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Human PGF Protein expressed in Escherichia coli (E. coli) - ABIN413725
Ding, Huang, Song, Gao, Yuan, Wang, Cai, Fu, Luo: NFAT1 mediates placental growth factor-induced myelomonocytic cell recruitment via the induction of TNF-alpha. in Journal of immunology (Baltimore, Md. : 1950) 2010
Rat (Rattus) PGF Protein expressed in Escherichia coli (E. coli) - ABIN2126310
Zhang, Zhao, Yuan, Wu, Jiang, Luo, Zhao: Knockdown of placental growth factor (PLGF) mitigates hyperoxia-induced acute lung injury in neonatal rats: Suppressive effects on NFκB signaling pathway. in International immunopharmacology 2016
PlGF level showed an inversely proportional effect on the foetal weight.
these data suggest that PlGF may increase non-small cell lung cancer metastasis through SRp40 (show SRSF5 Proteins)-mediated mRNA splicing of VEGF (show VEGFA Proteins).
The present study investigated the interplay of VEGF (show VEGFA Proteins)-A165a isoform, the anti-angiogenic VEGF (show VEGFA Proteins)-A165b, placental growth factor (PIGF) and their receptors, VEGFR1 (show FLT1 Proteins) and VEGFR2 (show KDR Proteins), on junctional occupancy of VE-cadherin (show CDH5 Proteins) and macromolecular leakage in human endothelial monolayers and the perfused placental microvascular bed.
PIGF (show PIGF Proteins) enhances TLR-signaling upstream of IKK (show CHUK Proteins) and contributes to an exaggerated pathologic pro-inflammatory state in response to activation of maternal and fetal mononuclear phagocytes by specific TLR agonists
Lower PIGF (show PIGF Proteins) and higher PAPP-A (show PAPPA Proteins) and free beta-hCG (show CGA Proteins) levels were found in the fetal circulation of near-term severe preeclamptic pregnancies
The early variations of PIGF (show PIGF Proteins) and soluble fms-like tyrosine kinase-1 (show FLT1 Proteins) concentrations in newly pregnant obstetric antiphospholipid syndrome (oAPS) may help to detect patients at low risk of placenta-mediated complications (PMC).
There is a significant negative correlation between the concentration of sFLt-1 and PIGF (show PIGF Proteins) in normal pregnancy.
knockdown of PIGF (show PIGF Proteins) in spheroid body cells derived from two gastric cancer cell lines reduced in vitro tumorigenicity and stemness properties of spheroid body cells such as self-renewal ability, colony forming, migratory, and MMPs activities and decreased ability to differentiation and angiogenesis
Data showed that sFlt-1/PIGF (show PIGF Proteins) ratio increases with volume overload and persistent hypoxia after surgery with CHD (show CHDH Proteins).
Glioma cell-released PIGF (show PIGF Proteins) can induce Bregs to suppress CD8 (show CD8A Proteins)(+) T cell activities.
Data indicate that VEGF-B (show VEGFB Proteins) is a high-affinity VEGFR-1 (show FLT1 Proteins) ligand that, unlike PIGF (show PIGF Proteins), cannot efficiently induce signaling downstream of VEGFR-1 (show FLT1 Proteins).
the lack of PIGF (show PIGF Proteins) may have contributed to the delayed angiogenic response and the prothrombotic phenotype observed in the PIGF (show PIGF Proteins)(-/-)animals.
Overexpression of VEGF (show VEGFA Proteins) but not PIGF (show PIGF Proteins) exacerbated the lipopolysaccharide-mediated toxic effects, supporting a pathophysiological role for VEGF (show VEGFA Proteins) in mediating the sepsis phenotype.
Findings indicate that placental growth factor (PlGF) plays an important role in liver fibrosis-associated angiogenesis and that blockage of PlGF could be an effective strategy for chronic liver disease.
Data demonstrate that PlGF is expressed in vivo and that its expression is restricted to the areas of the tumors where the highest levels of hypoxia occur. Also, hypoxia activates PlGF expression in cultured lymphatic endothelial cells.
Study provides mechanistic and clinical evidence that decreased PLGF levels in the placenta after in utero alcohol exposure are associated to brain angiogenesis defects. Measurement of PLGF levels at birth in the placenta or the fetal blood may serve as a predictive marker for subsequent neurodevelopmental outcomes of exposed fetuses.
PGF (show PTGFR Proteins) deficiency is associated with impaired cerebral vascular development in mice.
PlGF inhibition attenuates PERK (show EIF2AK3 Proteins) activation, likely by tempering hypoxia in HCC (show FAM126A Proteins) via vessel normalisation. The UPR is able to regulate PlGF expression, suggesting the existence of a feedback mechanism for hypoxia-mediated UPR
Placenta growth factor augments airway hyperresponsiveness via leukotrienes and IL-13 (show IL13 Proteins).
Data indicate that placental growth factor (PlGF) is needed in the spleen to allow the activation of T cells and blood pressure raising.
Study reports polymorphisms in the bovine PGF gene significantly associated with the maternal effect on stillbirth and calving ease in animals under selection.
placenta growth factor expression is regulated by both VEGF (show VEGFA Proteins) and hyperglycaemia via VEGFR-2 (show KDR Proteins)
This gene encodes a growth factor found in placenta which is homologous to vascular endothelial growth factor. Alternatively spliced transcripts encoding different isoforms have been found for this gene.
placenta growth factor
, placental growth factor, vascular endothelial growth factor-related protein
, placenta growth factor-like