Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all synonyms
Human Polyclonal NFATC4 Primary Antibody for IF (p), IHC (p) - ABIN1386186
Xie, Deng, Pan, Ren, Jin, Wang, Wang, Zhu, Guo, Yuan, Shang, Liu: Chronic intermittent hypoxia induces cardiac hypertrophy by impairing autophagy through the adenosine 5'-monophosphate-activated protein kinase pathway. in Archives of biochemistry and biophysics 2016
Dog (Canine) Polyclonal NFATC4 Primary Antibody for ELISA - ABIN450047
Yao, Cho, Bergen, Madden, Choi, Ma, Bode, Dong: Nuclear factor of activated T3 is a negative regulator of Ras-JNK1/2-AP-1 induced cell transformation. in Cancer research 2007
Human Polyclonal NFATC4 Primary Antibody for IHC (p), IHC - ABIN249616
Hoey, Sun, Williamson, Xu: Isolation of two new members of the NF-AT gene family and functional characterization of the NF-AT proteins. in Immunity 1995
Ca(2 (show CA2 Antibodies)+)/calcineurin (CaN)/nuclear factor of activated T-cells (NFAT (show NFATC1 Antibodies)) c4 axis is required for neuritin (show NRN1 Antibodies)-induced Kv4.2 (show KCND2 Antibodies) transcriptional expression and potentiation of IA densities in cerebellum granule neurons.
Nfatc4 expression was coordinately upregulated by top hypoxia-activated transcription factors, while NFATc4 target genes were enriched in hypoxic neural stem cells.
results indicate that NFAT (show NFATC1 Antibodies) is a BACE1 (show BACE Antibodies) transcription factor. Our study suggests that inhibition of NFAT (show NFATC1 Antibodies)-mediated BACE1 (show BACE Antibodies) expression may be a valuable drug target for AD therapy
our results suggest that activation of NHE1 (show SLC9A1 Antibodies) induces hypertrophy through the activation of NFAT3/Gata4 (show GATA4 Antibodies) and OPN (show SPP1 Antibodies) expression
L-carnitine reversed the activation of NFAT3 and PPARa (show PPARA Antibodies) caused by carboplatin through altering the phosphorylation of PTEN and AMPK (show PRKAA1 Antibodies).
Disrupting the calcineurin-NFAT (show NFATC1 Antibodies) axis by either genetic or pharmacologic approaches confers resistance to the development of social stress-induced voiding and dysfunction.
The NFATc4-dependent increase in hippocampal neurogenesis after GABAA (show GABRg1 Antibodies) receptor stimulation is required for suppression of anxiety response.
IL-13 (show IL13 Antibodies) specifically induced NFAT3 activation through promoting its dephosphorylation in air-liquid interface cultures of tracheal epithelial cells (mTECs).
Data suggest that expression of Itpr1 (inositol 1,4,5-triphosphate receptor 1 (show ITPR1 Antibodies)) in cerebral cortex neurons is regulated by dopamine D2 receptors; Itpr1 (show ITPR1 Antibodies) is up-regulated via increased binding of NFATc4 and transcription factor AP-1 (show JUN Antibodies) to Itpr1 (show ITPR1 Antibodies) promoter.
these results reveal a novel PI3K-independent role for p85alpha in controlling VEGF (show VEGFA Antibodies) induction during the cellular UVB response by regulating NFAT3 activity.
NFATc1 (show NFATC1 Antibodies) knockdown strongly reduced the number and the surface area of myotubes, NFATc4 knockdown increased the surface area of myotubes and reduced the pool of reserve cells.
TBX5 (show TBX5 Antibodies) deficiency-mediated downregulation of NFAT3 is crucial for the high cytokine-producing activity of T cells
These results provided evidence supporting the oncogenic potential of NFAT3 and suggested that CDK3 (show CDK3 Antibodies)-mediated phosphorylation of NFAT3 has an important role in skin tumorigenesis.
Data indicate that RNA interference of NFAT (show NFATC1 Antibodies) isoforms NFATc1 (show NFATC1 Antibodies), NFATc2 (show NFAT1 Antibodies), NFATc3 (show NFATC3 Antibodies) and NFATc4 regulate gene expression differentially in human retinal microvascular endothelial cells (HRMEC).
NFAT3 expression plays a role in regulating CXCR4 (show CXCR4 Antibodies) expression.
Suggest nuclear NF-AT3 and NF-AT4 (show NFATC3 Antibodies) participates in atrial structural remodeling, and that PICP and TGF-beta1 (show TGFB1 Antibodies) levels may be sensitive serum biomarkers to estimate atrial structural remodeling with atrial fibrillation.
This is the first study to provide evidence of new and differential roles for NFAT3 and SMAD3 (show SMAD3 Antibodies) in the osteoarthritis process in the regulation of miR (show MLXIP Antibodies)-140 transcription
Expression level of PPP3R1 (show PPP3R1 Antibodies) and GATA4 (show GATA4 Antibodies), and NFATC4 genes for transcription factors did not differ in studied subgroups of patients.
Data indicate that NFATc3 (show NFATC3 Antibodies) undergoes rapid dephosphorylation and nuclear translocation that are essentially complete within 20 min, although NFATc4 remains phosphorylated and localized to the cytosol.
The product of this gene is a member of the nuclear factors of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation and an inducible nuclear component. Other members of this family of nuclear factors of activated T cells also participate in the formation of this complex. The product of this gene plays a role in the inducible expression of cytokine genes in T cells, especially in the induction of the IL-2 and IL-4. Alternatively spliced transcript variants encoding different isoforms have been noted for this gene.
cytoplasmic nuclear factor of activated T-cells 4
, nuclear factor of activated T-cells, cytoplasmic, calcineurin-dependent 4
, nuclear factor of activated T-cells, cytoplasmic 4-like
, T-cell transcription factor NFAT3
, nuclear factor of activated T-cells, cytoplasmic 4
, transcription complex subunit NF-ATc4
, T cell transcription factor NFAT3