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Tumor-associated LECs produce sSEMA4C to promote lymphatic metastasis of tumors.
ectopic expression of miR-25-3p reversed the epithelial-mesenchymal transition phenotype and sensitized cisplatin-resistant cervical cancer cells to cisplatin via targeting Sema4C
Data show that G-protein-coupled receptor kinase-interacting protein 1 (GIT1) and semaphorin 4C (SEMA4C) were found to have putative microRNA miR-138 binding sites in their 3' untranslated region (3'UTRs).
Suggest that up-regulation of miR-125b or targeting Sema4C could serve as novel approaches to reverse chemotherapy resistance in breast cancers.
Data suggest that Erbin can interact with Sema4C, and co-expression of Erbin blocks the process of Sema4C-induced epithelial-mesenchymal transition.
There is a high expression of Sema4C in esophageal cancer, gastric cancer and rectal cancer, and expression is strongly correlated with lymphatic metastasis.
High Sema4C is associated with epithelial-mesenchymal transition and renal fibrosis.
Follicular T helper cells use Semaphorin 4C as a guidance receptor to sense germinal center-expressed Plexin B2.
we demonstrate that Sema4C is critical for optimal regulatory cytokine production in CD138(+) B cells.
Sema4C-Plexin B2 signalling regulates ureteric branching
We have identified here Sema4C and Sema4G as candidate ligands for Plexin-B2
Plexin-B2 and Sema4C are potential regulators of the vascular and endocrine system.
Results suggest a putative role of Sema4C during neurogenesis both in vivo and in vitro.
Sema4C-mediated interaction among myoblasts plays an important role in terminal myogenic differentiation
Sema4C promotes terminal myogenic differentiation in a p38 MAPK-dependent manner.
Cell surface receptor for PLXNB2 that plays an important role in cell-cell signaling. PLXNB2 binding promotes downstream activation of RHOA and phosphorylation of ERBB2 at 'Tyr-1248'. Required for normal brain development, axon guidance and cell migration. Probable signaling receptor which may play a role in myogenic differentiation through activation of the stress- activated MAPK cascade.
, sema I
, semaphorin I
, semaphorin-C-like 1