Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human EPH Receptor B1 Antibodies:
anti-Mouse (Murine) EPH Receptor B1 Antibodies:
anti-Rat (Rattus) EPH Receptor B1 Antibodies:
Go to our pre-filtered search.
Human Polyclonal EPH Receptor B1 Primary Antibody for IHC (p), WB - ABIN391917
Prevost, Woulfe, Tanaka, Brass: Interactions between Eph kinases and ephrins provide a mechanism to support platelet aggregation once cell-to-cell contact has occurred. in Proceedings of the National Academy of Sciences of the United States of America 2002
Show all 3 Pubmed References
Mouse (Murine) Polyclonal EPH Receptor B1 Primary Antibody for ICC, IF - ABIN152616
Dravis, Henkemeyer: Ephrin-B reverse signaling controls septation events at the embryonic midline through separate tyrosine phosphorylation-independent signaling avenues. in Developmental biology 2011
Rat (Rattus) Polyclonal EPH Receptor B1 Primary Antibody for CyTOF, FACS - ABIN4899356
Bush, Soriano: Ephrin-B1 regulates axon guidance by reverse signaling through a PDZ-dependent mechanism. in Genes & development 2009
SUMOylation of EphB1 repressed activation of its downstream signaling molecule PKC-gamma (show PRKCG Antibodies), and consequently inhibited neuroblastoma (show ARHGEF16 Antibodies) tumorigenesis.
some of the mutations found in EPHB1 may contribute to an increased invasive capacity of cancers.
Association of EPHB1 rs11918092 with symptoms of schizophrenia in Chinese Zhuang and Han populations.
The tumor-suppressor function of EphB1 is clinically relevant across many malignancies, suggesting that EphB1 is an important regulator of common cancer cell transforming pathways.
In medulloblastoma cell lines, EphB1 downregulation or knockdown reduced cell growth, viability, cell-cycle regulator expression, and migration, but increased radiosensitivity and the percentage of cells in G1 phase of the cell cycle.
Our results indicate that EphB1 may be involved in carcinogenesis of renal cell carcinoma (show MOK Antibodies)
The genes CD248 (show CD248 Antibodies), Ephb1 and P2RY2 (show P2RY2 Antibodies) were detected as the top overexpressed in GC biopsies.
The study presents the first structure of the EphB1 tyrosine kinase (show TXK Antibodies) domain determined by X-ray crystallography to 2.5A.
EphB1 and Ephrin-B could be regarded as independent good prognostic factors and important biological markers for Squamous cell/adenosquamous carcinoma and adenocarcinoma of gallbladder.
Our data indicate that loss of EphB1 protein is associated with metastasis and poorer survival in patients with serous ovarian cancer
EphA2 (show EPHA2 Antibodies) mAb treatment could partially inhibit LPSinduced inactivation of EphBEphrin B3 signalling, while Ephrin B3 (show EFNB3 Antibodies) overexpression could abrogate LPSinduced activation of EphA2Ephrin A1 signalling. EphB1/Ephrin B3 (show EFNB3 Antibodies) signalling may antagonise the EphA2 (show EPHA2 Antibodies)/Ephrin A1dependent pathway following LPS (show TLR4 Antibodies) treatment
ephrin type-B receptor 1 (EphB1) is upregulated in injured motor neurons, which in turn can activate astrocytes through ephrin-B1 (show EFNB1 Antibodies)-mediated stimulation of signal transducer and activator of transcription-3 (STAT3 (show STAT3 Antibodies))
Findings suggest that a combination of forward and reverse EphB1/2 receptor-mediated signaling contribute to posterior branch of the anterior commissure and corpus callosum axon guidance
During re-epithelialization ephrin-B1 (show EFNB1 Antibodies) and its receptor EphB2 (show EPHB2 Antibodies) are both upregulated in vivo, just for the duration of repair.
In a genetic medulloblastoma model, EphB1 knockout resulted in a significant delay in tumor recurrence following irradiation compared to EphB1-expressing control tumors.
EphB1 expression is activated in the spinal cord in a bone cancer model.
The studies introduce EphB1 as a new venous-restricted marker in a tissue-specific and time-dependent manner.
Reelin (show RELN Antibodies) induces EphB activation.
role of EphB1 receptors signalling in models of inflammatory and neuropathic pain
Ephrin receptors and their ligands, the ephrins, mediate numerous developmental processes, particularly in the nervous system. Based on their structures and sequence relationships, ephrins are divided into the ephrin-A (EFNA) class, which are anchored to the membrane by a glycosylphosphatidylinositol linkage, and the ephrin-B (EFNB) class, which are transmembrane proteins. The Eph family of receptors are divided into 2 groups based on the similarity of their extracellular domain sequences and their affinities for binding ephrin-A and ephrin-B ligands. Ephrin receptors make up the largest subgroup of the receptor tyrosine kinase (RTK) family. The protein encoded by this gene is a receptor for ephrin-B family members.
, EPH-like kinase 6
, eph tyrosine kinase 2
, ephrin type-B receptor 1
, neuronally-expressed EPH-related tyrosine kinase
, soluble EPHB1 variant 1
, tyrosine-protein kinase receptor EPH-2
, Elk-like kinase
, Xenopus Elk-like kinase
, ephrin type-B receptor 1-A
, receptor tyrosine kinase
, tyrosine-protein kinase receptor XEK
, ephrin receptor EphB1
, EPH receptor B1
, ephrin receptor EphB1-like
, ephrin type-B receptor 1-like
, ELK-related protein tyrosine kinase
, Eph receptor B2 (ELK-related protein tyrosine kinase)