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The protein encoded by TRIM33 is thought to be a transcriptional corepressor. Additionally we are shipping TRIM33 Proteins (6) and many more products for this protein.
Showing 10 out of 95 products:
Human Polyclonal TRIM33 Primary Antibody for WB - ABIN657566
Bai, Kim, Yang, Jurynec, Akie, Lee, LeBlanc, Sessa, Jiang, DiBiase, Zhou, Grunwald, Lin, Cantor, Orkin, Zon: TIF1gamma controls erythroid cell fate by regulating transcription elongation. in Cell 2010
Show all 3 references for ABIN657566
Human Polyclonal TRIM33 Primary Antibody for IF, WB - ABIN2780985
He, Dorn, Erdjument-Bromage, Tempst, Moore, Massagué: Hematopoiesis controlled by distinct TIF1gamma and Smad4 branches of the TGFbeta pathway. in Cell 2006
Human Monoclonal TRIM33 Primary Antibody for IF, IHC (p) - ABIN565640
Ding, Jin, Wang, Chen, Wu, Ai, Chen, Zhang, Liang, Laurence, Zhang, Datta, Zhang, Chen: Reduced expression of transcriptional intermediary factor 1 gamma promotes metastasis and indicates poor prognosis of hepatocellular carcinoma. in Hepatology (Baltimore, Md.) 2014
our findings reveal a new mechanism by which SOX2 (show SOX2 Antibodies)-mediated transcription repression of TIF1gamma promotes TGF-beta (show TGFB1 Antibodies)-induced epithelial-mesenchymal transition in non-small-cell lung cancer
our work indicates that TIF1gamma exerts its tumor-suppressive functions in part by promoting chromosomal stability.
Results show that TRIM33 is significantly downregulated in clear renal cell carcinoma (show MOK Antibodies) tissues which seems to correlate with pathologic stages and grades.
Tumour suppressor TRIM33 targets nuclear beta-catenin (show CTNNB1 Antibodies) degradation
Study suggests TRIM33 and NRAS (show NRAS Antibodies)-CSDE1 (show CDSE1 Antibodies) as candidate genes for autism, and may provide a novel insight into the etiology of autism
The dermatomyositis autoantigen TIF1gamma is markedly up-regulated during muscle regeneration in human and mouse muscle cells.
Data indicate that tripartite motif containing 33 protein TIF1gamma promotes sumoylation of SKI-like proto-oncogene (show RAB1A Antibodies) protein SnoN1 and regulates epithelial-mesenchymal transition (EMT (show ITK Antibodies)).
The ubiquitination of DHX33 (show DHX33 Antibodies) by TRIM33 is lysine 63 specific and is required for the formation of the DHX33 (show DHX33 Antibodies)-NLRP3 (show NLRP3 Antibodies) inflammasome complex.
Results identify a new TGFbeta (show TGFB1 Antibodies) regulatory layer, whereby sumoylation strengthens the TIF1gamma repressive action on canonical TGFbeta (show TGFB1 Antibodies) signaling.
Case Report: suggest that TIF1gamma expression in neoplasms not only determines the tumour activity but also causes dermatomyositis.
Ectodermin is a key switch in the control of TGF-beta (show TGFB1 Antibodies) gene responses during early embryonic development and cell proliferation.
Trim33 altered expression of a small group of genes in the testis and the gene with the most significant increase was transcribed from an upstream RLTR10B.
TRIM33 deficiency results in high expression of Ifnb1 (show IFNB1 Antibodies) at late stages of macrophages activation.
The findings reveal an essential role for TRIM33 in preventing apoptosis in B lymphoblastic leukemia by interfering with enhancer-mediated Bim (show BCL2L11 Antibodies) activation.
This study demonistrated that Smad4 (show SMAD4 Antibodies) and Trim33 regulate neural stem cells in the developing cortex in mice.
Epithelium-specific Tak1 (show NR2C2 Antibodies):Smad4 (show SMAD4 Antibodies)- and Trim33:Smad4 (show SMAD4 Antibodies)-double mutants display reduced expression of Mmp13 (show MMP13 Antibodies) in palatal medial edge epithelial cells.
These data provide a new paradigm for Tif1gamma in regulating the balance between lymphoid- and myeloid-derived hematopoietic stem cells (HSC (show FUT1 Antibodies)) through TGF-beta (show TGFB1 Antibodies) signaling, leading to HSC (show FUT1 Antibodies) aging.
TRIM33 plays a role in PARP (show PARP1 Antibodies)-dependent DNA damage response and regulates ALC1 (show MYL4 Antibodies) activity by promoting its timely removal from sites of DNA damage.
TIF1gamma plays essential roles in multiple murine blood lineages and that its function in transcription elongation is evolutionally conserved.
The authors show that tif1gamma modulates the erythroid versus myeloid fate outcomes from HSCs by differentially controlling the levels of gata1 (show GATA1 Antibodies) and pu.1.
TIF1gamma couples the blood-specific transcriptional complex with Pol II elongation machinery to promote the transcription elongation of erythroid genes by counteracting Pol II pausing.
The protein encoded by this gene is thought to be a transcriptional corepressor. However, molecules that interact with this protein have not yet been identified. The protein is a member of the tripartite motif family. This motif includes three zinc-binding domains, a RING, a B-box type 1 and a B-box type 2, and a coiled-coil region. Three alternatively spliced transcript variants for this gene have been described, however, the full-length nature of one variant has not been determined.
tripartite motif-containing 33
, tripartite motif-containing 33 protein
, E3 ubiquitin-protein ligase TRIM33
, e3 ubiquitin-protein ligase TRIM33-like
, tripartite motif containing 33
, RET-fused gene 7 protein
, ectodermin homolog
, protein Rfg7
, transcriptional intermediary factor 1 gamma
, transcription intermediary factor 1-gamma
, tripartite motif-containing protein 33
, tripartite motif protein 33