF6 (show ARF6 Antibodies) are up-regulated in GC from PCOS women but the mechanism of agonist-induced LHCGR internalization is unaltered
We showed that GNRHR and LHCGR were highly expressed in some wildtype aldosterone-producing adenoma samples, and that they positively correlated with GnRH-stimulated aldosterone production.
Mutation in the LHCGR gene is associated with Testotoxicosis.
The luteinizing hormone/human chorionic gonadotrophin receptor (LHCGR) variant N312S and the follicle-stimulating hormone receptor (FSHR (show FSHR Antibodies)) variant N680S can be utilized for the prediction of pregnancy chances in women undergoing IVF (show SCN5A Antibodies).
Activating mutations in LHCGR cause familial male-limited precocious puberty
Study tested for the first time a role of ZFP36L2 (show ZFP36L2 Antibodies) in the decay of LHR mRNA, when transcription was inhibited; results of our cell-based assay support the conclusion that LHR mRNA expression is controlled post-transcriptionally by ZFP36L2 (show ZFP36L2 Antibodies).
FSHR (show FSHR Antibodies) and LHR proteins are significantly upregulated in CCs (show CCS Antibodies) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (show ESR1 Antibodies), LHR, and FSHR (show FSHR Antibodies)
Data suggest that persistent cAMP signals from internalized luteinizing hormone receptor (LH receptors) contribute to transmitting LH effects inside follicle cells and ultimately to the oocyte.
Demonstrate the presence of LH receptors. Activation resulted in a dose-dependent increase in glucose-induced release of insulin (show INS Antibodies).
LHCGR signaling in regulating the Ahr (show AHR Antibodies) message involves protein kinase A pathway and is attributable to decreased transcription rate.
Data from mutant mouse strain (gain-of-function mutation in LHR, D578G; most common mutation found in familial male-limited precocious puberty) confirm that LHR is critical for male steroidogenesis, gametogenesis, and Leydig cell development.
LH/hCG (show CGA Antibodies) tightly up-regulates MKP-3 (show DUSP6 Antibodies) which in turn, dephosphorylates ERK1/2 and drives p21 (show D4S234E Antibodies) expression.
Data suggest that Lhcgr in endometrium and luteinizing hormone in blastocyst are involved in embryo/blastocyst implantation; expression of Lhcgr is up-regulated in endometrial epithelium in estrus cycle at time of implantation readiness (estrus).
The outcomes of the present study support a dynamic multi-facetted regulation of LHR during pre-translation.
expression of LHR mRNA in bovine granulosa cells is established after follicle deviation, and the lower abundance of LRBP (show MVK Antibodies) mRNA after the expected time of deviation may contribute to greater expression of LHR in the bovine dominant follicle
These results suggested an acute regulation of INSL3 (show INSL3 Antibodies) by luteinizing hormone (LH) because INSL3 (show INSL3 Antibodies) concentrations increased immediately after endogenous and exogenous LH stimulation.
INVESTIGATION OF STAT5A (show STAT5A Antibodies), FSHR (show FSHR Antibodies) AND LHR GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
These findings strongly support the concept that IGF-1 (show IGF1 Antibodies) upregulates LHR expression in granulosa cells and that IGF-1 (show IGF1 Antibodies) is required for determining which follicle becomes dominant and acquires ovulatory capacity.
LHCGR mRNA expression in granulosa cells was significantly higher in large antral follicles than in cysts, and not detected in granulosa cells of small and medium antral follicles.
The luteinizing hormone receptor [LHR] splicing pattern is complex in bovine Leydig cells, and expression of full-length LHR and isoforms A and B changes when induced with LH.
The LHCGR gene is a potential marker for superovulation response and can be used to predict the most appropriate dose of FSH (show BRD2 Antibodies) for superovulation in Chinese Holstein cows.
Dominant follicles experience a reduction in FSH (show BRD2 Antibodies) dependence (diminished expression of FSHR (show FSHR Antibodies)), but acquire increased LH dependence (enhanced expression of LHCGR) as they grow during the low FSH (show BRD2 Antibodies) milieu of follicular waves.
Three single nucleotide polymorphisms in LHCGR were significantly associated with variations in cattle fertility and production traits.
Data show that double mutation of follicle-stimulating hormone receptor (fshr (show FSHR Antibodies)) and luteinizing hormone receptor (lhcgr) resulted in infertile males
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr (show FSHR Antibodies) and lhcgr.
In porcine ovaries, MAb found 6 distinct LHR bands migrating at approximately 92, 80, 68, 59, 52 & 48 kDa. There is a possible role for LHR in the development of abnormal pregnancy, pelvic floor disorders & Alzheimer's disease.
This gene encodes the receptor for both luteinizing hormone and choriogonadotropin. This receptor belongs to the G-protein coupled receptor 1 family, and its activity is mediated by G proteins which activate adenylate cyclase. Mutations in this gene result in disorders of male secondary sexual character development, including familial male precocious puberty, also known as testotoxicosis, hypogonadotropic hypogonadism, Leydig cell adenoma with precocious puberty, and male pseudohermaphtoditism with Leydig cell hypoplasia.
, lutropin-choriogonadotropic hormone receptor
, lutropin/choriogonadotropin receptor
, luteinizing hormone receptor
, LH receptor
, luteinizing hormone receptor 2 protein
, luteinizing hormone receptor precursor variant 1
, luteinizing hormone receptor precursor variant 2
, lutropin-choriogonadotropin receptor
, luteinizing hormone/choriogonadotropin receptor
, lutropin-choriogonadotropic hormone receptor-like