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anti-Human NFYA Antibodies:
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Suggest NF-YAs and lamin A (show LMNA Antibodies) expression levels as novel potential biomarkers useful to identify G1 endometrial carcinoma patients with risk of recurrence.
increased expression of NF-YA may promote a malignant phenotype in OS cells via modulation of FASN (show FASN Antibodies) expression.
The findings indicate that overexpression of NF-YA contributes to tumor angiogenesis through EZH2 (show EZH2 Antibodies)-STAT3 (show STAT3 Antibodies) signaling in human melanoma cells, highlighting NF-YA as a potential therapeutic target in human melanoma.
Data indicate that specific cancer-driving nodes are generally under NF-YA/B control.
our results indicate that NF-YA alternative splicing is an influential muscle cell determinant, through direct regulation of selected cell cycle blocking genes, and, directly and indirectly, of muscle-specific (show EIF3K Antibodies) transcription factors
enhanced CDCA8 (show CDCA8 Antibodies) promoter activities by NF-Y overexpression and reduced CDCA8 (show CDCA8 Antibodies) transcription by NF-Y knockdown further verified that NF-Y is a positive regulator of CDCA8 (show CDCA8 Antibodies) transcription.
Data show that Zinc-fingers and homeoboxes 2 (ZHX2 (show ZHX2 Antibodies)) represses nuclear transcription factor Y alpha (NF-Y)-mediated activation of multidrug resistance 1 (MDR1 (show TBC1D9 Antibodies)) transcription.
NF-Y inhibits NT2/D1 cell growth in p53 (show TP53 Antibodies)-independent manner and decreases SOX2 (show SOX2 Antibodies) expression.
Stimulation of the lymphocytes with phytohemagglutinin, restores normal OGG1 (show OGG1 Antibodies) levels and repair rates. MAP kinase (show MAPK1 Antibodies) c-Jun N-terminal kinase (JNK) and the recruitment of the transcription factor NFYA to the promoter region of OGG1 (show OGG1 Antibodies) are shown to be involved.
Association of p21 (show CDKN1A Antibodies) with NF-YA suppresses the expression of PLK1 (show PLK1 Antibodies) and prevents mitotic death in response to DNA damage.
Interaction of C/EBP-beta (show CEBPB Antibodies) and NF-Y factors constrains activity levels of the nutritionally controlled promoter IA expressing the acetyl-CoA carboxylase-alpha (show ACACA Antibodies) gene in cattle.
Data show that nuclear factor Y (NF-Y) binds to the inverted CCAAT element (ICE) in the Fatty acid synthase (Fasn (show FASN Antibodies)) promoter specifically in refeeding states.
findings may point to a possible role of NF-YA in stress conditions that occur in chronic obesity, ER stress might be involved in the pathogenesis of obesity through NF-YA depletion.
we conclude that NF-Y and YY1 (show YY1 Antibodies) are important for the basal transcription of Pcyt2 (show PCYT2 Antibodies) and that NF-Y is involved in the inhibitory effects of 25-HC on Pcyt2 (show PCYT2 Antibodies) transcription.
these results indicate that the transcription factor NF-Y regulates the proximal promoter activity of mouse Col11a1 (show COL11A1 Antibodies) gene in chondrocytes
these results raise an exciting possibility that targeting CDK1 (show CDK1 Antibodies) or NF-Y in the diseased heart may inhibit fibrosis and subsequently confer cardioprotection.
NF-Ya is a functional activator of Bmal1 (show ARNTL Antibodies) transcription and it cooperates with Sp1 (show SP1 Antibodies) and Rev-Erbalpha (show NR1D1 Antibodies) to generate the daily cycle of Bmal1 (show ARNTL Antibodies) expression.
Sp1 (show SP1 Antibodies), CREB (show CREB1 Antibodies), HNF-1 (show HNF1A Antibodies), and NF-Y, known to be responsive to hormones and diet, regulate NAGS (show NAGS Antibodies) transcription
results indicate nuclear transcription factor NF-Y as a pivotal upstream participant in a regulatory network necessary for the preservation of cycling hematopoietic stem cell.
Findings establish that NF-Y acts upstream of E2F1 (show E2F1 Antibodies) in p53 (show TP53 Antibodies)-mediated apoptosis.
Transcriptional regulation of myeloid differentiation primary response (MyD) genes during myeloid differentiation is mediated by nuclear factor Y.
The protein encoded by this gene is one subunit of a trimeric complex, forming a highly conserved transcription factor that binds to CCAAT motifs in the promoter regions in a variety of genes. Subunit A associates with a tight dimer composed of the B and C subunits, resulting in a trimer that binds to DNA with high specificity and affinity. The sequence specific interactions of the complex are made by the A subunit, suggesting a role as the regulatory subunit. In addition, there is evidence of post-transcriptional regulation in this gene product, either by protein degradation or control of translation. Further regulation is represented by alternative splicing in the glutamine-rich activation domain, with clear tissue-specific preferences for the two isoforms.
, nuclear transcription factor Y, alpha
, CAAT box DNA-binding protein subunit A
, CAAT-box DNA binding protein subunit A
, CCAAT-binding transcription factor subunit B
, HAP2 CCAAT-binding protein
, Transcription factor NF-Y, A subunit
, nuclear transcription factor Y subunit A
, nuclear transcription factor Y subunit alpha
, CAAT-box DNA-binding protein subunit A
, NF-Y protein chain A
, nuclear transcription factor-Y alpha